scholarly article | Q13442814 |
P819 | ADS bibcode | 2015PLoSO..1021246P |
P356 | DOI | 10.1371/JOURNAL.PONE.0121246 |
P932 | PMC publication ID | 4364993 |
P698 | PubMed publication ID | 25785991 |
P5875 | ResearchGate publication ID | 273784114 |
P50 | author | Juan C Osorio | Q43176323 |
Chun Geun Lee | Q56810022 | ||
Augustine M K Choi | Q114342087 | ||
P2093 | author name string | Jack A Elias | |
Ying Shi | |||
Danielle Morse | |||
Ivan O Rosas | |||
Jin Woo Song | |||
Souheil El-Chemaly | |||
Kenji Mizumura | |||
Sarah G Chu | |||
Avignat S Patel | |||
P2860 | cites work | Mitochondria in cell death | Q50550781 |
Insufficient autophagy in idiopathic pulmonary fibrosis. | Q53138134 | ||
Blocking IL-17A promotes the resolution of pulmonary inflammation and fibrosis via TGF-beta1-dependent and -independent mechanisms | Q84762346 | ||
ROS-induced mitochondrial depolarization initiates PARK2/PARKIN-dependent mitochondrial degradation by autophagy | Q84788113 | ||
Proteasomes in lungs from organ donors and patients with end-stage pulmonary diseases | Q95558988 | ||
Autophagy in idiopathic pulmonary fibrosis | Q21134133 | ||
PINK1 is selectively stabilized on impaired mitochondria to activate Parkin | Q21145802 | ||
The mitochondrial intramembrane protease PARL cleaves human Pink1 to regulate Pink1 trafficking | Q24297960 | ||
Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism | Q24309753 | ||
Parkin is recruited selectively to impaired mitochondria and promotes their autophagy | Q24317471 | ||
Loss of PINK1 function promotes mitophagy through effects on oxidative stress and mitochondrial fission | Q24320327 | ||
PINK1 is necessary for long term survival and mitochondrial function in human dopaminergic neurons | Q24322788 | ||
Hereditary early-onset Parkinson's disease caused by mutations in PINK1 | Q24337084 | ||
Innate immune activation through Nalp3 inflammasome sensing of asbestos and silica | Q24649598 | ||
Selective degradation of mitochondria by mitophagy | Q24658163 | ||
Mechanisms of mitophagy | Q27310261 | ||
Parkinson phenotype in aged PINK1-deficient mice is accompanied by progressive mitochondrial dysfunction in absence of neurodegeneration | Q27348827 | ||
Oxidative stress and glutathione in TGF-beta-mediated fibrogenesis | Q28393454 | ||
p53-TIGAR axis attenuates mitophagy to exacerbate cardiac damage after ischemia | Q28509575 | ||
Impaired dopamine release and synaptic plasticity in the striatum of PINK1-deficient mice | Q28513036 | ||
A mitochondrial paradigm of metabolic and degenerative diseases, aging, and cancer: a dawn for evolutionary medicine | Q29547303 | ||
Mitochondrial dynamics--fusion, fission, movement, and mitophagy--in neurodegenerative diseases | Q29615646 | ||
Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin | Q29615684 | ||
PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy | Q29616005 | ||
Cytostatic and apoptotic actions of TGF-beta in homeostasis and cancer | Q29616379 | ||
Oxidative phosphorylation at the fin de siècle | Q33538525 | ||
Statins and pulmonary fibrosis: the potential role of NLRP3 inflammasome activation | Q34635121 | ||
PINK1 deficiency impairs mitochondrial homeostasis and promotes lung fibrosis. | Q35056685 | ||
A key role for NOX4 in epithelial cell death during development of lung fibrosis. | Q35185363 | ||
Mitochondrial Rac1 GTPase import and electron transfer from cytochrome c are required for pulmonary fibrosis. | Q35728305 | ||
Role of PINK1 binding to the TOM complex and alternate intracellular membranes in recruitment and activation of the E3 ligase Parkin | Q35783335 | ||
Spatial parkin translocation and degradation of damaged mitochondria via mitophagy in live cortical neurons | Q35855944 | ||
Mitochondrial processing peptidase regulates PINK1 processing, import and Parkin recruitment | Q35874914 | ||
Evolving concepts of apoptosis in idiopathic pulmonary fibrosis | Q36493966 | ||
Mitochondrial alterations in Alzheimer's disease | Q36549402 | ||
The interplay between mitochondrial dynamics and mitophagy | Q37815475 | ||
Altered cleavage and localization of PINK1 to aggresomes in the presence of proteasomal stress | Q40262236 | ||
TGF beta1 induces prolonged mitochondrial ROS generation through decreased complex IV activity with senescent arrest in Mv1Lu cells. | Q40462552 | ||
Asbestos-derived reactive oxygen species activate TGF-beta1. | Q40560323 | ||
Mitochondrial reactive oxygen species regulate transforming growth factor-β signaling | Q41921136 | ||
P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P6216 | copyright status | copyrighted | Q50423863 |
P4510 | describes a project that uses | ImageJ | Q1659584 |
P433 | issue | 3 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | pulmonary fibrosis | Q32446 |
P304 | page(s) | e0121246 | |
P577 | publication date | 2015-03-18 | |
P1433 | published in | PLOS One | Q564954 |
P1476 | title | Epithelial cell mitochondrial dysfunction and PINK1 are induced by transforming growth factor-beta1 in pulmonary fibrosis | |
P478 | volume | 10 |
Q47648313 | ATF3 represses PINK1 gene transcription in lung epithelial cells to control mitochondrial homeostasis. |
Q49881851 | Acadian variant of Fanconi syndrome is caused by mitochondrial respiratory chain complex I deficiency due to a non-coding mutation in complex I assembly factor NDUFAF6. |
Q90693925 | Alveolar Epithelial Type II Cells as Drivers of Lung Fibrosis in Idiopathic Pulmonary Fibrosis |
Q38743703 | Autophagy and Obesity-Related Lung Disease |
Q47663797 | Autophagy and inflammation in chronic respiratory disease. |
Q38942187 | Autophagy in Pulmonary Diseases |
Q38793559 | Autophagy: Friend or Foe in Lung Disease? |
Q49797808 | Autophagy: a Lysosome-Dependent Process with Implications in Cellular Redox Homeostasis and Human Disease. |
Q26764812 | Candidate genes of idiopathic pulmonary fibrosis: current evidence and research |
Q89615834 | Cellular stress responses and dysfunctional Mitochondrial-cellular senescence, and therapeutics in chronic respiratory diseases |
Q48231065 | Circulating Mitochondrial DNA as a Mechanism-based, Prognostic Biomarker for Idiopathic Pulmonary Fibrosis |
Q38893443 | Differential regulation of autophagy and mitophagy in pulmonary diseases |
Q47913152 | Emerging therapies for idiopathic pulmonary fibrosis, a progressive age-related disease |
Q55332823 | Emerging therapies for idiopathic pulmonary fibrosis, a progressive age-related disease. |
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Q52716546 | Fibroblast senescence in the pathology of idiopathic pulmonary fibrosis. |
Q100955747 | Growth Hormone-Releasing Hormone in Lung Physiology and Pulmonary Disease |
Q64282237 | Hypercapnic acidosis induces mitochondrial dysfunction and impairs the ability of mesenchymal stem cells to promote distal lung epithelial repair |
Q38599202 | IPF lung fibroblasts have a senescent phenotype |
Q49828907 | Idiopathic Pulmonary Fibrosis: Aging, Mitochondrial Dysfunction, and Cellular Bioenergetics |
Q52578942 | Impact of Transcriptomics on Our Understanding of Pulmonary Fibrosis. |
Q38706646 | Klotho, an antiaging molecule, attenuates oxidant-induced alveolar epithelial cell mtDNA damage and apoptosis |
Q36941974 | Lin28A induces energetic switching to glycolytic metabolism in human embryonic kidney cells |
Q36693106 | Macrophage Akt1 Kinase-Mediated Mitophagy Modulates Apoptosis Resistance and Pulmonary Fibrosis |
Q28387993 | Mitochondria in lung disease |
Q29248145 | Mitochondria in the spotlight of aging and idiopathic pulmonary fibrosis |
Q90626184 | Mitochondrial 8-Oxoguanine DNA Glycosylase Mitigates Alveolar Epithelial Cell PINK1 Deficiency, Mitochondrial DNA Damage, Apoptosis and Lung Fibrosis |
Q48261825 | Mitochondrial Dysfunction in Pulmonary Fibrosis. |
Q92814053 | Mitochondrial Quality Control in Age-Related Pulmonary Fibrosis |
Q90452513 | Mitochondrial Quality Control in COPD and IPF |
Q93113555 | Mitochondrial biology in airway pathogenesis and the role of NRF2 |
Q39191013 | Mitochondrial catalase overexpressed transgenic mice are protected against lung fibrosis in part via preventing alveolar epithelial cell mitochondrial DNA damage |
Q47655604 | Mitochondrial dysfunction and damage associated molecular patterns (DAMPs) in chronic inflammatory diseases |
Q38836727 | Mitochondrial dysfunction in inflammatory responses and cellular senescence: pathogenesis and pharmacological targets for chronic lung diseases |
Q38913254 | Mitochondrial redox system, dynamics, and dysfunction in lung inflammaging and COPD. |
Q36106217 | Mitochondrial uncoupler carbonyl cyanide m-chlorophenylhydrazone induces vasorelaxation without involving KATP channel activation in smooth muscle cells of arteries |
Q92265900 | Mitofusins regulate lipid metabolism to mediate the development of lung fibrosis |
Q90260610 | Oxidative Stress in Pulmonary Fibrosis |
Q38971958 | PINK1 in the limelight: multiple functions of an eclectic protein in human health and disease |
Q58567411 | PINK1-PARK2-mediated mitophagy in COPD and IPF pathogeneses |
Q38627245 | Parkin, PINK1 and DJ1 as possible modulators of mTOR pathway in ganglioglioma |
Q38930521 | SIRT3 deficiency promotes lung fibrosis by augmenting alveolar epithelial cell mitochondrial DNA damage and apoptosis |
Q26795653 | Smoking and interstitial lung diseases |
Q38591535 | Systemic sclerosis-associated fibrosis: an accelerated aging phenotype? |
Q60961513 | TGF-β Upregulated Mitochondria Mass through the SMAD2/3→C/EBPβ→PRMT1 Signal Pathway in Primary Human Lung Fibroblasts |
Q26786969 | The Role of Mitochondrial DNA in Mediating Alveolar Epithelial Cell Apoptosis and Pulmonary Fibrosis |
Q64087497 | The Role of Pathological Aging in Cardiac and Pulmonary Fibrosis |
Q90415361 | The mitochondria in lung fibrosis: friend or foe? |
Q42377386 | The pathophysiological role of mitochondrial oxidative stress in lung diseases |
Q38835390 | Uridine supplementation exerts anti-inflammatory and anti-fibrotic effects in an animal model of pulmonary fibrosis. |
Q39097324 | miR-34a promotes fibrosis in aged lungs by inducing alveolarepithelial dysfunctions |
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