| scholarly article | Q13442814 |
| P819 | ADS bibcode | 2014PLoSO...9j4741K |
| P356 | DOI | 10.1371/JOURNAL.PONE.0104741 |
| P932 | PMC publication ID | 4134227 |
| P698 | PubMed publication ID | 25127469 |
| P5875 | ResearchGate publication ID | 264831068 |
| P50 | author | Hajime Kono | Q64026836 |
| P2093 | author name string | Hiroshi Kataoka | |
| Yoshitaka Kimura | |||
| Kenneth L Rock | |||
| Zubin Patel | |||
| P2860 | cites work | Mechanisms of acetaminophen-induced liver necrosis | Q24629891 |
| I-PLA(2) activation during apoptosis promotes the exposure of membrane lysophosphatidylcholine leading to binding by natural immunoglobulin M antibodies and complement activation | Q24673242 | ||
| Apoptosis, oncosis, and necrosis. An overview of cell death | Q24684007 | ||
| Induction of intestinal inflammation in mouse by activation of proteinase-activated receptor-2 | Q24685396 | ||
| Release of chromatin protein HMGB1 by necrotic cells triggers inflammation | Q28131815 | ||
| The danger model: a renewed sense of self | Q28214633 | ||
| Cryopyrin activates the inflammasome in response to toxins and ATP | Q28291313 | ||
| Mincle is an ITAM-coupled activating receptor that senses damaged cells | Q28506757 | ||
| Altered cytokine production in mice lacking P2X(7) receptors | Q28586061 | ||
| The macrophage-inducible C-type lectin, mincle, is an essential component of the innate immune response to Candida albicans | Q28617622 | ||
| Tolerance, danger, and the extended family | Q29547532 | ||
| DAMPs, PAMPs and alarmins: all we need to know about danger | Q29547535 | ||
| Complement: a key system for immune surveillance and homeostasis | Q29615484 | ||
| How dying cells alert the immune system to danger | Q29615501 | ||
| Generation of C5a in the absence of C3: a new complement activation pathway | Q58523865 | ||
| Cellular death and necrosis: Chemical, physical and morphologic changes in rat liver | Q60361637 | ||
| Changes in cellular levels of ATP and its catabolites in ischemic rat liver | Q70402945 | ||
| Effect of acetaminophen administration on hepatic glutathione compartmentation and mitochondrial energy metabolism in the rat | Q71819976 | ||
| Nucleotides released by apoptotic cells act as a find-me signal to promote phagocytic clearance | Q30436506 | ||
| The chemokine receptors CCR2 and CX3CR1 mediate monocyte/macrophage trafficking in kidney ischemia-reperfusion injury | Q30439547 | ||
| Studies of group B streptococcal infection in mice deficient in complement component C3 or C4 demonstrate an essential role for complement in both innate and acquired immunity | Q33703237 | ||
| Uric acid promotes an acute inflammatory response to sterile cell death in mice | Q33882855 | ||
| Intravascular Danger Signals Guide Neutrophils to Sites of Sterile Inflammation | Q34143696 | ||
| The inflammatory response to cell death | Q34718276 | ||
| Innate and adaptive immune responses to cell death. | Q35205885 | ||
| Role of C5a in inflammatory responses | Q36072464 | ||
| Identification of the target self-antigens in reperfusion injury | Q36227933 | ||
| The phlogistic role of C3 leukotactic fragments in myocardial infarcts of rats | Q36270882 | ||
| Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement | Q36366602 | ||
| The P2X7 receptor: a key player in IL-1 processing and release | Q36425273 | ||
| The role of complement in danger sensing and transmission | Q36499763 | ||
| Protease-activated receptors: novel PARtners in innate immunity. | Q36989144 | ||
| Complement localization and mediation of ischemic injury in baboon myocardium | Q37024840 | ||
| Identification and characterization of infiltrating macrophages in acetaminophen-induced liver injury | Q37038993 | ||
| Acetaminophen-induced hepatotoxicity in mice is dependent on Tlr9 and the Nalp3 inflammasome. | Q37073714 | ||
| Role of protease-activated receptors in inflammatory responses, innate and adaptive immunity | Q37112308 | ||
| Complement-mediated ischemia-reperfusion injury: lessons learned from animal and clinical studies | Q37498110 | ||
| The sterile inflammatory response | Q37716003 | ||
| Identification of the cellular sensor that stimulates the inflammatory response to sterile cell death. | Q39729219 | ||
| Identification of a key pathway required for the sterile inflammatory response triggered by dying cells | Q40119160 | ||
| Complement Ligand-Receptor Interactions that Mediate Biological Responses | Q40119173 | ||
| Protease-activated receptor 2 mediates eosinophil infiltration and hyperreactivity in allergic inflammation of the airway | Q40695518 | ||
| A clinical and experimental study of the function of neutrophils in the inflammatory response | Q41814993 | ||
| A role of IgM antibodies in monosodium urate crystal formation and associated adjuvanticity | Q42105873 | ||
| The DNA sugar backbone 2' deoxyribose determines toll-like receptor 9 activation | Q42525772 | ||
| Cutting edge: critical role for mesothelial cells in necrosis-induced inflammation through the recognition of IL-1 alpha released from dying cells | Q42584586 | ||
| Involvement of mitochondrial permeability transition in acetaminophen-induced liver injury in mice | Q42644118 | ||
| Proteinase-activated receptor-2-induced colonic inflammation in mice: possible involvement of afferent neurons, nitric oxide, and paracellular permeability | Q44394560 | ||
| PAR2 activation alters colonic paracellular permeability in mice via IFN-gamma-dependent and -independent pathways | Q44934665 | ||
| P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
| P6216 | copyright status | copyrighted | Q50423863 |
| P433 | issue | 8 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | inflammation | Q101991 |
| cell death | Q2383867 | ||
| P304 | page(s) | e104741 | |
| P577 | publication date | 2014-08-15 | |
| P1433 | published in | PLOS One | Q564954 |
| P1476 | title | Evaluation of the contribution of multiple DAMPs and DAMP receptors in cell death-induced sterile inflammatory responses | |
| P478 | volume | 9 |
| Q90292217 | ATP-Nlrp3 Inflammasome-Complement Cascade Axis in Sterile Brain Inflammation in Psychiatric Patients and its Impact on Stem Cell Trafficking |
| Q91703599 | Apoptosis and necroptosis in the liver: a matter of life and death |
| Q26747619 | Biomaterial Applications in Cell-Based Therapy in Experimental Stroke |
| Q35609496 | Biomaterial-mediated modification of the local inflammatory environment. |
| Q90055176 | C-Type Lectin-Like Receptors: Head or Tail in Cell Death Immunity |
| Q40641396 | Cell death and inflammation: the case for IL-1 family cytokines as the canonical DAMPs of the immune system |
| Q60949506 | De-Ritis Ratio Improves Long-Term Risk Prediction after Acute Myocardial Infarction |
| Q40327548 | Exacerbated and prolonged inflammation impairs wound healing and increases scarring |
| Q39269625 | Fueling Inflamm-Aging through Mitochondrial Dysfunction: Mechanisms and Molecular Targets. |
| Q38521274 | Inflammasome activation and function in liver disease |
| Q28595434 | Inflammation-a Critical Appreciation of the Role of Myeloid Cells |
| Q90638952 | Liver Immune Cells Release Type 1 Interferon Due to DNA Sensing and Amplify Liver Injury from Acetaminophen Overdose |
| Q54261138 | Mitochondrial DNA as an inflammatory mediator in cardiovascular diseases. |
| Q58745474 | NLRP3 Inflammasome and IL-33: Novel Players in Sterile Liver Inflammation |
| Q38645385 | Potential Roles for C1 Inhibitor in Transplantation |
| Q39027620 | Role of the inflammasome in acetaminophen-induced liver injury and acute liver failure |
| Q26747404 | STAT3, a Key Parameter of Cytokine-Driven Tissue Protection during Sterile Inflammation - the Case of Experimental Acetaminophen (Paracetamol)-Induced Liver Damage |
| Q33847610 | The Impact of Sterile Inflammation in Acute Liver Injury |
| Q38555406 | The danger model: questioning an unconvincing theory |
| Q38856385 | The early wound signals |
| Q91907252 | The innate immune response to allotransplants: mechanisms and therapeutic potentials |
| Q64259768 | ccf-mtDNA as a Potential Link Between the Brain and Immune System in Neuro-Immunological Disorders |
Search more.