Loss of F-box only protein 2 (Fbxo2) disrupts levels and localization of select NMDA receptor subunits, and promotes aberrant synaptic connectivity.

scientific article

Loss of F-box only protein 2 (Fbxo2) disrupts levels and localization of select NMDA receptor subunits, and promotes aberrant synaptic connectivity. is …
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scholarly articleQ13442814

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P356DOI10.1523/JNEUROSCI.3013-14.2015
P932PMC publication ID4397610
P698PubMed publication ID25878288

P50authorJohannes W HellQ46677974
Shannon J MooreQ93020816
P2093author name stringYuan Lu
Geoffrey G Murphy
Henry Paulson
Graham Atkin
Rick F Nelson
William Tennant
Jack Hunt
Gautam Rajpal
Nathan Tipper
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P4510describes a project that usesImageJQ1659584
P433issue15
P407language of work or nameEnglishQ1860
P921main subjectnmdAQ62663735
P304page(s)6165-6178
P577publication date2015-04-01
P1433published inJournal of NeuroscienceQ1709864
P1476titleLoss of F-box only protein 2 (Fbxo2) disrupts levels and localization of select NMDA receptor subunits, and promotes aberrant synaptic connectivity
P478volume35

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cites work (P2860)
Q59355883Epstein-Barr virus activates F-box protein FBXO2 to limit viral infectivity by targeting glycoprotein B for degradation
Q91282455Fbxo2VHC mouse and embryonic stem cell reporter lines delineate in vitro-generated inner ear sensory epithelia cells and enable otic lineage selection and Cre-recombination
Q48144297Molecular mechanisms underlying monosynaptic sensory-motor circuit development in the spinal cord.
Q90929165Pathological priming causes developmental gene network heterochronicity in autistic subject-derived neurons
Q64228390Sugar-Recognizing Ubiquitin Ligases: Action Mechanisms and Physiology
Q89493873The Acute Effects of Amyloid-Beta1-42 on Glutamatergic Receptor and Transporter Expression in the Mouse Hippocampus
Q33361879The Role of Proteases in Hippocampal Synaptic Plasticity: Putting Together Small Pieces of a Complex Puzzle
Q26777280Ubiquitin-dependent trafficking and turnover of ionotropic glutamate receptors
Q37021737nArgBP2 regulates excitatory synapse formation by controlling dendritic spine morphology

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