scholarly article | Q13442814 |
P50 | author | Uwe Völker | Q58949104 |
Julia Mayerle | Q65559556 | ||
Henry Völzke | Q28039332 | ||
Claudia Schurmann | Q28833429 | ||
Markus M Lerch | Q38327735 | ||
P2093 | author name string | Georg Beyer | |
Georg Homuth | |||
Vivien Kauschke | |||
Thomas Wartmann | |||
Walter Halangk | |||
Peter Hegyi | |||
Ujjwal M Mahajan | |||
Matthias Sendler | |||
Frank-Ulrich Weiss | |||
Sandrina Maertin | |||
P2860 | cites work | Angiopoietin-2, a regulator of vascular permeability in inflammation, is associated with persistent organ failure in patients with acute pancreatitis from the United States and Germany. | Q51771520 |
Autoimmune pancreatitis in MRL/Mp mice is a T cell-mediated disease responsive to cyclosporine A and rapamycin treatment | Q56897015 | ||
Cohort Profile: The Study of Health in Pomerania | Q57429582 | ||
Toll-like receptor 4 polymorphisms in German and US patients are not associated with occurrence or severity of acute pancreatitis | Q58436119 | ||
Transcriptome analysis of human hepatic and pancreatic stellate cells: organ-specific variations of a common transcriptional phenotype | Q60609526 | ||
Balance of expression of genes coding for extracellular matrix proteins and extracellular matrix degrading proteases in chronic pancreatitis | Q60609552 | ||
Does alcohol directly stimulate pancreatic fibrogenesis? Studies with rat pancreatic stellate cells | Q60961219 | ||
Anaphylatoxin generation in acute pancreatitis | Q69742106 | ||
Complement component C5 deficiency reduces edema formation in murine ligation-induced acute pancreatitis | Q73154847 | ||
Early complement system activation and neutrophil priming in acute pancreatitis: participation of trypsin | Q73880761 | ||
Upregulation of fibronectin but not of entactin, collagen IV and smooth muscle actin by anaphylatoxin C5a in rat hepatic stellate cells | Q80581322 | ||
Fibrogenesis in alcoholic chronic pancreatitis: the role of tissue necrosis, macrophages, myofibroblasts and cytokines | Q83299268 | ||
Withdrawal of alcohol promotes regression while continued alcohol intake promotes persistence of LPS-induced pancreatic injury in alcohol-fed rats | Q85093731 | ||
Complement 5a stimulates hepatic stellate cells in vitro, and is increased in the plasma of patients with chronic hepatitis B | Q85323799 | ||
Senescence of activated stellate cells limits liver fibrosis | Q24621822 | ||
Mechanisms of hepatic fibrogenesis | Q29619336 | ||
Transient receptor potential ankyrin 1 mediates chronic pancreatitis pain in mice. | Q30437425 | ||
Treatment of severe neurological deficits with IgG depletion through immunoadsorption in patients with Escherichia coli O104:H4-associated haemolytic uraemic syndrome: a prospective trial | Q33397004 | ||
Identification of a potent and orally active non-peptide C5a receptor antagonist | Q34154819 | ||
Periacinar stellate shaped cells in rat pancreas: identification, isolation, and culture. | Q34475071 | ||
Pancreatic stellate cells respond to inflammatory cytokines: potential role in chronic pancreatitis | Q35594510 | ||
StellaTUM: current consensus and discussion on pancreatic stellate cell research | Q35633517 | ||
Activation of pancreatic stellate cells in human and experimental pancreatic fibrosis | Q35788265 | ||
Mononuclear cells modulate the activity of pancreatic stellate cells which in turn promote fibrosis and inflammation in chronic pancreatitis | Q36446987 | ||
IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality | Q36638151 | ||
Inhibition of biomaterial-induced complement activation attenuates the inflammatory host response to implantation | Q36945134 | ||
Chronic pancreatitis--definition, etiology, investigation and treatment | Q36974615 | ||
Animal models for investigating chronic pancreatitis. | Q37963556 | ||
Models of acute and chronic pancreatitis. | Q38102103 | ||
Role of cathepsin B in intracellular trypsinogen activation and the onset of acute pancreatitis | Q40735691 | ||
The role of C5 in septic lung injury | Q41507371 | ||
Evaluation of complement factor 5 variants as genetic risk factors for the development of advanced fibrosis in chronic hepatitis C infection | Q41604038 | ||
Pancreatic stellate cells are activated by proinflammatory cytokines: implications for pancreatic fibrogenesis | Q42116957 | ||
Protective effects of MCP-1 inhibitor on a rat model of severe acute pancreatitis. | Q43103762 | ||
The morphological basis for the evolution of acute pancreatitis into chronic pancreatitis | Q43541521 | ||
Complement factor C5a exerts an anti-inflammatory effect in acute pancreatitis and associated lung injury | Q43568320 | ||
Identification of mediators stimulating proliferation and matrix synthesis of rat pancreatic stellate cells | Q43666355 | ||
Complement factor 5 is a quantitative trait gene that modifies liver fibrogenesis in mice and humans | Q44042948 | ||
Pancreatic neuropathy and neuropathic pain--a comprehensive pathomorphological study of 546 cases | Q44124539 | ||
Genome-wide analysis of hepatic fibrosis in inbred mice identifies the susceptibility locus Hfib1 on chromosome 15. | Q44231634 | ||
Anaphylatoxins and terminal complement complexes in pancreatitis. Evidence of complement activation in plasma and ascites fluid of patients with acute pancreatitis | Q44521059 | ||
Correlation among complement activation, protease inhibitors, and clinical course in acute pancreatitis in man. | Q44523799 | ||
Tumour necrosis factor α secretion induces protease activation and acinar cell necrosis in acute experimental pancreatitis in mice | Q45056690 | ||
Camostat, an oral trypsin inhibitor, reduces pancreatic fibrosis induced by repeated administration of a superoxide dismutase inhibitor in rats | Q46536581 | ||
Identification of genetic loci associated with Helicobacter pylori serologic status. | Q51012428 | ||
P4510 | describes a project that uses | ImageJ | Q1659584 |
P433 | issue | 3 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | chronic pancreatitis | Q1996053 |
P304 | page(s) | 765-76.e10 | |
P577 | publication date | 2015-05-19 | |
P1433 | published in | Gastroenterology | Q4039279 |
P1476 | title | Complement Component 5 Mediates Development of Fibrosis, via Activation of Stellate Cells, in 2 Mouse Models of Chronic Pancreatitis | |
P478 | volume | 149 |
Q28392302 | Animal models of gastrointestinal and liver diseases. Animal models of acute and chronic pancreatitis |
Q41862502 | Cathepsin B Activity Initiates Apoptosis via Digestive Protease Activation in Pancreatic Acinar Cells and Experimental Pancreatitis. |
Q45991735 | Cathepsin B-mediated Activation of Trypsinogen in Endocytosing Macrophages Increases Severity of Pancreatitis in Mice. |
Q52519464 | Chronic pancreatitis. |
Q47238226 | Complement C5a inhibition moderates lipid metabolism and reduces tubulointerstitial fibrosis in diabetic nephropathy. |
Q38988021 | Complement in Pancreatic Disease-Perpetrator or Savior? |
Q36913772 | Contribution of the anaphylatoxin receptors, C3aR and C5aR, to the pathogenesis of pulmonary fibrosis |
Q93222396 | Deficiency of cathepsin C ameliorates severity of acute pancreatitis by reduction of neutrophil elastase activation and cleavage of E-cadherin |
Q38656177 | Do Animal Models of Acute Pancreatitis Reproduce Human Disease? |
Q36583714 | Higher plasma levels of complement C3a, C4a and C5a increase the risk of subretinal fibrosis in neovascular age-related macular degeneration: Complement activation in AMD. |
Q33687509 | Inhibition of Jak/STAT signaling reduces the activation of pancreatic stellate cells in vitro and limits caerulein-induced chronic pancreatitis in vivo |
Q36357685 | Lysosome associated membrane proteins maintain pancreatic acinar cell homeostasis: LAMP-2 deficient mice develop pancreatitis |
Q38900762 | New insights into the pathways initiating and driving pancreatitis. |
Q58712490 | RB1CC1-enhanced autophagy facilitates PSCs activation and pancreatic fibrogenesis in chronic pancreatitis |
Q52946378 | Systems Genetics of Liver Fibrosis. |
Q28066426 | The Immune System in Tissue Environments Regaining Homeostasis after Injury: Is "Inflammation" Always Inflammation? |
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