| scholarly article | Q13442814 |
| P50 | author | Rachel A. Henry Bonami | Q43108114 |
| P2093 | author name string | James W Thomas | |
| P2860 | cites work | Basal immunoglobulin signaling actively maintains developmental stage in immature B cells | Q24795003 |
| Receptor editing in a transgenic mouse model: site, efficiency, and role in B cell tolerance and antibody diversification | Q28259085 | ||
| Receptor editing is the main mechanism of B cell tolerance toward membrane antigens | Q28262805 | ||
| Peripheral B cell maturation. II. Heat-stable antigen(hi) splenic B cells are an immature developmental intermediate in the production of long-lived marrow-derived B cells | Q28264835 | ||
| Efficient in vivo manipulation of mouse genomic sequences at the zygote stage | Q29547306 | ||
| Predominant autoantibody production by early human B cell precursors | Q29619656 | ||
| Anergy and not clonal ignorance determines the fate of B cells that recognize a physiological autoantigen | Q32024493 | ||
| The use of phage display to distinguish insulin autoantibody (IAA) from insulin antibody (IA) idiotypes | Q33187255 | ||
| Toxin-coupled MHC class I tetramers can specifically ablate autoreactive CD8+ T cells and delay diabetes in nonobese diabetic mice | Q33844333 | ||
| Bruton's tyrosine kinase promotes persistence of mature anti-insulin B cells. | Q33852815 | ||
| B cell development in the spleen takes place in discrete steps and is determined by the quality of B cell receptor-derived signals. | Q33869680 | ||
| Vkappa polymorphisms in NOD mice are spread throughout the entire immunoglobulin kappa locus and are shared by other autoimmune strains | Q34267403 | ||
| Clonal deletion of autoreactive B lymphocytes in bone marrow chimeras | Q34313426 | ||
| Age of islet autoantibody appearance and mean levels of insulin, but not GAD or IA-2 autoantibodies, predict age of diagnosis of type 1 diabetes: diabetes autoimmunity study in the young | Q35043330 | ||
| The PTPN22 allele encoding an R620W variant interferes with the removal of developing autoreactive B cells in humans | Q35187072 | ||
| Selective deletion of antigen-specific CD8+ T cells by MHC class I tetramers coupled to the type I ribosome-inactivating protein saporin | Q35751049 | ||
| Autoantigen-specific B-cell depletion overcomes failed immune tolerance in type 1 diabetes | Q36109213 | ||
| Tissue-type plasminogen activator promotes murine myofibroblast activation through LDL receptor-related protein 1-mediated integrin signaling | Q36151652 | ||
| Receptor editing: an approach by autoreactive B cells to escape tolerance | Q36361497 | ||
| Receptor editing in self-reactive bone marrow B cells | Q36361578 | ||
| B lymphocytes may escape tolerance by revising their antigen receptors | Q36361618 | ||
| B cells are exquisitely sensitive to central tolerance and receptor editing induced by ultralow affinity, membrane-bound antigen. | Q36367608 | ||
| Bruton's tyrosine kinase regulates the activation of gene rearrangements at the lambda light chain locus in precursor B cells in the mouse | Q36369010 | ||
| Autoreactive B cells in the marginal zone that express dual receptors | Q36369869 | ||
| B cell receptor-independent stimuli trigger immunoglobulin (Ig) class switch recombination and production of IgG autoantibodies by anergic self-reactive B cells. | Q36370721 | ||
| Defective B cell tolerance checkpoints in systemic lupus erythematosus | Q36402748 | ||
| Impaired early B cell tolerance in patients with rheumatoid arthritis. | Q36402969 | ||
| Effect of rituximab on human in vivo antibody immune responses | Q36860483 | ||
| B lymphocyte "original sin" in the bone marrow enhances islet autoreactivity in type 1 diabetes-prone nonobese diabetic mice | Q36920508 | ||
| RS rearrangement frequency as a marker of receptor editing in lupus and type 1 diabetes | Q37019638 | ||
| Early expression of antiinsulin autoantibodies of humans and the NOD mouse: evidence for early determination of subsequent diabetes | Q37114100 | ||
| Basal B cell receptor-directed phosphatidylinositol 3-kinase signaling turns off RAGs and promotes B cell-positive selection. | Q37183404 | ||
| B cell selection defects underlie the development of diabetogenic APCs in nonobese diabetic mice | Q37220772 | ||
| CXCL13 blockade disrupts B lymphocyte organization in tertiary lymphoid structures without altering B cell receptor bias or preventing diabetes in nonobese diabetic mice | Q37299504 | ||
| Functional silencing is initiated and maintained in immature anti-insulin B cells | Q37353561 | ||
| B cell receptor basal signaling regulates antigen-induced Ig light chain rearrangements | Q38292514 | ||
| Central tolerance regulates B cells reactive with Goodpasture antigen alpha3(IV)NC1 collagen | Q39926777 | ||
| Affinity dependence of the B cell response to antigen: a threshold, a ceiling, and the importance of off-rate | Q41027647 | ||
| BCR ligation induces receptor editing in IgM+IgD- bone marrow B cells in vitro | Q41118611 | ||
| Elimination from peripheral lymphoid tissues of self-reactive B lymphocytes recognizing membrane-bound antigens | Q41170875 | ||
| Murine lambda gene rearrangements: the stochastic model prevails over the ordered model | Q41202410 | ||
| Altered immunoglobulin expression and functional silencing of self-reactive B lymphocytes in transgenic mice. | Q41423746 | ||
| Glucose homeostasis in the nonobese diabetic mouse at the prediabetic stage | Q42451830 | ||
| Tertiary lymphoid structures in the pancreas promote selection of B lymphocytes in autoimmune diabetes | Q42617543 | ||
| Multiple germline kappa light chains generate anti-insulin B cells in nonobese diabetic mice | Q42661803 | ||
| VH and VL gene usage by murine IgG antibodies that bind autologous insulin. | Q42800281 | ||
| Binding characteristics and crossreactivity of insulin autoantibodies and insulin antibodies directed to three different insulin molecules. | Q51701636 | ||
| Uncoupling of anergy from developmental arrest in anti-insulin B cells supports the development of autoimmune diabetes. | Q52060984 | ||
| Mapping epitopes on the insulin molecule using monoclonal antibodies | Q52826827 | ||
| Analysis of VH gene utilisation in the non-obese diabetic mouse. | Q52882960 | ||
| Cutting edge: impaired transitional B cell production and selection in the nonobese diabetic mouse. | Q53619792 | ||
| Differences in epitope restriction of autoantibodies to native human insulin (IAA) and antibodies to heterologous insulin (IA). | Q53884125 | ||
| Binding of insulin to its receptor impairs recognition by monoclonal anti-insulin antibodies. | Q53909829 | ||
| Somatically mutated B cell pool provides precursors for insulin antibodies | Q56903328 | ||
| Rituximab, B-Lymphocyte Depletion, and Preservation of Beta-Cell Function | Q57491711 | ||
| Continued RAG expression in late stages of B cell development and no apparent re-induction after immunizion | Q59066328 | ||
| Clonal deletion of B lymphocytes in a transgenic mouse bearing anti-MHC class I antibody genes | Q59098044 | ||
| Longitudinal epitope analysis of insulin-binding antibodies in type 1 diabetes | Q61861585 | ||
| Insulin Antibodies in Insulin-Dependent Diabetics Before Insulin Treatment | Q70311233 | ||
| Development of myelin oligodendrocyte glycoprotein autoreactive transgenic B lymphocytes: receptor editing in vivo after encounter of a self-antigen distinct from myelin oligodendrocyte glycoprotein | Q73113005 | ||
| Editing disease-associated autoantibodies | Q73115270 | ||
| Ig light chain receptor editing in anergic B cells | Q73306765 | ||
| A functional B cell receptor transgene allows efficient IL-7-independent maturation of B cell precursors | Q73535809 | ||
| B cell specificity contributes to the outcome of diabetes in nonobese diabetic mice | Q77136854 | ||
| The efficiency of B cell receptor (BCR) editing is dependent on BCR light chain rearrangement status | Q77879751 | ||
| Coordinate regulation of RAG1 and RAG2 by cell type-specific DNA elements 5' of RAG2 | Q78121917 | ||
| Generation of peripheral B cells occurs via two spatially and temporally distinct pathways | Q79359570 | ||
| In vivo assessment of the relative contributions of deletion, anergy, and editing to B cell self-tolerance | Q80345129 | ||
| Receptor editing can lead to allelic inclusion and development of B cells that retain antibodies reacting with high avidity autoantigens | Q81322342 | ||
| Treatment with CD20-specific antibody prevents and reverses autoimmune diabetes in mice | Q95789881 | ||
| P433 | issue | 10 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | type-1 diabetes | Q124407 |
| P304 | page(s) | 4730-4741 | |
| P577 | publication date | 2015-10-02 | |
| P1433 | published in | Journal of Immunology | Q3521441 |
| P1476 | title | Targeting Anti-Insulin B Cell Receptors Improves Receptor Editing in Type 1 Diabetes-Prone Mice | |
| P478 | volume | 195 |
| Q89295096 | Anti-Insulin B Cells Are Poised for Antigen Presentation in Type 1 Diabetes | cites work | P2860 |
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