| scholarly article | Q13442814 |
| P2093 | author name string | Bobby R Scott | |
| P2860 | cites work | Strength of the purifying selection against different categories of the point mutations in the coding regions of the human genome | Q82659927 |
| In vivo formation and repair of DNA double-strand breaks after computed tomography examinations | Q24529558 | ||
| Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses | Q24677973 | ||
| Pathways of DNA double-strand break repair during the mammalian cell cycle | Q28608969 | ||
| Repair of x-ray-induced DNA double-strand breaks in specific Not I restriction fragments in human fibroblasts: joining of correct and incorrect ends | Q33643540 | ||
| Inducible response required for repair of low-dose radiation damage in human fibroblasts | Q34069451 | ||
| DNA double-strand break repair signalling: the case of RAD51 post-translational regulation. | Q34929930 | ||
| The bystander effect | Q35174047 | ||
| Mechanism of radiation-induced bystander effects: a unifying model | Q35541188 | ||
| Molecular interaction map of the p53 and Mdm2 logic elements, which control the Off-On switch of p53 in response to DNA damage | Q36113154 | ||
| Five repair pathways in one context: chromatin modification during DNA repair | Q36579359 | ||
| Leukocyte DNA damage after multi-detector row CT: a quantitative biomarker of low-level radiation exposure. | Q36690930 | ||
| Whole-body responses to low-level radiation exposure: new concepts in mammalian radiobiology | Q36767048 | ||
| Stochastic thresholds: a novel explanation of nonlinear dose-response relationships for stochastic radiobiological effects | Q36780513 | ||
| Low dose radiation and intercellular induction of apoptosis: potential implications for the control of oncogenesis | Q37024045 | ||
| What histone code for DNA repair? | Q37093152 | ||
| Low-dose radiation and genotoxic chemicals can protect against stochastic biological effects | Q37132981 | ||
| Radiation-stimulated epigenetic reprogramming of adaptive-response genes in the lung: an evolutionary gift for mounting adaptive protection against lung cancer | Q37225328 | ||
| Cellular responses to DNA double-strand breaks after low-dose gamma-irradiation | Q37259025 | ||
| Intercellular communication of cellular stress monitored by gamma-H2AX induction | Q37376590 | ||
| Response of heterogeneous ribonuclear proteins (hnRNP) to ionising radiation and their involvement in DNA damage repair | Q37447357 | ||
| Higher-order chromatin structure in DSB induction, repair and misrepair | Q37689982 | ||
| Effects of ionizing radiation on cell-to-cell communication. | Q39665206 | ||
| Participation of DNA-PKcs in DSB Repair after Exposure to High- and Low-LET Radiation | Q39672164 | ||
| Hierarchy of nonhomologous end-joining, single-strand annealing and gene conversion at site-directed DNA double-strand breaks | Q39974024 | ||
| Low-dose irradiation of nontransformed cells stimulates the selective removal of precancerous cells via intercellular induction of apoptosis | Q40174169 | ||
| Constitutive nitric oxide acting as a possible intercellular signaling molecule in the initiation of radiation-induced DNA double strand breaks in non-irradiated bystander cells | Q40224665 | ||
| Histone H2AX phosphorylation in normal human cells irradiated with focused ultrasoft X rays: evidence for chromatin movement during repair | Q40261508 | ||
| The loss of gammaH2AX signal is a marker of DNA double strand breaks repair only at low levels of DNA damage | Q40276148 | ||
| No induction of p53 phosphorylation and few focus formation of phosphorylated H2AX suggest efficient repair of DNA damage during chronic low-dose-rate irradiation in human cells. | Q40474093 | ||
| A double-strand break repair defect in ATM-deficient cells contributes to radiosensitivity | Q40595505 | ||
| Bystander and delayed effects after fractionated radiation exposure | Q40696664 | ||
| DNA breakage and repair | Q40849877 | ||
| Chromatin structure and DNA damage repair | Q41189289 | ||
| Evidence that natural selection acts on silent mutation | Q41543626 | ||
| Repair and misrepair of site-specific DNA double-strand breaks by human cell extracts | Q41558547 | ||
| Ionizing radiation-induced, mitochondria-dependent generation of reactive oxygen/nitrogen. | Q43612248 | ||
| A biological-based model that links genomic instability, bystander effects, and adaptive response | Q47899673 | ||
| Inter-individual variation in DNA double-strand break repair in human fibroblasts before and after exposure to low doses of ionizing radiation. | Q53304915 | ||
| A dynamic model for the p53 stress response networks under ion radiation. | Q53590923 | ||
| DNA double-strand break repair by homologous recombination | Q64387340 | ||
| Joining of correct and incorrect DNA double-strand break ends in normal human and ataxia telangiectasia fibroblasts | Q64388739 | ||
| P433 | issue | 4 | |
| P304 | page(s) | 579-601 | |
| P577 | publication date | 2011-02-10 | |
| P1433 | published in | Dose-Response | Q20181483 |
| P1476 | title | Modeling DNA double-strand break repair kinetics as an epiregulated cell-community-wide (epicellcom) response to radiation stress | |
| P478 | volume | 9 |
| Q27334912 | Dynamics between cancer cell subpopulations reveals a model coordinating with both hierarchical and stochastic concepts |
| Q42060141 | First generation stochastic gene episilencing (step1) model and applications to in vitro carcinogen exposure |
| Q35333526 | Radiation-hormesis phenotypes, the related mechanisms and implications for disease prevention and therapy |
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