scholarly article | Q13442814 |
P50 | author | Cord Brakebusch | Q30093323 |
P2093 | author name string | Julie Ruston | |
Tony Pawson | |||
Nina Jones | |||
Steve P Hawley | |||
Jianmei Du | |||
Rizaldy P Scott | |||
P2860 | cites work | An ultrastructural study of the glomerular slit diaphragm in New Zealand black/white mice | Q40009914 |
On the ultrastructural organization of the microfilament system and the possible role of profilactin | Q40305110 | ||
Rac1 deletion in mouse neutrophils has selective effects on neutrophil functions. | Q40646102 | ||
Cytoskeletal changes in podocytes associated with foot process effacement in Masugi nephritis. | Q42049689 | ||
Insulin signaling to the glomerular podocyte is critical for normal kidney function | Q42110064 | ||
Filament arrangements in negatively stained cultured cells: the organization of actin | Q42830885 | ||
Loss of podocyte aPKClambda/iota causes polarity defects and nephrotic syndrome | Q43152476 | ||
Fluvastatin ameliorates podocyte injury in proteinuric rats via modulation of excessive Rho signaling | Q46923382 | ||
Cdc42 controls the polarity of the actin and microtubule cytoskeletons through two distinct signal transduction pathways. | Q53673562 | ||
Progressive impairment of kidneys and reproductive organs in mice lacking Rho GDIalpha | Q73023855 | ||
Nck adaptor proteins link nephrin to the actin cytoskeleton of kidney podocytes | Q24307359 | ||
Rho, rac, and cdc42 GTPases regulate the assembly of multimolecular focal complexes associated with actin stress fibers, lamellipodia, and filopodia | Q27861126 | ||
Arhgap24 inactivates Rac1 in mouse podocytes, and a mutant form is associated with familial focal segmental glomerulosclerosis | Q28504659 | ||
The Rho-kinase inhibitor, fasudil, attenuates diabetic nephropathy in streptozotocin-induced diabetic rats | Q28580779 | ||
Cdc42 controls progenitor cell differentiation and beta-catenin turnover in skin | Q28587207 | ||
Mammalian Rho GTPases: new insights into their functions from in vivo studies | Q29615191 | ||
An essential role of the universal polarity protein, aPKClambda, on the maintenance of podocyte slit diaphragms | Q33399707 | ||
Cofilin-1 inactivation leads to proteinuria--studies in zebrafish, mice and humans | Q33691530 | ||
Actin-depolymerizing factor cofilin-1 is necessary in maintaining mature podocyte architecture | Q33991246 | ||
INF2 mutations in Charcot-Marie-Tooth disease with glomerulopathy | Q34242050 | ||
RhoA is dispensable for skin development, but crucial for contraction and directed migration of keratinocytes | Q34611649 | ||
Glomerular-specific alterations of VEGF-A expression lead to distinct congenital and acquired renal diseases | Q34835409 | ||
Gene targeting of Cdc42 and Cdc42GAP affirms the critical involvement of Cdc42 in filopodia induction, directed migration, and proliferation in primary mouse embryonic fibroblasts | Q35128385 | ||
Rac1 GTPase in rodent kidneys is essential for salt-sensitive hypertension via a mineralocorticoid receptor-dependent pathway | Q35142803 | ||
Activation of RhoA in podocytes induces focal segmental glomerulosclerosis | Q35210190 | ||
Synaptopodin protects against proteinuria by disrupting Cdc42:IRSp53:Mena signaling complexes in kidney podocytes | Q35916016 | ||
Hereditary proteinuria syndromes and mechanisms of proteinuria | Q36435101 | ||
Molecular make-up of the glomerular filtration barrier | Q37759816 | ||
Modification of mineralocorticoid receptor function by Rac1 GTPase: implication in proteinuric kidney disease | Q39912709 | ||
P433 | issue | 7 | |
P921 | main subject | congenital disorder | Q727096 |
kidney disease | Q1054718 | ||
P304 | page(s) | 1149-1154 | |
P577 | publication date | 2012-04-19 | |
P1433 | published in | Journal of the American Society of Nephrology | Q17123893 |
P1476 | title | Podocyte-specific loss of Cdc42 leads to congenital nephropathy | |
P478 | volume | 23 |
Q85964497 | AKT2 is essential to maintain podocyte viability and function during chronic kidney disease |
Q42163264 | ARHGDIA mutations cause nephrotic syndrome via defective RHO GTPase signaling |
Q36764175 | ARHGDIA: a novel gene implicated in nephrotic syndrome |
Q90437131 | ARHGEF7 (β-PIX) Is Required for the Maintenance of Podocyte Architecture and Glomerular Function |
Q52573827 | Actin dynamics at focal adhesions: a common endpoint and putative therapeutic target for proteinuric kidney diseases. |
Q37402598 | Actin-associated Proteins in the Pathogenesis of Podocyte Injury |
Q48118712 | Adrenomedullin ameliorates podocyte injury induced by puromycin aminonucleoside in vitro and in vivo through modulation of Rho GTPases. |
Q34537282 | An in vivo functional analysis system for renal gene discovery in Drosophila pericardial nephrocytes |
Q92535740 | Catalpol Ameliorates Podocyte Injury by Stabilizing Cytoskeleton and Enhancing Autophagy in Diabetic Nephropathy |
Q37119933 | Cdc42 deficiency causes ciliary abnormalities and cystic kidneys. |
Q36774995 | Cdc42 deficiency induces podocyte apoptosis by inhibiting the Nwasp/stress fibers/YAP pathway |
Q38332511 | Cell biology of diabetic nephropathy: Roles of endothelial cells, tubulointerstitial cells and podocytes |
Q38075788 | Cell-matrix adhesion of podocytes in physiology and disease |
Q35124266 | Deficiency of the planar cell polarity protein Vangl2 in podocytes affects glomerular morphogenesis and increases susceptibility to injury. |
Q42730836 | Disease-causing mutations of RhoGDIα induce Rac1 hyperactivation in podocytes |
Q37274491 | Divergent functions of the Rho GTPases Rac1 and Cdc42 in podocyte injury |
Q36810711 | Dosage-dependent role of Rac1 in podocyte injury. |
Q51631984 | Dynamin Autonomously Regulates Podocyte Focal Adhesion Maturation. |
Q35932259 | FAT1 mutations cause a glomerulotubular nephropathy |
Q64080545 | FHL2 mediates podocyte Rac1 activation and foot process effacement in hypertensive nephropathy |
Q34674987 | Genetic causes of proteinuria and nephrotic syndrome: impact on podocyte pathobiology |
Q35719874 | Gq signaling causes glomerular injury by activating TRPC6. |
Q37729204 | Hepatitis B Virus X Protein Stimulates Proliferation, Wound Closure and Inhibits Apoptosis of HuH-7 Cells via CDC42. |
Q40824690 | Human Kidney Disease-causing INF2 Mutations Perturb Rho/Dia Signaling in the Glomerulus |
Q36880430 | Inverted formin 2 regulates actin dynamics by antagonizing Rho/diaphanous-related formin signaling. |
Q35836132 | KANK deficiency leads to podocyte dysfunction and nephrotic syndrome |
Q55038898 | Lipid peroxidation regulates podocyte migration and cytoskeletal structure through redox sensitive RhoA signaling. |
Q55069283 | Loss of Rho-GDIα sensitizes podocytes to lipopolysaccharide-mediated injury. |
Q27015885 | Membrane trafficking in podocyte health and disease |
Q41825141 | Mice with mutant Inf2 show impaired podocyte and slit diaphragm integrity in response to protamine-induced kidney injury |
Q57151123 | Mutations in multiple components of the nuclear pore complex cause nephrotic syndrome |
Q55056709 | Mutations in six nephrosis genes delineate a pathogenic pathway amenable to treatment. |
Q40129134 | N-wasp is required for stabilization of podocyte foot processes |
Q49846333 | Networks that link cytoskeletal regulators and diaphragm proteins underpin filtration function in Drosophila nephrocytes |
Q58699371 | Nuclear YAP localization as a key regulator of podocyte function |
Q90835768 | PLCE1 regulates the migration, proliferation, and differentiation of podocytes |
Q34227115 | Podocyte injury caused by indoxyl sulfate, a uremic toxin and aryl-hydrocarbon receptor ligand |
Q38940494 | Podocyte-actin dynamics in health and disease |
Q26766118 | Podocytes |
Q57652246 | Proteinuric Kidney Diseases: A Podocyte's Slit Diaphragm and Cytoskeleton Approach |
Q42872034 | Rac1 activation in podocytes induces rapid foot process effacement and proteinuria |
Q51736357 | Rac1 in podocytes promotes glomerular repair and limits the formation of sclerosis. |
Q49853226 | Reciprocal regulation of YAP/TAZ by the Hippo pathway and the Small GTPase pathway |
Q35543167 | Review series: The cell biology of renal filtration |
Q36147929 | Rho GTPases in embryonic development |
Q59052275 | Rho-GTPase Signalling in the Pathogenesis of Nephrotic Syndrome |
Q35124219 | Rhophilin-1 is a key regulator of the podocyte cytoskeleton and is essential for glomerular filtration |
Q38072297 | Role of Rac1-mineralocorticoid-receptor signalling in renal and cardiac disease |
Q38154296 | Role of Rho-GTPases and their regulatory proteins in glomerular podocyte function |
Q41565785 | Studying the role of fascin-1 in mechanically stressed podocytes |
Q92257319 | TRPC Channels in Proteinuric Kidney Diseases |
Q38770231 | Targeting the podocyte cytoskeleton: from pathogenesis to therapy in proteinuric kidney disease |
Q50231801 | The Evolving Complexity of the Podocyte Cytoskeleton |
Q33573218 | The Rac-specific exchange factors Dock1 and Dock5 are dispensable for the establishment of the glomerular filtration barrier in vivo |
Q40758250 | The Rho-GTPase binding protein IQGAP2 is required for the glomerular filtration barrier. |
Q46062672 | The WD40-domain containing protein CORO2B is specifically enriched in glomerular podocytes and regulates the ventral actin cytoskeleton. |
Q27006914 | The function of endocytosis in podocytes |
Q38263509 | The importance of podocyte adhesion for a healthy glomerulus |
Q39721946 | The podocyte cytoskeleton in health and in disease |
Q100750188 | The potential roles of NAD(P)H:quinone oxidoreductase 1 in the development of diabetic nephropathy and actin polymerization |
Q38100984 | The role of the podocyte in albumin filtration |
Q91624576 | The ubiquitin-proteasome system in kidney physiology and disease |
Q90644144 | Transcriptional regulation of cell shape during organ morphogenesis |
Q37670295 | Two-photon microscopy reveals stationary podocytes in living zebrafish larvae |
Q36574757 | aPKCλ/ι and aPKCζ contribute to podocyte differentiation and glomerular maturation |
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