scholarly article | Q13442814 |
P819 | ADS bibcode | 2013PLoSO...872155V |
P356 | DOI | 10.1371/JOURNAL.PONE.0072155 |
P932 | PMC publication ID | 3756074 |
P698 | PubMed publication ID | 24015214 |
P5875 | ResearchGate publication ID | 256470609 |
P50 | author | Benjamin Vandendriessche | Q49092829 |
Johannes-Peter Stasch | Q66360867 | ||
Peter Vandenabeele | Q30112482 | ||
P2093 | author name string | Peter Brouckaert | |
Vera Goossens | |||
Anje Cauwels | |||
Elke Rogge | |||
P2860 | cites work | Duration of hypotension before initiation of effective antimicrobial therapy is the critical determinant of survival in human septic shock | Q83177700 |
Nitric oxide-independent vasodilator rescues heme-oxidized soluble guanylate cyclase from proteasomal degradation | Q39845753 | ||
Uncoupling of biological oscillators: a complementary hypothesis concerning the pathogenesis of multiple organ dysfunction syndrome | Q41010995 | ||
Protection from lethal apoptosis in lipopolysaccharide-induced acute lung injury in mice by a caspase inhibitor | Q42048652 | ||
Smooth muscle-specific deletion of nitric oxide-sensitive guanylyl cyclase is sufficient to induce hypertension in mice | Q43197344 | ||
The expression of soluble guanylate cyclase in the vasculature of rat skeletal muscle | Q43222467 | ||
Infusion of Escherichia coli lipopolysaccharide toxin in rats produces an early and severe impairment of baroreflex function in absence of blood pressure changes. | Q43797224 | ||
Caspase inhibition causes hyperacute tumor necrosis factor-induced shock via oxidative stress and phospholipase A2. | Q44372580 | ||
A role for interleukin 1 in the in vivo actions of tumor necrosis factor | Q44714244 | ||
Fractal rigidity by enhanced sympatho-vagal antagonism in heartbeat interval dynamics elicited by central application of corticotropin-releasing factor in mice. | Q48744796 | ||
Drotrecogin alfa (activated) in adults with septic shock. | Q55056165 | ||
Autonomic cardiovascular control in conscious mice | Q73194342 | ||
Mediator modulation therapy of severe sepsis and septic shock: does it work? | Q75225181 | ||
NO-independent stimulators and activators of soluble guanylate cyclase: discovery and therapeutic potential | Q24656131 | ||
PhysioBank, PhysioToolkit, and PhysioNet : Components of a New Research Resource for Complex Physiologic Signals | Q28020278 | ||
Spare guanylyl cyclase NO receptors ensure high NO sensitivity in the vascular system | Q28513504 | ||
The epidemiology of sepsis in the United States from 1979 through 2000 | Q29547760 | ||
NO-independent regulatory site on soluble guanylate cyclase | Q32165040 | ||
Nitrite protects against morbidity and mortality associated with TNF- or LPS-induced shock in a soluble guanylate cyclase-dependent manner | Q33590497 | ||
Cyclic GMP phosphodiesterase-5: target of sildenafil | Q33630333 | ||
Guanylyl cyclases and signaling by cyclic GMP. | Q33917038 | ||
Multiple organ dysfunction syndrome: exploring the paradigm of complex nonlinear systems | Q33988265 | ||
Reactive oxygen species and small-conductance calcium-dependent potassium channels are key mediators of inflammation-induced hypotension and shock | Q34063569 | ||
Quantification of scaling exponents and crossover phenomena in nonstationary heartbeat time series | Q34089528 | ||
Myocardial dysfunction in septic shock: Part I. Clinical manifestation of cardiovascular dysfunction | Q34293378 | ||
Autonomic dysfunction in the ICU patient | Q34503014 | ||
Pharmacokinetics, pharmacodynamics, tolerability, and safety of the soluble guanylate cyclase activator cinaciguat (BAY 58-2667) in healthy male volunteers | Q34823612 | ||
NO-independent, haem-dependent soluble guanylate cyclase stimulators | Q34906036 | ||
Targeting the heme-oxidized nitric oxide receptor for selective vasodilatation of diseased blood vessels | Q35009484 | ||
NO- and haem-independent activation of soluble guanylyl cyclase: molecular basis and cardiovascular implications of a new pharmacological principle | Q35044464 | ||
Novel strategies for the treatment of sepsis | Q35118750 | ||
Physiology and pathophysiology of vascular signaling controlled by guanosine 3',5'-cyclic monophosphate-dependent protein kinase [corrected]. | Q35575494 | ||
Interpretation of normalized spectral heart rate variability indices in sleep research: a critical review | Q35995533 | ||
Severe sepsis and septic shock: review of the literature and emergency department management guidelines. | Q36510126 | ||
Survival of TNF toxicity: dependence on caspases and NO. | Q36744895 | ||
sGC(alpha)1(beta)1 attenuates cardiac dysfunction and mortality in murine inflammatory shock models. | Q37298332 | ||
NO- and haem-independent soluble guanylate cyclase activators | Q37352787 | ||
P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P6216 | copyright status | copyrighted | Q50423863 |
P433 | issue | 8 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | endotoxic shock | Q11291420 |
P304 | page(s) | e72155 | |
P577 | publication date | 2013-08-28 | |
P1433 | published in | PLOS One | Q564954 |
P1476 | title | The soluble guanylate cyclase activator BAY 58-2667 protects against morbidity and mortality in endotoxic shock by recoupling organ systems | |
P478 | volume | 8 |
Q36424922 | Cardiovascular and pharmacological implications of haem-deficient NO-unresponsive soluble guanylate cyclase knock-in mice |
Q34538482 | Development of Therapeutics That Induce Mitochondrial Biogenesis for the Treatment of Acute and Chronic Degenerative Diseases. |
Q27021649 | Extending the translational potential of targeting NO/cGMP-regulated pathways in the CVS |
Q51687813 | Guanylyl cyclase activation reverses resistive breathing-induced lung injury and inflammation. |
Q49047822 | Influence of cinaciguat on gastrointestinal motility in apo-sGC mice. |
Q42380504 | Inhibition of soluble guanylyl cyclase by small molecules targeting the catalytic domain |
Q27021987 | Nitrate/Nitrite as Critical Mediators to Limit Oxidative Injury and Inflammation |
Q36430759 | Progesterone receptor membrane component-1 regulates hepcidin biosynthesis |
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