scholarly article | Q13442814 |
P2093 | author name string | Ismail Iraqui | |
Audrey Costes | |||
Karine Fréon | |||
Nada Jmari | |||
Sarah A E Lambert | |||
Violena Pietrobon | |||
Yasmina Chekkal | |||
P2860 | cites work | The major roles of DNA polymerases epsilon and delta at the eukaryotic replication fork are evolutionarily conserved | Q21144935 |
Replication stalling at unstable inverted repeats: interplay between DNA hairpins and fork stabilizing proteins | Q24655285 | ||
Break-induced replication requires all essential DNA replication factors except those specific for pre-RC assembly. | Q27930164 | ||
Break-induced replication and telomerase-independent telomere maintenance require Pol32. | Q27933035 | ||
Increased mutagenesis and unique mutation signature associated with mitotic gene conversion | Q27934880 | ||
S-phase checkpoint proteins Tof1 and Mrc1 form a stable replication-pausing complex | Q27938718 | ||
Break-induced replication and recombinational telomere elongation in yeast | Q28244552 | ||
Chromosomal translocations in yeast induced by low levels of DNA polymerase a model for chromosome fragile sites. | Q52563158 | ||
Template switching during break-induced replication | Q56521356 | ||
SUMOylation regulates Rad18-mediated template switch | Q58883728 | ||
Homologous Recombination Restarts Blocked Replication Forks at the Expense of Genome Rearrangements by Template Exchange | Q59198307 | ||
A Postincision-Deficient TFIIH Causes Replication Fork Breakage and Uncovers Alternative Rad51- or Pol32-Mediated Restart Mechanisms | Q63383451 | ||
Smc5/6: a link between DNA repair and unidirectional replication? | Q63383716 | ||
Gross Chromosomal Rearrangements and Elevated Recombination at an Inducible Site-Specific Replication Fork Barrier | Q63383718 | ||
POL32, a subunit of the Saccharomyces cerevisiae DNA polymerase delta, defines a link between DNA replication and the mutagenic bypass repair pathway | Q73319794 | ||
Replisome fate upon encountering a leading strand block and clearance from DNA by recombination proteins | Q80563737 | ||
The replication fork's five degrees of freedom, their failure and genome rearrangements | Q37632795 | ||
Maintaining genome stability at the replication fork | Q37696954 | ||
Pathways of mammalian replication fork restart. | Q37788950 | ||
How dormant origins promote complete genome replication | Q37884997 | ||
The consequences of structural genomic alterations in humans: genomic disorders, genomic instability and cancer | Q37908515 | ||
Replication stress and mechanisms of CNV formation | Q37988151 | ||
Control of translocations between highly diverged genes by Sgs1, the Saccharomyces cerevisiae homolog of the Bloom's syndrome protein | Q38406137 | ||
Molecular profiling of common fragile sites in human fibroblasts. | Q39447595 | ||
Failure of origin activation in response to fork stalling leads to chromosomal instability at fragile sites | Q39512958 | ||
Genetic control of microsatellite stability | Q41355776 | ||
Rad8Rad5/Mms2-Ubc13 ubiquitin ligase complex controls translesion synthesis in fission yeast | Q41465123 | ||
Gross chromosomal rearrangements in Saccharomyces cerevisiae replication and recombination defective mutants | Q41688249 | ||
The DNA helicase Pfh1 promotes fork merging at replication termination sites to ensure genome stability | Q41779826 | ||
Fbh1 limits Rad51-dependent recombination at blocked replication forks. | Q41865081 | ||
Specific pathways prevent duplication-mediated genome rearrangements | Q41927457 | ||
The F-Box DNA helicase Fbh1 prevents Rhp51-dependent recombination without mediator proteins. | Q42107587 | ||
Chromosomal translocations caused by either pol32-dependent or pol32-independent triparental break-induced replication | Q42160533 | ||
Stalled fork rescue via dormant replication origins in unchallenged S phase promotes proper chromosome segregation and tumor suppression. | Q42702121 | ||
Nearby inverted repeats fuse to generate acentric and dicentric palindromic chromosomes by a replication template exchange mechanism. | Q42945056 | ||
Differential regulation of homologous recombination at DNA breaks and replication forks by the Mrc1 branch of the S-phase checkpoint | Q43125336 | ||
Replisome stability at defective DNA replication forks is independent of S phase checkpoint kinases | Q44294870 | ||
Exo1 processes stalled replication forks and counteracts fork reversal in checkpoint-defective cells | Q45209742 | ||
Multiple pathways cooperate in the suppression of genome instability in Saccharomyces cerevisiae | Q46062549 | ||
Genome instability: a mechanistic view of its causes and consequences | Q28266306 | ||
Replisome assembly and the direct restart of stalled replication forks | Q28276681 | ||
Recombination proteins and rescue of arrested replication forks | Q28295681 | ||
Choreography of the DNA damage response: spatiotemporal relationships among checkpoint and repair proteins | Q29615270 | ||
Segmental duplications arise from Pol32-dependent repair of broken forks through two alternative replication-based mechanisms | Q33367138 | ||
Hypermutability of damaged single-strand DNA formed at double-strand breaks and uncapped telomeres in yeast Saccharomyces cerevisiae. | Q33385991 | ||
A microhomology-mediated break-induced replication model for the origin of human copy number variation | Q33404060 | ||
Fusion of nearby inverted repeats by a replication-based mechanism leads to formation of dicentric and acentric chromosomes that cause genome instability in budding yeast | Q33565837 | ||
Replication checkpoint kinase Cds1 regulates Mus81 to preserve genome integrity during replication stress | Q33751481 | ||
Break-induced replication is highly inaccurate | Q33828325 | ||
Position effects in ectopic and allelic mitotic recombination in Saccharomyces cerevisiae | Q33955786 | ||
The prevention of repeat-associated deletions in Saccharomyces cerevisiae by mismatch repair depends on size and origin of deletions. | Q33968093 | ||
Observation and prediction of recurrent human translocations mediated by NAHR between nonhomologous chromosomes | Q34452268 | ||
Chromosome break-induced DNA replication leads to nonreciprocal translocations and telomere capture. | Q34605651 | ||
Cell-type-specific replication initiation programs set fragility of the FRA3B fragile site | Q34626544 | ||
An oncogene-induced DNA damage model for cancer development | Q34759268 | ||
RAD51- and MRE11-dependent reassembly of uncoupled CMG helicase complex at collapsed replication forks | Q35011618 | ||
A DNA replication-arrest site RTS1 regulates imprinting by determining the direction of replication at mat1 in S. pombe | Q35080713 | ||
Inverted genomic segments and complex triplication rearrangements are mediated by inverted repeats in the human genome | Q35606491 | ||
Complex human chromosomal and genomic rearrangements | Q35731269 | ||
DNA replication through hard-to-replicate sites, including both highly transcribed RNA Pol II and Pol III genes, requires the S. pombe Pfh1 helicase. | Q35860068 | ||
Checkpoint activation regulates mutagenic translesion synthesis | Q35963618 | ||
Mechanisms for recurrent and complex human genomic rearrangements | Q36044446 | ||
Temporal separation of replication and recombination requires the intra-S checkpoint. | Q36321415 | ||
Visualization of eukaryotic DNA mismatch repair reveals distinct recognition and repair intermediates. | Q36338245 | ||
Replication stress induces tumor-like microdeletions in FHIT/FRA3B | Q36423451 | ||
Cleavage of stalled forks by fission yeast Mus81/Eme1 in absence of DNA replication checkpoint | Q36438748 | ||
Replication slippage between distant short repeats in Saccharomyces cerevisiae depends on the direction of replication and the RAD50 and RAD52 genes. | Q36555338 | ||
Mus81 is essential for sister chromatid recombination at broken replication forks | Q36642712 | ||
Replication fork stalling at natural impediments | Q36755308 | ||
Arrested replication fork processing: interplay between checkpoints and recombination | Q36782513 | ||
Rtf1-mediated eukaryotic site-specific replication termination | Q36873696 | ||
Nucleotide deficiency promotes genomic instability in early stages of cancer development | Q37087191 | ||
Break-induced replication: what is it and what is it for? | Q37138963 | ||
Mechanisms of polar arrest of a replication fork | Q37418139 | ||
P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P6216 | copyright status | copyrighted | Q50423863 |
P433 | issue | 10 | |
P921 | main subject | mitotic recombination-dependent replication fork processing | Q21095321 |
regulation of mitotic recombination involved in replication fork processing | Q22244291 | ||
P304 | page(s) | e1002976 | |
P577 | publication date | 2012-10-18 | |
P1433 | published in | PLOS Genetics | Q1893441 |
P1476 | title | Recovery of arrested replication forks by homologous recombination is error-prone | |
P478 | volume | 8 |
Q47434394 | A Molecular Toolbox to Engineer Site-Specific DNA Replication Perturbation. |
Q39378331 | Break-induced replication links microsatellite expansion to complex genome rearrangements |
Q47121267 | Cdc7-Dbf4-mediated phosphorylation of HSP90-S164 stabilizes HSP90-HCLK2-MRN complex to enhance ATR/ATM signaling that overcomes replication stress in cancer |
Q41190098 | Characterization of 26 deletion CNVs reveals the frequent occurrence of micro-mutations within the breakpoint-flanking regions and frequent repair of double-strand breaks by templated insertions derived from remote genomic regions |
Q90424392 | Chromatin remodeler Fft3 plays a dual role at blocked DNA replication forks |
Q38197136 | Chromosome replication origins: do we really need them? |
Q39669947 | Chronic p53-independent p21 expression causes genomic instability by deregulating replication licensing |
Q91800201 | Defects in the GINS complex increase the instability of repetitive sequences via a recombination-dependent mechanism |
Q93127935 | EXD2 Protects Stressed Replication Forks and Is Required for Cell Viability in the Absence of BRCA1/2 |
Q61811594 | Factors affecting template switch recombination associated with restarted DNA replication |
Q92189777 | Fam208a orchestrates interaction protein network essential for early embryonic development and cell division |
Q28538056 | Fission yeast Rad52 phosphorylation restrains error prone recombination pathways |
Q28264035 | Genetic instability in budding and fission yeast-sources and mechanisms |
Q90482844 | Histone deposition promotes recombination-dependent replication at arrested forks |
Q38223677 | Homologous recombination as a replication fork escort: fork-protection and recovery. |
Q45875729 | Host factors that promote retrotransposon integration are similar in distantly related eukaryotes. |
Q42719848 | Hyperactive Cdc2 kinase interferes with the response to broken replication forks by trapping S.pombe Crb2 in its mitotic T215 phosphorylated state. |
Q48590181 | Increased activity of both CDK1 and CDK2 is necessary for the combinatorial activity of WEE1 inhibition and cytarabine. |
Q33736829 | Is homologous recombination really an error-free process? |
Q37696358 | Loss of Caenorhabditis elegans BRCA1 promotes genome stability during replication in smc-5 mutants |
Q37148499 | Loss of heterozygosity preferentially occurs in early replicating regions in cancer genomes |
Q35712405 | Massive interstitial copy-neutral loss-of-heterozygosity as evidence for cancer being a disease of the DNA-damage response |
Q26765934 | Mechanisms underlying structural variant formation in genomic disorders |
Q34294018 | Mutation frequency dynamics in HPRT locus in culture-adapted human embryonic stem cells and induced pluripotent stem cells correspond to their differentiated counterparts |
Q28268105 | Polymerase δ replicates both strands after homologous recombination-dependent fork restart |
Q58701167 | Preserving replication fork integrity and competence via the homologous recombination pathway |
Q38214443 | Quality control of homologous recombination |
Q39893522 | Recombination occurs within minutes of replication blockage by RTS1 producing restarted forks that are prone to collapse |
Q39206989 | Recombination-restarted replication makes inverted chromosome fusions at inverted repeats. |
Q35635445 | Regulation of recombination at yeast nuclear pores controls repair and triplet repeat stability. |
Q35680075 | Repeat instability during DNA repair: Insights from model systems |
Q37271098 | Replication fork collapse is a major cause of the high mutation frequency at three-base lesion clusters |
Q37290645 | Replicative mechanisms for CNV formation are error prone |
Q39392243 | Role of recombination and replication fork restart in repeat instability |
Q36997781 | Role of the double-strand break repair pathway in the maintenance of genomic stability. |
Q92637329 | Schizosaccharomyces pombe Assays to Study Mitotic Recombination Outcomes |
Q34973379 | Size of gene specific inverted repeat--dependent gene deletion In Saccharomyces cerevisiae. |
Q38615461 | Stalled replication forks generate a distinct mutational signature in yeast. |
Q38619238 | Systematic Identification of Determinants for Single Strand Annealing Mediated Deletion Formation in Saccharomyces cerevisiae |
Q26830817 | The FHIT gene product: tumor suppressor and genome "caretaker" |
Q34337447 | The chromatin assembly factor 1 promotes Rad51-dependent template switches at replication forks by counteracting D-loop disassembly by the RecQ-type helicase Rqh1 |
Q47107185 | The end-joining factor Ku acts in the end-resection of double strand break-free arrested replication forks. |
Q33822400 | The extent of error-prone replication restart by homologous recombination is controlled by Exo1 and checkpoint proteins. |
Q101409898 | The nuclear pore primes recombination-dependent DNA synthesis at arrested forks by promoting SUMO removal |
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