| scholarly article | Q13442814 |
| P50 | author | Pamela Shaw | Q20716854 |
| Christopher Shaw | Q21469470 | ||
| Kurt J De Vos | Q37379258 | ||
| Declan McLoughlin | Q42306007 | ||
| Andrew Grierson | Q42874347 | ||
| Kwok-Fai Lau | Q58455128 | ||
| P Nigel Leigh | Q64857298 | ||
| P2093 | author name string | Christopher C J Miller | |
| Catherine Manser | |||
| Janet Brownlees | |||
| Steven Ackerley | |||
| Anna L Chapman | |||
| Elizabeth L Tudor | |||
| Maria E Tennant | |||
| P2860 | cites work | The atypical Rho GTPases Miro-1 and Miro-2 have essential roles in mitochondrial trafficking | Q24319150 |
| Amyotrophic lateral sclerosis-associated SOD1 mutant proteins bind and aggregate with Bcl-2 in spinal cord mitochondria | Q28270155 | ||
| Mutant Cu, Zn superoxide dismutase that causes motoneuron degeneration is present in mitochondria in the CNS | Q28610671 | ||
| ALS: a disease of motor neurons and their nonneuronal neighbors | Q29618000 | ||
| Axonal membrane proteins are transported in distinct carriers: a two-color video microscopy study in cultured hippocampal neurons | Q30477482 | ||
| The genetics of axonal transport and axonal transport disorders | Q33258819 | ||
| Identification and transport of full-length amyloid precursor proteins in rat peripheral nervous system. | Q34351242 | ||
| Axonal transport of neurofilaments in normal and disease states | Q34582316 | ||
| Familial ALS-superoxide dismutases associate with mitochondria and shift their redox potentials. | Q35012498 | ||
| Molecular and cellular pathways of neurodegeneration in motor neurone disease | Q35489359 | ||
| Molecular motors in neuronal development, intracellular transport and diseases | Q35909233 | ||
| Expression of phosphatidylinositol (4,5) bisphosphate-specific pleckstrin homology domains alters direction but not the level of axonal transport of mitochondria. | Q35973416 | ||
| Axonal transport of mitochondria requires milton to recruit kinesin heavy chain and is light chain independent | Q36117548 | ||
| Neurofilaments and orthograde transport are reduced in ventral root axons of transgenic mice that express human SOD1 with a G93A mutation | Q36274995 | ||
| A mutation in dynein rescues axonal transport defects and extends the life span of ALS mice | Q36321318 | ||
| The axonal transport of mitochondria. | Q36321480 | ||
| Axonal transport and neurodegenerative disease | Q36491093 | ||
| The role of excitotoxicity in the pathogenesis of amyotrophic lateral sclerosis. | Q36522329 | ||
| Visualization and quantification of mitochondrial dynamics in living animal cells. | Q36796290 | ||
| Precursor of amyloid protein in Alzheimer disease undergoes fast anterograde axonal transport | Q37701379 | ||
| Interaction between familial amyotrophic lateral sclerosis (ALS)-linked SOD1 mutants and the dynein complex | Q40150834 | ||
| JNK mediates pathogenic effects of polyglutamine-expanded androgen receptor on fast axonal transport | Q40271687 | ||
| Tumor necrosis factor induces hyperphosphorylation of kinesin light chain and inhibits kinesin-mediated transport of mitochondria | Q40874645 | ||
| Slowing of axonal transport is a very early event in the toxicity of ALS-linked SOD1 mutants to motor neurons | Q41644415 | ||
| Tau blocks traffic of organelles, neurofilaments, and APP vesicles in neurons and enhances oxidative stress | Q41830251 | ||
| Axonal transport of mitochondria along microtubules and F-actin in living vertebrate neurons | Q41903637 | ||
| Glutamate slows axonal transport of neurofilaments in transfected neurons | Q42118341 | ||
| Axonal mitochondrial transport and potential are correlated | Q44901240 | ||
| Toxicity of familial ALS-linked SOD1 mutants from selective recruitment to spinal mitochondria. | Q45967463 | ||
| p38alpha stress-activated protein kinase phosphorylates neurofilaments and is associated with neurofilament pathology in amyotrophic lateral sclerosis. | Q47317586 | ||
| Persistent activation of p38 mitogen-activated protein kinase in a mouse model of familial amyotrophic lateral sclerosis correlates with disease progression. | Q47822839 | ||
| Protein kinase and protein phosphatase expression in the central nervous system of G93A mSOD over-expressing mice | Q48345349 | ||
| Inhibition of APP trafficking by tau protein does not increase the generation of amyloid-beta peptides | Q48517342 | ||
| Mitochondrial function and actin regulate dynamin-related protein 1-dependent mitochondrial fission. | Q50769548 | ||
| Mutated human SOD1 causes dysfunction of oxidative phosphorylation in mitochondria of transgenic mice. | Q51714635 | ||
| Motoneuron death triggered by a specific pathway downstream of Fas. potentiation by ALS-linked SOD1 mutations. | Q52114306 | ||
| Chloromethyl-X-Rosamine is an aldehyde-fixable potential-sensitive fluorochrome for the detection of early apoptosis. | Q52522800 | ||
| Variation in the biochemical/biophysical properties of mutant superoxide dismutase 1 enzymes and the rate of disease progression in familial amyotrophic lateral sclerosis kindreds. | Q54089001 | ||
| Impairment of fast axonal transport in the proximal axons of anterior horn neurons in amyotrophic lateral sclerosis | Q71395993 | ||
| Mitochondrial alterations in the spinal cord of patients with sporadic amyotrophic lateral sclerosis | Q79495290 | ||
| Activation of programmed cell death markers in ventral horn motor neurons during early presymptomatic stages of amyotrophic lateral sclerosis in a transgenic mouse model | Q80883798 | ||
| P4510 | describes a project that uses | ImageJ | Q1659584 |
| P433 | issue | 22 | |
| P921 | main subject | mitochondrion | Q39572 |
| amyotrophic lateral sclerosis | Q206901 | ||
| P304 | page(s) | 2720-2728 | |
| P577 | publication date | 2007-08-28 | |
| P1433 | published in | Human Molecular Genetics | Q2720965 |
| P1476 | title | Familial amyotrophic lateral sclerosis-linked SOD1 mutants perturb fast axonal transport to reduce axonal mitochondria content | |
| P478 | volume | 16 |
| Q28079299 | A Case for Microtubule Vulnerability in Amyotrophic Lateral Sclerosis: Altered Dynamics During Disease |
| Q60958104 | A Variant Identified in a Sporadic Amyotrophic Lateral Sclerosis Patient Impairs Microtubule Stability and Axonal Mitochondria Distribution |
| Q26775401 | A comprehensive review of amyotrophic lateral sclerosis |
| Q46070173 | A role for motoneuron subtype-selective ER stress in disease manifestations of FALS mice |
| Q34983748 | A switch in retrograde signaling from survival to stress in rapid-onset neurodegeneration |
| Q39665668 | A transgenic mouse expressing CHMP2Bintron5 mutant in neurons develops histological and behavioural features of amyotrophic lateral sclerosis and frontotemporal dementia |
| Q26774824 | ALS Patient Stem Cells for Unveiling Disease Signatures of Motoneuron Susceptibility: Perspectives on the Deadly Mitochondria, ER Stress and Calcium Triad |
| Q30572264 | Abnormal mitochondrial transport and morphology are common pathological denominators in SOD1 and TDP43 ALS mouse models |
| Q36506599 | Abnormal mitochondrial transport and morphology as early pathological changes in human models of spinal muscular atrophy. |
| Q33845021 | Abnormalities of Mitochondrial Dynamics in Neurodegenerative Diseases |
| Q34766996 | Absence of disturbed axonal transport in spinal and bulbar muscular atrophy |
| Q37249167 | Acetyltransferases (HATs) as targets for neurological therapeutics |
| Q47707422 | Amyotrophic lateral sclerosis-associated mutant SOD1 inhibits anterograde axonal transport of mitochondria by reducing Miro1 levels |
| Q35860341 | Amyotrophic lateral sclerosis-associated mutant VAPBP56S perturbs calcium homeostasis to disrupt axonal transport of mitochondria |
| Q38137892 | Amyotrophic lateral sclerosis: Problems and prospects |
| Q27313222 | An ALS-linked mutant SOD1 produces a locomotor defect associated with aggregation and synaptic dysfunction when expressed in neurons of Caenorhabditis elegans |
| Q37993651 | Antioxidant and bioenergetic coupling between neurons and astrocytes. |
| Q48673272 | Assessing Mitochondrial Movement Within Neurons: Manual Versus Automated Tracking Methods |
| Q41475139 | Axonal Degeneration during Aging and Its Functional Role in Neurodegenerative Disorders |
| Q34806938 | Axonal Transport Defects in a Mitofusin 2 Loss of Function Model of Charcot-Marie-Tooth Disease in Zebrafish. |
| Q92856606 | Axonal Transport and Mitochondrial Function in Neurons |
| Q33742751 | Axonal mitochondrial clusters containing mutant SOD1 in transgenic models of ALS. |
| Q39774347 | Axonal transport defects are a common phenotype in Drosophila models of ALS. |
| Q37614744 | Axonal transport defects in neurodegenerative diseases |
| Q30511133 | Axonal transport deficits and degeneration can evolve independently in mouse models of amyotrophic lateral sclerosis. |
| Q38077679 | Axonal transport deficits and neurodegenerative diseases |
| Q36488715 | Biology of mitochondria in neurodegenerative diseases |
| Q34262615 | Calsyntenin-1 mediates axonal transport of the amyloid precursor protein and regulates Aβ production |
| Q38006693 | Can transcriptomics cut the gordian knot of amyotrophic lateral sclerosis? |
| Q36407372 | Cargo distributions differentiate pathological axonal transport impairments |
| Q93159371 | Chain length of saturated fatty acids regulates mitochondrial trafficking and function in sensory neurons |
| Q86822652 | Characterization and changes in neurotrophin receptor p75-Expressing motor neurons in SOD1(G93A) G1H mice [corrected] |
| Q37407284 | Characterization of early pathogenesis in the SOD1(G93A) mouse model of ALS: part I, background and methods. |
| Q48449807 | Characterization of mitochondrial transport in neurons |
| Q26824841 | Charcot-Marie-Tooth disease and intracellular traffic |
| Q35357235 | Comparison of dendritic calcium transients in juvenile wild type and SOD1(G93A) mouse lumbar motoneurons |
| Q38810383 | Connecting mitochondrial dynamics and life-or-death events via Bcl-2 family proteins |
| Q37122007 | Cytoplasmic dynein could be key to understanding neurodegeneration |
| Q34143936 | Defects in mitochondrial axonal transport and membrane potential without increased reactive oxygen species production in a Drosophila model of Friedreich ataxia |
| Q30497554 | Deficits in axonal transport precede ALS symptoms in vivo |
| Q41818231 | Deregulation of PKN1 activity disrupts neurofilament organisation and axonal transport |
| Q92017242 | Developmental regulation of microtubule-based trafficking and anchoring of axonal mitochondria in health and diseases |
| Q28364642 | Difference Tracker: ImageJ plugins for fully automated analysis of multiple axonal transport parameters |
| Q36934610 | Different regulation of wild-type and mutant Cu,Zn superoxide dismutase localization in mammalian mitochondria |
| Q27335303 | Disruption of mitochondrial DNA replication in Drosophila increases mitochondrial fast axonal transport in vivo |
| Q34171101 | Distinct pathogenic processes between Fig4-deficient motor and sensory neurons. |
| Q50114087 | Dyslipidemia impairs mitochondrial trafficking and function in sensory neurons. |
| Q26753110 | Dysregulated axonal RNA translation in amyotrophic lateral sclerosis |
| Q36012034 | Dysregulated expression of death, stress and mitochondrion related genes in the sciatic nerve of presymptomatic SOD1(G93A) mouse model of Amyotrophic Lateral Sclerosis |
| Q37830720 | Dysregulation of axonal transport and motorneuron diseases. |
| Q24315910 | Effects of ALS-related SOD1 mutants on dynein- and KIF5-mediated retrograde and anterograde axonal transport |
| Q37832956 | Elongator: an ancestral complex driving transcription and migration through protein acetylation |
| Q35879849 | Endolysosomal Deficits Augment Mitochondria Pathology in Spinal Motor Neurons of Asymptomatic fALS Mice. |
| Q52651163 | Energy metabolism in ALS: an underappreciated opportunity? |
| Q28391199 | Exploring new pathways of neurodegeneration in ALS: the role of mitochondria quality control |
| Q35551935 | Expression of mutant TDP-43 induces neuronal dysfunction in transgenic mice |
| Q37241079 | FTD and ALS: a tale of two diseases |
| Q36106275 | Genetic inactivation of mitochondria-targeted redox enzyme p66ShcA preserves neuronal viability and mitochondrial integrity in response to oxidative challenges |
| Q28468383 | HDAC6 Inhibitors Rescued the Defective Axonal Mitochondrial Movement in Motor Neurons Derived from the Induced Pluripotent Stem Cells of Peripheral Neuropathy Patients with HSPB1 Mutation |
| Q24338481 | HUMMR, a hypoxia- and HIF-1alpha-inducible protein, alters mitochondrial distribution and transport |
| Q47195067 | High content image analysis reveals function of miR-124 upstream of Vimentin in regulating motor neuron mitochondria. |
| Q27321452 | INaP selective inhibition reverts precocious inter- and motorneurons hyperexcitability in the Sod1-G93R zebrafish ALS model |
| Q41235266 | Imaging axonal transport of mitochondria |
| Q26766040 | Impaired Autophagy and Defective Mitochondrial Function: Converging Paths on the Road to Motor Neuron Degeneration |
| Q35989856 | In vivo imaging of axonal transport of mitochondria in the diseased and aged mammalian CNS. |
| Q35067857 | Increased axonal mitochondrial mobility does not slow amyotrophic lateral sclerosis (ALS)-like disease in mutant SOD1 mice |
| Q37534549 | Increased axonal ribosome numbers is an early event in the pathogenesis of amyotrophic lateral sclerosis |
| Q30596172 | Increasing microtubule acetylation rescues axonal transport and locomotor deficits caused by LRRK2 Roc-COR domain mutations |
| Q48240848 | Inhibition of Drp1/Fis1 interaction slows progression of amyotrophic lateral sclerosis. |
| Q28533850 | Inhibition of fast axonal transport by pathogenic SOD1 involves activation of p38 MAP kinase |
| Q37689883 | Insulin and Insulin-Sensitizing Drugs in Neurodegeneration: Mitochondria as Therapeutic Targets |
| Q37043070 | Is SOD1 loss of function involved in amyotrophic lateral sclerosis? |
| Q91729458 | Kinesin light chain-1 serine-460 phosphorylation is altered in Alzheimer's disease and regulates axonal transport and processing of the amyloid precursor protein |
| Q43577107 | Links between electrophysiological and molecular pathology of amyotrophic lateral sclerosis |
| Q55409966 | Live Imaging Mitochondrial Transport in Neurons. |
| Q30564134 | Loss of c-Jun N-terminal kinase-interacting protein-1 does not affect axonal transport of the amyloid precursor protein or Aβ production |
| Q37625840 | Loss of spastin function results in disease-specific axonal defects in human pluripotent stem cell-based models of hereditary spastic paraplegia |
| Q34323864 | Loss of the m-AAA protease subunit AFG₃L₂ causes mitochondrial transport defects and tau hyperphosphorylation |
| Q41965822 | Loss of vps54 function leads to vesicle traffic impairment, protein mis-sorting and embryonic lethality. |
| Q58787868 | Maximizing the Survival of Amyotrophic Lateral Sclerosis Patients: Current Perspectives |
| Q26998585 | Mechanisms of mutant SOD1 induced mitochondrial toxicity in amyotrophic lateral sclerosis |
| Q26865234 | Microtubule-stabilizing agents as potential therapeutics for neurodegenerative disease |
| Q35643403 | Miro1 deficiency in amyotrophic lateral sclerosis |
| Q33968208 | Misfolded SOD1 associated with motor neuron mitochondria alters mitochondrial shape and distribution prior to clinical onset. |
| Q37844421 | Mitochondria as crucial players in demyelinated axons: lessons from neuropathology and experimental demyelination |
| Q47853116 | Mitochondria at the neuronal presynapse in health and disease. |
| Q37216404 | Mitochondria in neuroplasticity and neurological disorders |
| Q36124905 | Mitochondria: the next (neurode)generation |
| Q98224396 | Mitochondrial Dysfunction, Neurogenesis, and Epigenetics: Putative Implications for Amyotrophic Lateral Sclerosis Neurodegeneration and Treatment |
| Q42580316 | Mitochondrial Dysfunction: The Road to Alpha-Synuclein Oligomerization in PD. |
| Q46563454 | Mitochondrial Membrane Potential-dependent Endoplasmic Reticulum Fragmentation is an Important Step in Neuritic Degeneration. |
| Q57155833 | Mitochondrial Targeting in Neurodegeneration: A Heme Perspective |
| Q91982212 | Mitochondrial Transport and Turnover in the Pathogenesis of Amyotrophic Lateral Sclerosis |
| Q26865358 | Mitochondrial abnormalities in Alzheimer's disease: possible targets for therapeutic intervention |
| Q34502221 | Mitochondrial and Cell Death Mechanisms in Neurodegenerative Diseases |
| Q37348590 | Mitochondrial approaches for neuroprotection |
| Q36709397 | Mitochondrial changes within axons in multiple sclerosis |
| Q35899260 | Mitochondrial dynamic abnormalities in amyotrophic lateral sclerosis |
| Q36593996 | Mitochondrial dynamics and bioenergetic dysfunction is associated with synaptic alterations in mutant SOD1 motor neurons |
| Q36082495 | Mitochondrial dynamics in cancer and neurodegenerative and neuroinflammatory diseases |
| Q38365008 | Mitochondrial dynamism and the pathogenesis of Amyotrophic Lateral Sclerosis. |
| Q34103782 | Mitochondrial dysfunction and intracellular calcium dysregulation in ALS |
| Q37588819 | Mitochondrial dysfunction in amyotrophic lateral sclerosis. |
| Q37432118 | Mitochondrial function, morphology, and axonal transport in amyotrophic lateral sclerosis |
| Q34038775 | Mitochondrial pathobiology in ALS. |
| Q37686252 | Mitochondrial protein quality control in health and disease |
| Q41429779 | Mitochondrial quality control in amyotrophic lateral sclerosis: towards a common pathway? |
| Q37677601 | Mitochondrial trafficking and anchoring in neurons: New insight and implications |
| Q27310252 | Mitochondrial transport in neurons: impact on synaptic homeostasis and neurodegeneration |
| Q92547045 | Mitochondrial transport serves as a mitochondrial quality control strategy in axons: Implications for central nervous system disorders |
| Q28473111 | Modification of superoxide dismutase 1 (SOD1) properties by a GFP tag--implications for research into amyotrophic lateral sclerosis (ALS) |
| Q30538475 | Molecular chaperone Hsp110 rescues a vesicle transport defect produced by an ALS-associated mutant SOD1 protein in squid axoplasm |
| Q26820831 | Molecular motor proteins and amyotrophic lateral sclerosis |
| Q37952528 | Molecular pathways of motor neuron injury in amyotrophic lateral sclerosis |
| Q91970420 | Motor Neuron Susceptibility in ALS/FTD |
| Q35027903 | Motor neuron trophic factors: Therapeutic use in ALS? |
| Q37411790 | Mutant SOD1 in neuronal mitochondria causes toxicity and mitochondrial dynamics abnormalities |
| Q38844786 | Mutant SOD1G93A triggers mitochondrial fragmentation in spinal cord motor neurons: neuroprotection by SIRT3 and PGC-1α. |
| Q41409358 | Necrotic cell death and neurodegeneration: The involvement of endocytosis and intracellular trafficking |
| Q39149919 | Neurobiology of axonal transport defects in motor neuron diseases: Opportunities for translational research? |
| Q91972707 | Neurodegenerative Diseases - Is Metabolic Deficiency the Root Cause? |
| Q40345993 | Neuronal overexpression of human VAPB slows motor impairment and neuromuscular denervation in a mouse model of ALS. |
| Q27026022 | Neuroprotection of antioxidant enzymes against transient global cerebral ischemia in gerbils |
| Q33783000 | New Therapeutics to Modulate Mitochondrial Function in Neurodegenerative Disorders |
| Q24596378 | Olesoxime, a cholesterol-like neuroprotectant for the potential treatment of amyotrophic lateral sclerosis |
| Q91741049 | Omics Approach to Axonal Dysfunction of Motor Neurons in Amyotrophic Lateral Sclerosis (ALS) |
| Q34005426 | Optimised and rapid pre-clinical screening in the SOD1(G93A) transgenic mouse model of amyotrophic lateral sclerosis (ALS). |
| Q39235670 | PINK1 and Parkin are genetic modifiers for FUS-induced neurodegeneration. |
| Q30431829 | Palmitoylation of superoxide dismutase 1 (SOD1) is increased for familial amyotrophic lateral sclerosis-linked SOD1 mutants |
| Q34307291 | Pharmacologic rescue of axon growth defects in a human iPSC model of hereditary spastic paraplegia SPG3A. |
| Q34667617 | Phosphorylation of kinesin light chain 1 at serine 460 modulates binding and trafficking of calsyntenin-1. |
| Q90753145 | Pleiotropic Mitochondria: The Influence of Mitochondria on Neuronal Development and Disease |
| Q34026859 | Protocol for a double-blind randomised placebo-controlled trial of lithium carbonate in patients with amyotrophic lateral sclerosis (LiCALS) [Eudract number: 2008-006891-31]. |
| Q46265628 | ROS Control Mitochondrial Motility through p38 and the Motor Adaptor Miro/Trak |
| Q36460627 | Redox signaling pathways involved in neuronal ischemic preconditioning |
| Q36518622 | Reducing Lissencephaly-1 levels augments mitochondrial transport and has a protective effect in adult Drosophila neurons. |
| Q39243689 | Regulation of motor proteins, axonal transport deficits and adult-onset neurodegenerative diseases |
| Q51019981 | Releasing Syntaphilin Removes Stressed Mitochondria from Axons Independent of Mitophagy under Pathophysiological Conditions. |
| Q43021472 | Reply: Two novel mutations in conserved codons indicate that CHCHD10 is a gene associated with motor neuron disease. |
| Q90044486 | Restoring Cellular Energetics Promotes Axonal Regeneration and Functional Recovery after Spinal Cord Injury |
| Q33987923 | Retrograde axonal transport: pathways to cell death? |
| Q38050513 | Review: Axon pathology in age-related neurodegenerative disorders |
| Q37836981 | Review: The role of mitochondria in the pathogenesis of amyotrophic lateral sclerosis. |
| Q26749357 | Role of Intermediate Filaments in Vesicular Traffic |
| Q33819255 | Role of mitochondria in mutant SOD1 linked amyotrophic lateral sclerosis |
| Q35916588 | Role of mitochondrial homeostasis and dynamics in Alzheimer's disease |
| Q36396423 | Rosmarinic Acid Alleviates Neurological Symptoms in the G93A-SOD1 Transgenic Mouse Model of Amyotrophic Lateral Sclerosis |
| Q34438640 | SOD1 targeted to the mitochondrial intermembrane space prevents motor neuropathy in the Sod1 knockout mouse |
| Q39468775 | Selective mitochondrial Ca2+ uptake deficit in disease endstage vulnerable motoneurons of the SOD1G93A mouse model of amyotrophic lateral sclerosis |
| Q28536678 | Small molecule suppressors of Drosophila kinesin deficiency rescue motor axon development in a zebrafish model of spinal muscular atrophy |
| Q36494315 | State of the field: An informatics-based systematic review of the SOD1-G93A amyotrophic lateral sclerosis transgenic mouse model |
| Q64075421 | Targeting the Sigma-1 Receptor via Pridopidine Ameliorates Central Features of ALS Pathology in a SOD1 Model |
| Q92459188 | The Divergent Roles of Dietary Saturated and Monounsaturated Fatty Acids on Nerve Function in Murine Models of Obesity |
| Q64069200 | The Role of the Antioxidant Response in Mitochondrial Dysfunction in Degenerative Diseases: Cross-Talk between Antioxidant Defense, Autophagy, and Apoptosis |
| Q28263899 | The complex molecular biology of amyotrophic lateral sclerosis (ALS) |
| Q37168377 | The four major N- and C-terminal splice variants of the excitatory amino acid transporter GLT-1 form cell surface homomeric and heteromeric assemblies. |
| Q30435889 | The legs at odd angles (Loa) mutation in cytoplasmic dynein ameliorates mitochondrial function in SOD1G93A mouse model for motor neuron disease |
| Q38006810 | The meaning of mitochondrial movement to a neuron's life |
| Q37261104 | The mitochondrial permeability transition pore in motor neurons: involvement in the pathobiology of ALS mice |
| Q37466115 | The mitochondrial permeability transition pore: a molecular target for amyotrophic lateral sclerosis therapy |
| Q33938412 | The potential for transition metal-mediated neurodegeneration in amyotrophic lateral sclerosis |
| Q33633778 | The role of oxidative stress in degeneration of the neuromuscular junction in amyotrophic lateral sclerosis |
| Q95819747 | The ubiquitous role of mitochondria in Parkinson and other neurodegenerative diseases |
| Q38070882 | The ultimate question: why do axons degenerate? A tribute to the work and mentorship of John W. Griffin, MD. |
| Q52649242 | The voltage-gated calcium channel blocker lomerizine is neuroprotective in motor neurons expressing mutant SOD1, but not TDP-43. |
| Q36878198 | The wobbler mouse, an ALS animal model |
| Q26742053 | Transporting mitochondria in neurons |
| Q48092663 | Understanding Miro GTPases: Implications in the Treatment of Neurodegenerative Disorders. |
| Q57816866 | Unraveling the Role of Heme in Neurodegeneration |
| Q24298739 | VAPB interacts with the mitochondrial protein PTPIP51 to regulate calcium homeostasis |
| Q42503556 | WldS prevents axon degeneration through increased mitochondrial flux and enhanced mitochondrial Ca2+ buffering |
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