scholarly article | Q13442814 |
P2093 | author name string | Minyoung Lee | |
Jae-Seon Lee | |||
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A novel type of cellular senescence that can be enhanced in mouse models and human tumor xenografts to suppress prostate tumorigenesis | Q33685619 | ||
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Cellular and molecular mechanisms of stress-induced premature senescence (SIPS) of human diploid fibroblasts and melanocytes | Q34106524 | ||
Aging, Cellular Senescence, and Cancer | Q34199776 | ||
Therapy-Induced Senescence in Cancer | Q34212773 | ||
Proteolyzed matrix as a template for the regulation of tumor progression | Q34218154 | ||
The p53 network: cellular and systemic DNA damage responses in aging and cancer | Q34248711 | ||
The essence of senescence | Q34288983 | ||
Secretome analysis of ionizing radiation‐induced senescent cancer cells reveals that secreted RKIP plays a critical role in neighboring cell migration | Q34352155 | ||
The expression signature of in vitro senescence resembles mouse but not human aging | Q34481078 | ||
Oncogenic functions of tumour suppressor p21(Waf1/Cip1/Sdi1): association with cell senescence and tumour-promoting activities of stromal fibroblasts | Q34552889 | ||
The regulation of INK4/ARF in cancer and aging | Q34575801 | ||
The identification of senescence-specific genes during the induction of senescence in prostate cancer cells | Q34769742 | ||
SASP reflects senescence. | Q34945503 | ||
Oncogenic Braf induces melanocyte senescence and melanoma in mice | Q34974162 | ||
PTEN status switches cell fate between premature senescence and apoptosis in glioma exposed to ionizing radiation | Q35092641 | ||
The ARF-p53 senescence pathway in mouse and human cells | Q35619087 | ||
The normal response to RAS: senescence or transformation? | Q35778503 | ||
The signals and pathways activating cellular senescence | Q36060167 | ||
Tumour-associated macrophages are a distinct M2 polarised population promoting tumour progression: potential targets of anti-cancer therapy | Q36414978 | ||
Senescence surveillance of pre-malignant hepatocytes limits liver cancer development | Q53206297 | ||
Senescence-associated oxidative DNA damage promotes the generation of neoplastic cells. | Q53365811 | ||
Induction of p53-dependent senescence by the MDM2 antagonist nutlin-3a in mouse cells of fibroblast origin | Q53536765 | ||
Molecular signature of oncogenic ras-induced senescence | Q53632631 | ||
Expression and growth-promoting function of the IL-8 receptor beta in human melanoma cells | Q70890742 | ||
Role of telomeres and telomerase in genomic instability, senescence and cancer | Q36927502 | ||
Evolving role of uPA/uPAR system in human cancers | Q37046231 | ||
Stress-induced premature senescence (SIPS)--influence of SIPS on radiotherapy | Q37064796 | ||
Senescent stromal-derived osteopontin promotes preneoplastic cell growth | Q37078729 | ||
Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation | Q37082277 | ||
Cellular senescence in oral cancer and precancer and treatment implications: a review | Q37209689 | ||
Control of senescence by CXCR2 and its ligands | Q37288307 | ||
Cell surface-bound IL-1alpha is an upstream regulator of the senescence-associated IL-6/IL-8 cytokine network | Q37385995 | ||
The role of epigenetics in aging and age-related diseases | Q37589034 | ||
Healing and hurting: molecular mechanisms, functions, and pathologies of cellular senescence | Q37612255 | ||
Tumor suppression by ARF: gatekeeper and caretaker | Q37656961 | ||
Oncogene-induced senescence: the bright and dark side of the response | Q37784324 | ||
Extracellular signals in young and aging breast epithelial cells and possible connections to age-associated breast cancer development | Q37867199 | ||
Pro-senescence therapy for cancer treatment | Q37893959 | ||
Oncogene- and tumor suppressor gene-mediated suppression of cellular senescence | Q37950784 | ||
Emerging role of NF-κB signaling in the induction of senescence-associated secretory phenotype (SASP). | Q37969844 | ||
Senescence: a new weapon for cancer therapy | Q37975834 | ||
Emerging roles of RB family: new defense mechanisms against tumor progression | Q38034093 | ||
p21WAF1 and tumourigenesis: 20 years after | Q38060451 | ||
Stressing the cell cycle in senescence and aging | Q38126692 | ||
Immunosurveillance of senescent cells: the bright side of the senescence program | Q38151547 | ||
A novel telomerase template antagonist (GRN163) as a potential anticancer agent | Q38352368 | ||
alpha5beta1 integrin antagonists reduce chemotherapy-induced premature senescence and facilitate apoptosis in human glioblastoma cells | Q39748375 | ||
Prevention of premature senescence requires JNK regulation of Bcl-2 and reactive oxygen species | Q39783612 | ||
Cytokine expression and signaling in drug-induced cellular senescence. | Q39791621 | ||
Cathepsin D and eukaryotic translation elongation factor 1 as promising markers of cellular senescence | Q39844229 | ||
Fibroblasts isolated from common sites of breast cancer metastasis enhance cancer cell growth rates and invasiveness in an interleukin-6-dependent manner. | Q39923052 | ||
Accelerated senescence: an emerging role in tumor cell response to chemotherapy and radiation | Q39957013 | ||
Senescent human fibroblasts increase the early growth of xenograft tumors via matrix metalloproteinase secretion | Q40149352 | ||
Secretion of vascular endothelial growth factor by primary human fibroblasts at senescence | Q40249487 | ||
The role of histone acetylation versus DNA damage in drug-induced senescence and apoptosis | Q40300478 | ||
Cellular mechanisms for low-dose ionizing radiation-induced perturbation of the breast tissue microenvironment | Q40390428 | ||
Characterization of insulin-like growth factor-binding protein-related proteins (IGFBP-rPs) 1, 2, and 3 in human prostate epithelial cells: potential roles for IGFBP-rP1 and 2 in senescence of the prostatic epithelium | Q40840841 | ||
Murine fibroblasts lacking p21 undergo senescence and are resistant to transformation by oncogenic Ras. | Q40928334 | ||
Replicative senescence: an old lives' tale? | Q40968415 | ||
Characterization of IGFBP-3, PAI-1 and SPARC mRNA expression in senescent fibroblasts | Q41144365 | ||
AKT induces senescence in primary esophageal epithelial cells but is permissive for differentiation as revealed in organotypic culture. | Q41284390 | ||
Epithelial-mesenchymal transition and senescence: two cancer-related processes are crossing paths | Q41438730 | ||
Interleukin 1 alpha is a marker of endothelial cellular senescent. | Q41676538 | ||
Irradiated mammary gland stroma promotes the expression of tumorigenic potential by unirradiated epithelial cells | Q41726147 | ||
Epithelial-Mesenchymal Transition Induced by Senescent Fibroblasts | Q41835409 | ||
Oncogenic Ras induces p19ARF and growth arrest in mouse embryo fibroblasts lacking p21Cip1 and p27Kip1 without activating cyclin D-dependent kinases | Q42801129 | ||
Tumour biology: senescence in premalignant tumours | Q42802962 | ||
VHL loss actuates a HIF-independent senescence programme mediated by Rb and p400. | Q43455466 | ||
Absence of p21WAF1 cooperates with c-myc in bypassing Ras-induced senescence and enhances oncogenic cooperation | Q47349068 | ||
Rb Regulates DNA damage response and cellular senescence through E2F-dependent suppression of N-ras isoprenylation | Q48673271 | ||
The gene expression program of prostate fibroblast senescence modulates neoplastic epithelial cell proliferation through paracrine mechanisms | Q51285272 | ||
P275 | copyright license | Creative Commons Attribution-NonCommercial 3.0 Unported | Q18810331 |
P433 | issue | 2 | |
P921 | main subject | cellular senescence | Q9075999 |
neoplasm | Q1216998 | ||
antineoplastic | Q2853144 | ||
P5008 | on focus list of Wikimedia project | ScienceSource | Q55439927 |
P304 | page(s) | 51-59 | |
P577 | publication date | 2014-02-01 | |
P13046 | publication type of scholarly work | review article | Q7318358 |
P1433 | published in | BMB Reports | Q15764027 |
P1476 | title | Exploiting tumor cell senescence in anticancer therapy | |
P478 | volume | 47 |
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Q64883108 | High COX-2 expression contributes to a poor prognosis through the inhibition of chemotherapy-induced senescence in nasopharyngeal carcinoma. |
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