| P2093 | author name string | Kato K | |
| | Matsuda T | |
| | Inoue T | |
| | Yoshikawa T | |
| | Arima T | |
| | Wake N | |
| | Kondoh H | |
| | Asanoma K | |
| | Kato HD | |
| P2860 | cites work | Mediation of the DCC apoptotic signal by DIP13 alpha | Q24297448 |
| | Deleted in Colorectal Cancer (DCC) encodes a netrin receptor | Q24313278 |
| | The dependence receptor DCC (deleted in colorectal cancer) defines an alternative mechanism for caspase activation | Q24630141 |
| | The netrin-1 receptors UNC5H are putative tumor suppressors controlling cell death commitment | Q24683182 |
| | The DCC gene product induces apoptosis by a mechanism requiring receptor proteolysis | Q28116063 |
| | Homozygous deletions inactivate DCC, but not MADH4/DPC4/SMAD4, in a subset of pancreatic and biliary cancers | Q28138752 |
| | p53RDL1 regulates p53-dependent apoptosis | Q28211258 |
| | Phenotype of mice lacking functional Deleted in colorectal cancer (Dcc) gene | Q28236533 |
| | The DCC gene product in cellular differentiation and colorectal tumorigenesis | Q28238617 |
| | The DCC gene: structural analysis and mutations in colorectal carcinomas | Q28253945 |
| | Identification of a chromosome 18q gene that is altered in colorectal cancers | Q28274172 |
| | Detection of messenger RNA by in situ hybridization to tissue sections and whole mounts | Q29618776 |
| | Netrin-1 acts as a survival factor via its receptors UNC5H and DCC. | Q33949395 |
| | Netrin-1-mediated axon outgrowth requires deleted in colorectal cancer-dependent MAPK activation | Q34126386 |
| | Loss of heterozygosity on chromosome 18q is associated with muscle-invasive transitional cell carcinoma of the bladder | Q36079675 |
| | Loss of expression and loss of heterozygosity in the DCC gene in neoplasms of the human female reproductive tract | Q36080488 |
| | Suppressed tumorigenicity of human endometrial cancer cells by the restored expression of the DCC gene | Q36621039 |
| | Rac1 and Cdc42 but not RhoA or Rho kinase activities are required for neurite outgrowth induced by the Netrin-1 receptor DCC (deleted in colorectal cancer) in N1E-115 neuroblastoma cells | Q38292096 |
| | Evidence for a common molecular pathogenesis in colorectal, gastric, and pancreatic cancer | Q45022137 |
| | Expression of Netrin-1 and its two receptors DCC and UNC5H2 in the developing mouse lung. | Q54768547 |
| | DCC gene alteration in human endometrial carcinomas | Q71623317 |
| | Loss of expression of the gene deleted in colon carcinoma (DCC) is closely related to histologic differentiation and lymph node metastasis in endometrial carcinoma | Q74451725 |
| | Expression of DCC protein in colorectal tumors and its relationship to tumor progression and metastasis | Q78016109 |
| P433 | issue | 2 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | carcinogenesis | Q1637543 |
| P304 | page(s) | 281-289 | |
| P577 | publication date | 2004-11-01 | |
| P1433 | published in | Gynecologic Oncology | Q5625182 |
| P1476 | title | Expression of DCC and netrin-1 in normal human endometrium and its implication in endometrial carcinogenesis | |
| P478 | volume | 95 | |