| scholarly article | Q13442814 |
| P356 | DOI | 10.1111/J.1460-9568.2010.07215.X |
| P8608 | Fatcat ID | release_jd4v6tnb7ndsth6n4ekcbnmota |
| P932 | PMC publication ID | 2931320 |
| P698 | PubMed publication ID | 20497470 |
| P5875 | ResearchGate publication ID | 44625541 |
| P2093 | author name string | Jianning Wei | |
| Mark McCollum | |||
| Nithya Bhagavatula | |||
| Onome Ulukpo | |||
| Rebecca Leon | |||
| P2860 | cites work | BH3-only proteins Bid and Bim(EL) are differentially involved in neuronal dysfunction in mouse models of Huntington's disease | Q45304659 |
| Global changes to the ubiquitin system in Huntington's disease. | Q45305824 | ||
| Cytoplasmic inclusions of Htt exon1 containing an expanded polyglutamine tract suppress execution of apoptosis in sympathetic neurons. | Q46178162 | ||
| Bim is elevated in Alzheimer's disease neurons and is required for beta-amyloid-induced neuronal apoptosis. | Q48298601 | ||
| PARP cleavage as a means of assessing apoptosis | Q79317596 | ||
| Regulatory phosphorylation of Bim: sorting out the ERK from the JNK | Q81833475 | ||
| ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats | Q24298956 | ||
| Bid, a Bcl2 Interacting Protein, Mediates Cytochrome c Release from Mitochondria in Response to Activation of Cell Surface Death Receptors | Q24310559 | ||
| Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis | Q24310597 | ||
| Polyglutamine expansion of huntingtin impairs its nuclear export | Q24338269 | ||
| JNK phosphorylation of Bim-related members of the Bcl2 family induces Bax-dependent apoptosis | Q24554008 | ||
| Characterization of the c-Jun N-terminal kinase-BimEL signaling pathway in neuronal apoptosis. | Q27919665 | ||
| The proapoptotic activity of the Bcl-2 family member Bim is regulated by interaction with the dynein motor complex | Q28141249 | ||
| Induction of BIM, a proapoptotic BH3-only BCL-2 family member, is critical for neuronal apoptosis | Q28212224 | ||
| Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis | Q28511080 | ||
| Amino-terminal fragments of mutant huntingtin show selective accumulation in striatal neurons and synaptic toxicity | Q28513938 | ||
| Impairment of the ubiquitin-proteasome system by protein aggregation | Q29614556 | ||
| ER stress triggers apoptosis by activating BH3-only protein Bim | Q29617730 | ||
| Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain | Q29617982 | ||
| Increased susceptibility of cytoplasmic over nuclear polyglutamine aggregates to autophagic degradation | Q33947993 | ||
| BH3-Only proteins-essential initiators of apoptotic cell death | Q34117362 | ||
| Failure of the ubiquitin-proteasome system in Parkinson's disease | Q34325371 | ||
| Mutant huntingtin alters MAPK signaling pathways in PC12 and striatal cells: ERK1/2 protects against mutant huntingtin-associated toxicity. | Q34473189 | ||
| Apoptosis in Huntington's disease. | Q35092246 | ||
| Nonapoptotic neurodegeneration in a transgenic mouse model of Huntington's disease | Q35176635 | ||
| The proapoptotic BH3-only protein bim is expressed in hematopoietic, epithelial, neuronal, and germ cells | Q35745742 | ||
| Two molecular pathways initiate mitochondria-dependent dopaminergic neurodegeneration in experimental Parkinson's disease | Q35808960 | ||
| Impaired ubiquitin-proteasome system activity in the synapses of Huntington's disease mice | Q36534628 | ||
| Multisite phosphorylation regulates Bim stability and apoptotic activity | Q36756298 | ||
| Aggregation of huntingtin in yeast varies with the length of the polyglutamine expansion and the expression of chaperone proteins | Q37109961 | ||
| Inhibition of the ubiquitin-proteasome system in Alzheimer's disease. | Q37387675 | ||
| Increased expression of Bim contributes to the potentiation of serum deprivation-induced apoptotic cell death in Huntington's disease knock-in striatal cell line | Q39952286 | ||
| Global impairment of the ubiquitin-proteasome system by nuclear or cytoplasmic protein aggregates precedes inclusion body formation. | Q40461698 | ||
| Extracellular signal-regulated kinases 1/2 are serum-stimulated "Bim(EL) kinases" that bind to the BH3-only protein Bim(EL) causing its phosphorylation and turnover | Q40606380 | ||
| Phosphorylation of Bim-EL by Erk1/2 on serine 69 promotes its degradation via the proteasome pathway and regulates its proapoptotic function. | Q40626751 | ||
| Altered proteasomal function due to the expression of polyglutamine-expanded truncated N-terminal huntingtin induces apoptosis by caspase activation through mitochondrial cytochrome c release | Q40808988 | ||
| Intranuclear huntingtin increases the expression of caspase-1 and induces apoptosis | Q40840397 | ||
| Importance of poly(ADP-ribose) polymerase and its cleavage in apoptosis. Lesson from an uncleavable mutant | Q40990869 | ||
| Intraneuronal aggregate formation and cell death after viral expression of expanded polyglutamine tracts in the adult rat brain. | Q41713142 | ||
| Endoplasmic reticulum stress and alteration in calcium homeostasis are involved in cadmium-induced apoptosis | Q42827151 | ||
| FOXO transcription factors directly activate bim gene expression and promote apoptosis in sympathetic neurons | Q42918105 | ||
| Aggregate formation inhibits proteasomal degradation of polyglutamine proteins. | Q44175807 | ||
| JNK-mediated BIM phosphorylation potentiates BAX-dependent apoptosis | Q44485247 | ||
| Neuronal induction of the immunoproteasome in Huntington's disease. | Q44699105 | ||
| Sequential activation of individual caspases, and of alterations in Bcl‐2 proapoptotic signals in a mouse model of Huntington's disease | Q45291644 | ||
| Accumulation of aberrant ubiquitin induces aggregate formation and cell death in polyglutamine diseases | Q45293883 | ||
| Proteasome impairment does not contribute to pathogenesis in R6/2 Huntington's disease mice: exclusion of proteasome activator REGgamma as a therapeutic target | Q45299199 | ||
| P433 | issue | 11 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | cell death | Q2383867 |
| P304 | page(s) | 1915-1925 | |
| P577 | publication date | 2010-05-24 | |
| P1433 | published in | European Journal of Neuroscience | Q5412733 |
| P1476 | title | BimEL as a possible molecular link between proteasome dysfunction and cell death induced by mutant huntingtin | |
| P478 | volume | 31 |
| Q47288108 | Aberrant subcellular localization of SQSTM1/p62 contributes to increased vulnerability to proteotoxic stress recovery in Huntington's disease |
| Q45289973 | Bcl-2/adenovirus E1B 19-kDa interacting protein (BNip3) has a key role in the mitochondrial dysfunction induced by mutant huntingtin. |
| Q38818046 | Bim gene dosage is critical in modulating nephron progenitor survival in the absence of microRNAs during kidney development |
| Q37527207 | Caspase-2 is essential for c-Jun transcriptional activation and Bim induction in neuron death. |
| Q58994518 | Crosstalk between Endoplasmic Reticulum Stress and Protein Misfolding in Neurodegenerative Diseases |
| Q26822846 | Defining the role of the Bcl-2 family proteins in Huntington's disease |
| Q36283269 | Palmitoylation and trafficking of GAD65 are impaired in a cellular model of Huntington's disease |
| Q37930038 | Pathogenic Mechanisms in Huntington's Disease |
| Q26781203 | Regulation of Bim in Health and Disease |
| Q58801228 | The Parkinson's disease gene product DJ-1 modulates miR-221 to promote neuronal survival against oxidative stress |
| Q26771343 | The Unfolded Protein Response and the Role of Protein Disulfide Isomerase in Neurodegeneration |
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