| scholarly article | Q13442814 |
| P2093 | author name string | J Courcelle | |
| C T Courcelle | |||
| H A Jeiranian | |||
| P2860 | cites work | Nature of transforming deoxyribonucleic acid in calcium-treated Escherichia coli | Q39977833 |
| Resident enhanced repair: novel repair process action on plasmid DNA transformed into Escherichia coli K-12. | Q39983926 | ||
| Dissociation of synthetic Holliday junctions by E. coli RecG protein. | Q40871651 | ||
| Factors affecting the transformation of Escherichia coli strain χ1776 by pBR322 plasmid DNA | Q40978542 | ||
| Transformation of plasmid DNA into E. coli using the heat shock method | Q43043819 | ||
| Analysis of comparative efficiencies of different transformation methods of E. coli using two common plasmid vectors | Q43211914 | ||
| pBluescriptII: multifunctional cloning and mapping vectors | Q46737419 | ||
| Replication of pSC101: effects of mutations in the E. coli DNA binding protein IHF. | Q48363199 | ||
| RecA-mediated excision repair: a novel mechanism for repairing DNA lesions at sites of arrested DNA synthesis. | Q50974783 | ||
| Preferential post-replication repair of DNA lesions situated on the leading strand of plasmids in Escherichia coli. | Q53047505 | ||
| Survival and induction of SOS in Escherichia coli treated with cisplatin, UV-irradiation, or mitomycin C are dependent on the function of the RecBC and RecFOR pathways of homologous recombination. | Q54009598 | ||
| RecBCD and RecJ/RecQ initiate DNA degradation on distinct substrates in UV-irradiated Escherichia coli. | Q54433680 | ||
| RuvABC is required to resolve holliday junctions that accumulate following replication on damaged templates in Escherichia coli. | Q54458103 | ||
| Multimerization of high copy number plasmids causes instability: Cole 1 encodes a determinant essential for plasmid monomerization and stability | Q54477200 | ||
| DNA damage-induced replication fork regression and processing in Escherichia coli. | Q54531654 | ||
| Evidence for a recombination-independent pathway for the repair of DNA interstrand cross-links based on a site-specific study with nitrogen mustard. | Q54568901 | ||
| Nature of the SOS-inducing signal in Escherichia coli. The involvement of DNA replication. | Q54716069 | ||
| THYMINE DIMERS AND INHIBITION OF DNA SYNTHESIS BY ULTRAVIOLET IRRADIATION OF CELLS. | Q54718372 | ||
| Extensive unwinding of the plasmid template during staged enzymatic initiation of DNA replication from the origin of the Escherichia coli chromosome. | Q54781669 | ||
| Repair in E. coli of transforming plasmid DNA damaged by psoralen plus near-ultraviolet irradiation. | Q54783361 | ||
| Efficiency of Escherichia coli repair processes on uv-damaged transforming plasmid DNA | Q70156228 | ||
| Effect of lipopolysaccharide mutations and temperature on plasmid transformation efficiency in Pseudomonas aeruginosa | Q70292550 | ||
| Mutagenic SOS repair of site-specific psoralen damage in plasmid pBR322 | Q70390153 | ||
| RecQ and RecJ process blocked replication forks prior to the resumption of replication in UV-irradiated Escherichia coli | Q73253665 | ||
| PCR-mediated gene replacement in Escherichia coli | Q73682242 | ||
| Action of RuvAB at replication fork structures | Q74499929 | ||
| One-step inactivation of chromosomal genes in Escherichia coli K-12 using PCR products | Q27860842 | ||
| SOS Repair Hypothesis: Phenomenology of an Inducible DNA Repair Which is Accompanied by Mutagenesis | Q28141658 | ||
| A new revision of the sequence of plasmid pBR322 | Q28289568 | ||
| An efficient recombination system for chromosome engineering in Escherichia coli | Q29615038 | ||
| Gene disruption in Escherichia coli: TcR and KmR cassettes with the option of Flp-catalyzed excision of the antibiotic-resistance determinant | Q29615039 | ||
| High efficiency transformation of E. coli by high voltage electroporation | Q29615278 | ||
| Involvement of nucleotide excision repair in a recombination-independent and error-prone pathway of DNA interstrand cross-link repair | Q30648450 | ||
| Induction of the SOS response increases the efficiency of global nucleotide excision repair of cyclobutane pyrimidine dimers, but not 6-4 photoproducts, in UV-irradiated Escherichia coli. | Q33732387 | ||
| Recovery of DNA replication in UV-irradiated Escherichia coli requires both excision repair and recF protein function | Q33991164 | ||
| Nucleotide excision repair or polymerase V-mediated lesion bypass can act to restore UV-arrested replication forks in Escherichia coli | Q34077275 | ||
| Therefore, what are recombination proteins there for? | Q34243612 | ||
| Participation of recombination proteins in rescue of arrested replication forks in UV-irradiated Escherichia coli need not involve recombination | Q34311635 | ||
| RuvAB and RecG are not essential for the recovery of DNA synthesis following UV-induced DNA damage in Escherichia coli | Q34644327 | ||
| Nascent DNA processing by RecJ favors lesion repair over translesion synthesis at arrested replication forks in Escherichia coli | Q34694893 | ||
| Genome stability and the processing of damaged replication forks by RecG. | Q34762160 | ||
| Characteristics of Some Multiply Recombination-Deficient Strains of Escherichia coli | Q35157370 | ||
| recF and recR are required for the resumption of replication at DNA replication forks in Escherichia coli | Q36104718 | ||
| Propagation of pSC101 plasmids defective in binding of integration host factor | Q36112988 | ||
| Identification and characterization of recD, a gene affecting plasmid maintenance and recombination in Escherichia coli | Q36249724 | ||
| Homologous recombination between plasmids in mammalian cells can be enhanced by treatment of input DNA | Q36261445 | ||
| Exonucleases I, III, and V are required for stability of ColE1-related plasmids in Escherichia coli | Q36297616 | ||
| Repair and mutagenesis of plasmid DNA modified by ultraviolet irradiation or N-acetoxy-N-2-acetylaminofluorene | Q36302391 | ||
| Inducibility of a gene product required for UV and chemical mutagenesis in Escherichia coli | Q36369577 | ||
| Plasmid uptake by bacteria: a comparison of methods and efficiencies | Q37496825 | ||
| Initiation of enzymatic replication at the origin of the Escherichia coli chromosome: primase as the sole priming enzyme | Q37691147 | ||
| Opening of the replication origin of Escherichia coli by DnaA protein with protein HU or IHF. | Q38324468 | ||
| DNA polymerase II (polB) is involved in a new DNA repair pathway for DNA interstrand cross-links in Escherichia coli | Q39495577 | ||
| Use of bacteriophage lambda recombination functions to promote gene replacement in Escherichia coli | Q39565526 | ||
| The RuvABC proteins and Holliday junction processing in Escherichia coli | Q39840417 | ||
| Conjugational recombination in resolvase-deficient ruvC mutants of Escherichia coli K-12 depends on recG | Q39942962 | ||
| Chromosomal genes essential for stable maintenance of the mini-F plasmid in Escherichia coli. | Q39954877 | ||
| P4510 | describes a project that uses | ImageQuant | Q112270642 |
| P433 | issue | 2 | |
| P921 | main subject | Escherichia coli | Q25419 |
| P304 | page(s) | 113-124 | |
| P577 | publication date | 2012-04-19 | |
| P1433 | published in | Plasmid | Q15754448 |
| P1476 | title | Inefficient replication reduces RecA-mediated repair of UV-damaged plasmids introduced into competent Escherichia coli | |
| P478 | volume | 68 |
| Q41537227 | Cho Endonuclease Functions during DNA Interstrand Cross-Link Repair in Escherichia coli |
| Q51539028 | Inducing a Site Specific Replication Blockage in E. coli Using a Fluorescent Repressor Operator System. |
| Q96431880 | Strand with mutagenic lesion is preferentially used as a template in the region of a bi-stranded clustered DNA damage site in Escherichia coli |
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