| scholarly article | Q13442814 |
| P6179 | Dimensions Publication ID | 1020489619 |
| P356 | DOI | 10.1186/1756-6606-4-43 |
| P932 | PMC publication ID | 3278372 |
| P698 | PubMed publication ID | 22188897 |
| P5875 | ResearchGate publication ID | 51905919 |
| P50 | author | Birgit Liß | Q865268 |
| Sarina K Mueller | Q89065311 | ||
| Douglass Matthew Turnbull | Q24631614 | ||
| Holger Prokisch | Q28050058 | ||
| Eliezer Masliah | Q28324209 | ||
| Falk Schlaudraff | Q40638443 | ||
| P2093 | author name string | Chris Morris | |
| Andreas Bender | |||
| Thomas Klopstock | |||
| Christoph Laub | |||
| Lars Leidolt | |||
| Lena Krieg | |||
| Matthias Elstner | |||
| Sarina K Müller | |||
| P2860 | cites work | Individual dopaminergic neurons show raised iron levels in Parkinson disease. | Q45305197 |
| Neuromelanin can protect against iron-mediated oxidative damage in system modeling iron overload of brain aging and Parkinson's disease | Q46485512 | ||
| Dopaminergic midbrain neurons are the prime target for mitochondrial DNA deletions | Q46499311 | ||
| The substantia nigra of the human brain. II. Patterns of loss of dopamine-containing neurons in Parkinson's disease | Q48147372 | ||
| Mitochondrial DNA deletions in human brain: regional variability and increase with advanced age | Q48403808 | ||
| Is the vulnerability of neurons in the substantia nigra of patients with Parkinson's disease related to their neuromelanin content? | Q48435040 | ||
| Mitochondrial DNA deletions are abundant and cause functional impairment in aged human substantia nigra neurons | Q48588748 | ||
| Specific A10 dopaminergic nuclei in the midbrain degenerate in Parkinson's disease | Q48775742 | ||
| Correspondence of melanin-pigmented neurons in human brain with A1-A14 catecholamine cell groups | Q48973700 | ||
| Kinetic and structural analysis of the early oxidation products of dopamine: analysis of the interactions with alpha-synuclein. | Q53285842 | ||
| Parkinsonism, premature menopause, and mitochondrial DNA polymerase gamma mutations: clinical and molecular genetic study. | Q53612457 | ||
| Evidence for specific phases in the development of human neuromelanin | Q59544627 | ||
| Tyrosine hydroxylase and methionine-enkephalin in the human mesencephalon | Q59578576 | ||
| A multiplex real-time PCR method to detect and quantify mitochondrial DNA deletions in individual cells | Q80699747 | ||
| Increased expression of α-synuclein in aged human brain associated with neuromelanin accumulation | Q83774812 | ||
| Neuromelanin in human dopamine neurons: Comparison with peripheral melanins and relevance to Parkinson's disease | Q22251216 | ||
| Staging of brain pathology related to sporadic Parkinson's disease | Q28131702 | ||
| The biologic clock: the mitochondria? | Q28249363 | ||
| Oxidant stress evoked by pacemaking in dopaminergic neurons is attenuated by DJ-1 | Q28510145 | ||
| Diagnosis and management of dementia with Lewy bodies: third report of the DLB Consortium | Q29614409 | ||
| Ageing and Parkinson's disease: substantia nigra regional selectivity | Q29615835 | ||
| High levels of mitochondrial DNA deletions in substantia nigra neurons in aging and Parkinson disease | Q29619113 | ||
| Neuromelanin activates microglia and induces degeneration of dopaminergic neurons: implications for progression of Parkinson's disease | Q30537842 | ||
| Alpha-synuclein redistributes to neuromelanin lipid in the substantia nigra early in Parkinson's disease. | Q30856480 | ||
| Expression analysis of dopaminergic neurons in Parkinson's disease and aging links transcriptional dysregulation of energy metabolism to cell death. | Q33889585 | ||
| Neuromelanin and its interaction with iron as a potential risk factor for dopaminergic neurodegeneration underlying Parkinson's disease | Q35165428 | ||
| Modifications of the iron-neuromelanin system in Parkinson's disease | Q36370608 | ||
| Multiple hit hypotheses for dopamine neuron loss in Parkinson's disease | Q36785396 | ||
| Redox imbalance in Parkinson's disease | Q36899782 | ||
| Anatomy, pigmentation, ventral and dorsal subpopulations of the substantia nigra, and differential cell death in Parkinson's disease | Q36947501 | ||
| The comparative biology of neuromelanin and lipofuscin in the human brain | Q37085791 | ||
| What causes mitochondrial DNA deletions in human cells? | Q37096545 | ||
| A pattern of accumulation of a somatic deletion of mitochondrial DNA in aging human tissues | Q37147048 | ||
| Interplay between cytosolic dopamine, calcium, and alpha-synuclein causes selective death of substantia nigra neurons | Q37180671 | ||
| The origins of oxidant stress in Parkinson's disease and therapeutic strategies. | Q37780206 | ||
| Mitochondrial contribution to Parkinson's disease pathogenesis | Q37891820 | ||
| Mitochondrial DNA deletions cause the biochemical defect observed in Alzheimer's disease. | Q39706666 | ||
| Neuromelanin selectively induces apoptosis in dopaminergic SH-SY5Y cells by deglutathionylation in mitochondria: involvement of the protein and melanin component | Q39993657 | ||
| Differential effects of human neuromelanin and synthetic dopamine melanin on neuronal and glial cells. | Q40379280 | ||
| Melanized dopaminergic neurons are differentially susceptible to degeneration in Parkinson's disease | Q41346993 | ||
| Glutathione Peroxidase 4 is associated with Neuromelanin in Substantia Nigra and Dystrophic Axons in Putamen of Parkinson's brain | Q41981348 | ||
| Neuronal loss is greater in the locus coeruleus than nucleus basalis and substantia nigra in Alzheimer and Parkinson diseases | Q42438205 | ||
| Nature of mitochondrial DNA deletions in substantia nigra neurons | Q43058638 | ||
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | mitochondrial DNA | Q27075 |
| neurotransmitter | Q162657 | ||
| P304 | page(s) | 43 | |
| P577 | publication date | 2011-12-21 | |
| P1433 | published in | Molecular Brain | Q6895938 |
| P1476 | title | Neuromelanin, neurotransmitter status and brainstem location determine the differential vulnerability of catecholaminergic neurons to mitochondrial DNA deletions | |
| P478 | volume | 4 |
| Q34864555 | Accumulation of mitochondrial DNA deletions within dopaminergic neurons triggers neuroprotective mechanisms |
| Q27021628 | Ageing and Parkinson's disease: why is advancing age the biggest risk factor? |
| Q38770352 | Causes, consequences, and cures for neuroinflammation mediated via the locus coeruleus: noradrenergic signaling system |
| Q36845872 | Digital PCR methods improve detection sensitivity and measurement precision of low abundance mtDNA deletions. |
| Q91495371 | Enhancers active in dopamine neurons are a primary link between genetic variation and neuropsychiatric disease |
| Q90553009 | Loss of Brain Norepinephrine Elicits Neuroinflammation-Mediated Oxidative Injury and Selective Caudo-Rostral Neurodegeneration |
| Q34090228 | Mitochondrial DNA damage: molecular marker of vulnerable nigral neurons in Parkinson's disease |
| Q38310102 | Molecular determinants of selective dopaminergic vulnerability in Parkinson's disease: an update |
| Q42828340 | Molecular pathogenesis of polymerase γ-related neurodegeneration |
| Q26829851 | Mouse models of Parkinson's disease associated with mitochondrial dysfunction |
| Q33676222 | Neuroimaging in pre-motor Parkinson's disease |
| Q38074802 | Oxidative damage to macromolecules in human Parkinson disease and the rotenone model |
| Q92186851 | Spread of α-synuclein pathology through the brain connectome is modulated by selective vulnerability and predicted by network analysis |
| Q38034189 | The mitochondrial genome and psychiatric illness |
| Q24307946 | The role of oxidative stress in Parkinson's disease |
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