Prenatal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin produces alterations in cortical neuron development and a long-term dysfunction of glutamate transmission in rat cerebral cortex.

scientific article

Prenatal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin produces alterations in cortical neuron development and a long-term dysfunction of glutamate transmission in rat cerebral cortex. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1016/J.NEUINT.2012.07.004
P698PubMed publication ID22796212

P50authorLuca FerraroQ38548471
Sergio TanganelliQ40454146
Tiziana AntonelliQ42804912
Maria C. TomasiniQ55153359
Sarah BeggiatoQ92522061
P2093author name stringLuca Marani
Luca Lorenzini
P2860cites workAn overview of the effects of dioxins and dioxin-like compounds on vertebrates, as documented in human and ecological epidemiologyQ37459469
P433issue5
P304page(s)759-766
P577publication date2012-07-10
P1433published inNeurochemistry InternationalQ7002160
P1476titlePrenatal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin produces alterations in cortical neuron development and a long-term dysfunction of glutamate transmission in rat cerebral cortex
P478volume61

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cites work (P2860)
Q387853232,3,7,8-tetrachlorodibenzo-p-dioxin exposure influence the expression of glutamate transporter GLT-1 in C6 glioma cells via the Ca(2+) /protein kinase C pathway
Q26827706Actions of endocrine-disrupting chemicals on stem/progenitor cells during development and disease
Q46684857GET73 Prevents Ethanol-Induced Neurotoxicity in Primary Cultures of Rat Hippocampal Neurons
Q36747763Prenatal Exposure to Arsenic Impairs Behavioral Flexibility and Cortical Structure in Mice
Q58704111The Aryl Hydrocarbon Receptor and the Nervous System

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