| P50 | author | Pier Paolo Pandolfi | Q7191735 |
| | Peter Hemmerich | Q84263864 |
| P2093 | author name string | Shamci Monajembashi | |
| | Stefanie Weidtkamp-Peters | |
| | Paolo Salomoni | |
| | Sandra Münch | |
| | Karolin Klement | |
| | Paulius Grigaravicius | |
| | Klaus Weißhart | |
| P2860 | cites work | PML regulates p53 acetylation and premature senescence induced by oncogenic Ras | Q22254666 |
| | Regulation of p53 activity in nuclear bodies by a specific PML isoform | Q24290520 |
| | Regulation and localization of the Bloom syndrome protein in response to DNA damage | Q24291133 |
| | Telomere shortening triggers senescence of human cells through a pathway involving ATM, p53, and p21(CIP1), but not p16(INK4a) | Q24293520 |
| | PML regulates p53 stability by sequestering Mdm2 to the nucleolus | Q24296743 |
| | Evidence for a role of the cellular ND10 protein PML in mediating intrinsic immunity against human cytomegalovirus infections | Q24298226 |
| | MageA2 restrains cellular senescence by targeting the function of PMLIV/p53 axis at the PML-NBs | Q24298306 |
| | PML promotes MHC class II gene expression by stabilizing the class II transactivator | Q24299467 |
| | PML colocalizes with and stabilizes the DNA damage response protein TopBP1 | Q24303540 |
| | Promyelocytic leukemia protein interacts with werner syndrome helicase and regulates double-strand break repair in γ-irradiation-induced DNA damage responses | Q24305398 |
| | ATR kinase activity regulates the intranuclear translocation of ATR and RPA following ionizing radiation | Q24305520 |
| | The promyelocytic leukemia protein PML regulates c-Jun function in response to DNA damage | Q24337110 |
| | A biomarker that identifies senescent human cells in culture and in aging skin in vivo | Q24562644 |
| | PML is critical for ND10 formation and recruits the PML-interacting protein daxx to this nuclear structure when modified by SUMO-1 | Q24670576 |
| | Colocalization of multiple DNA double-strand breaks at a single Rad52 repair centre | Q27940019 |
| | The function of PML in p53-dependent apoptosis | Q28140732 |
| | THE LIMITED IN VITRO LIFETIME OF HUMAN DIPLOID CELL STRAINS | Q28202269 |
| | PML interaction with p53 and its role in apoptosis and replicative senescence | Q28204830 |
| | Accumulation of senescent cells in mitotic tissue of aging primates | Q28274849 |
| | Cytoplasmic PML function in TGF-beta signalling | Q28281152 |
| | Computational analysis of the number, area and density of gamma-H2AX foci in breast cancer cells exposed to (111)In-DTPA-hEGF or gamma-rays using Image-J software | Q28365506 |
| | PML is essential for multiple apoptotic pathways | Q28508348 |
| | PML is a direct p53 target that modulates p53 effector functions | Q28586691 |
| | Nuclear foci of mammalian recombination proteins are located at single-stranded DNA regions formed after DNA damage | Q28610023 |
| | Accumulation of annexin A5 at the nuclear envelope is a biomarker of cellular aging. | Q54502242 |
| | Stress-induced premature senescence or stress-induced senescence-like phenotype: one in vivo reality, two possible definitions? | Q55489204 |
| | DNA damage response and cellular senescence in tissues of aging mice | Q60284563 |
| | Delayed kinetics of DNA double-strand break processing in normal and pathological aging | Q64387738 |
| | PML NBs associate with the hMre11 complex and p53 at sites of irradiation induced DNA damage | Q64387887 |
| | Cell-cycle regulation of DNA damage-induced expression of the suppressor gene PML | Q73941080 |
| | Promyelocytic leukemia nuclear bodies are predetermined processing sites for damaged DNA | Q79988209 |
| | DNA damage foci at dysfunctional telomeres | Q29614811 |
| | A DNA damage checkpoint response in telomere-initiated senescence | Q29615919 |
| | Promyelocytic leukemia nuclear bodies behave as DNA damage sensors whose response to DNA double-strand breaks is regulated by NBS1 and the kinases ATM, Chk2, and ATR. | Q30480527 |
| | Functional connection between Rad51 and PML in homology-directed repair | Q31037336 |
| | DNA-SCARS: distinct nuclear structures that sustain damage-induced senescence growth arrest and inflammatory cytokine secretion | Q33759285 |
| | DNA damage-dependent nuclear dynamics of the Mre11 complex | Q33975168 |
| | Deconstructing PML-induced premature senescence | Q34089507 |
| | Aging, cellular senescence, and cancer | Q34199776 |
| | The essence of senescence | Q34288983 |
| | PML nuclear bodies: dynamic sensors of DNA damage and cellular stress | Q34346231 |
| | ATM signaling facilitates repair of DNA double-strand breaks associated with heterochromatin | Q34801409 |
| | PML a target of translocations in APL is a regulator of cellular senescence. | Q34926594 |
| | PML is induced by oncogenic ras and promotes premature senescence | Q35201220 |
| | Heterogeneous nuclear expression of the promyelocytic leukemia (PML) protein in normal and neoplastic human tissues. | Q35782722 |
| | Promyelocytic leukemia nuclear bodies support a late step in DNA double-strand break repair by homologous recombination | Q35914635 |
| | Human cell senescence as a DNA damage response. | Q35989854 |
| | Role of promyelocytic leukemia (PML) protein in tumor suppression | Q36376155 |
| | ATR signaling can drive cells into senescence in the absence of DNA breaks | Q36411149 |
| | The DNA helicase activity of BLM is necessary for the correction of the genomic instability of bloom syndrome cells | Q36845106 |
| | Activation of the cellular DNA damage response in the absence of DNA lesions | Q36953953 |
| | Role of nuclear bodies in apoptosis signalling | Q37235467 |
| | Regulation of apoptosis by PML and the PML-NBs. | Q37302759 |
| | The repair and signaling responses to DNA double-strand breaks | Q38110631 |
| | Altered distribution of the promyelocytic leukemia-associated protein is associated with cellular senescence. | Q39448587 |
| | Regulation of E2Fs and senescence by PML nuclear bodies. | Q39612304 |
| | PML enhances the regulation of p53 by CK1 in response to DNA damage | Q40017147 |
| | Evidence for the receipt of DNA damage stimuli by PML nuclear domains | Q40186162 |
| | Senescing human cells and ageing mice accumulate DNA lesions with unrepairable double-strand breaks | Q40593721 |
| | DNA damage-induced translocation of the Werner helicase is regulated by acetylation | Q40696914 |
| | Human papillomavirus oncoprotein E7 targets the promyelocytic leukemia protein and circumvents cellular senescence via the Rb and p53 tumor suppressor pathways | Q40890409 |
| | A role for PML and the nuclear body in genomic stability | Q40905918 |
| | Promyelocytic leukemia (PML) nuclear bodies are protein structures that do not accumulate RNA | Q42258547 |
| | High resolution imaging of changes in the structure and spatial organization of chromatin, gamma-H2A.X and the MRN complex within etoposide-induced DNA repair foci | Q43255343 |
| | The promyelocytic leukemia protein protects p53 from Mdm2-mediated inhibition and degradation | Q44480812 |
| | PML is required for homeodomain-interacting protein kinase 2 (HIPK2)-mediated p53 phosphorylation and cell cycle arrest but is dispensable for the formation of HIPK domains. | Q44542308 |
| | Fixation-induced redistribution of hyperphosphorylated RNA polymerase II in the nucleus of human cells | Q44849036 |
| | Human fibroblasts require the Rb family of tumor suppressors, but not p53, for PML-induced senescence. | Q47277811 |
| | Disappearance of the telomere dysfunction-induced stress response in fully senescent cells | Q47407109 |
| | Dynamics of DNA double-strand breaks revealed by clustering of damaged chromosome domains. | Q51830570 |
| | Age- and oxidative stress-induced DNA damage in Drosophila intestinal stem cells as marked by Gamma-H2AX. | Q52734949 |
| | DNA damage response activation in mouse embryonic fibroblasts undergoing replicative senescence and following spontaneous immortalization. | Q53316064 |
| | Cellular senescence in aging primates. | Q53638045 |
| P4510 | describes a project that uses | ImageJ | Q1659584 |
| P433 | issue | 10 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | DNA damage | Q5205747 |
| P304 | page(s) | 1733-1746 | |
| P577 | publication date | 2014-03-10 | |
| P1433 | published in | Molecular and Cellular Biology | Q3319478 |
| P1476 | title | The tumor suppressor PML specifically accumulates at RPA/Rad51-containing DNA damage repair foci but is nonessential for DNA damage-induced fibroblast senescence | |
| P478 | volume | 34 | |