| scholarly article | Q13442814 |
| P2093 | author name string | Dima Youssef | |
| Charles E McCall | |||
| Mohamed El Gazzar | |||
| Zhi Q Yao | |||
| Clara McClure | |||
| Ekram Ali | |||
| P2860 | cites work | MicroRNAs: target recognition and regulatory functions | Q24609584 |
| Myeloid-derived suppressor cells as regulators of the immune system | Q28131637 | ||
| PI3K pathway alterations in cancer: variations on a theme | Q29615530 | ||
| A minicircuitry comprised of microRNA-223 and transcription factors NFI-A and C/EBPalpha regulates human granulopoiesis | Q29619201 | ||
| Reactivation of multiple viruses in patients with sepsis | Q33740315 | ||
| Sepsis-induced immunosuppression: from cellular dysfunctions to immunotherapy | Q33828888 | ||
| Roles of the NFI/CTF gene family in transcription and development | Q33930287 | ||
| Hepatic acute-phase proteins control innate immune responses during infection by promoting myeloid-derived suppressor cell function. | Q33979918 | ||
| p21(WAF1/Cip1): more than a break to the cell cycle? | Q34020123 | ||
| STAT3 Activation of miR-21 and miR-181b-1 via PTEN and CYLD Are Part of the Epigenetic Switch Linking Inflammation to Cancer | Q34088885 | ||
| MicroRNA 21 (miR-21) and miR-181b couple with NFI-A to generate myeloid-derived suppressor cells and promote immunosuppression in late sepsis | Q34298403 | ||
| The JAK/STAT signaling pathway | Q34305849 | ||
| A syndrome of multiorgan hyperplasia with features of gigantism, tumorigenesis, and female sterility in p27(Kip1)-deficient mice | Q34381923 | ||
| Hematopoietic stem cell quiescence maintained by p21cip1/waf1 | Q73532192 | ||
| Pathophysiology of sepsis | Q80210901 | ||
| A paradoxical role for myeloid-derived suppressor cells in sepsis and trauma | Q34699543 | ||
| NF-kappaB dysregulation in microRNA-146a-deficient mice drives the development of myeloid malignancies | Q35022054 | ||
| The Surgical Infection Society guidelines on antimicrobial therapy for intra-abdominal infections: an executive summary | Q35051051 | ||
| Loss of the miR-21 allele elevates the expression of its target genes and reduces tumorigenesis. | Q35064340 | ||
| Myeloid-derived suppressor cells: paradoxical roles in infection and immunity | Q35143001 | ||
| Epigenetics, bioenergetics, and microRNA coordinate gene-specific reprogramming during acute systemic inflammation. | Q35170021 | ||
| Transcriptional regulation of the cyclin-dependent kinase inhibitor 1A (p21) gene by NFI in proliferating human cells | Q35225482 | ||
| Hematopoietic stem-progenitor cells restore immunoreactivity and improve survival in late sepsis | Q35689431 | ||
| Inflammation and the reciprocal production of granulocytes and lymphocytes in bone marrow | Q35951254 | ||
| Immunosuppression in patients who die of sepsis and multiple organ failure | Q35991015 | ||
| Myeloid-derived suppressor cells evolve during sepsis and can enhance or attenuate the systemic inflammatory response | Q36018144 | ||
| The late phase of sepsis is characterized by an increased microbiological burden and death rate | Q36070469 | ||
| MyD88-dependent expansion of an immature GR-1(+)CD11b(+) population induces T cell suppression and Th2 polarization in sepsis | Q36229458 | ||
| The interplay between the master transcription factor PU.1 and miR-424 regulates human monocyte/macrophage differentiation. | Q36288893 | ||
| Cell cycle machinery and stroke. | Q36714695 | ||
| Adenosine negative feedback on A2A adenosine receptors mediates hyporesponsiveness in chronically septic mice | Q36957177 | ||
| Myeloid-derived suppressor cells: more mechanisms for inhibiting antitumor immunity | Q36996588 | ||
| Immunosuppression in sepsis: a novel understanding of the disorder and a new therapeutic approach | Q37233354 | ||
| Gene regulatory networks directing myeloid and lymphoid cell fates within the immune system | Q37260838 | ||
| Loss of duplexmiR-223 (5p and 3p) aggravates myocardial depression and mortality in polymicrobial sepsis. | Q37647960 | ||
| The new normal: immunomodulatory agents against sepsis immune suppression | Q37686624 | ||
| Myeloid-derived suppressor cell heterogeneity and subset definition. | Q37695457 | ||
| Gene-Specific Epigenetic Regulation in Serious Infections with Systemic Inflammation | Q37781968 | ||
| Molecular mechanisms regulating myeloid-derived suppressor cell differentiation and function | Q37808561 | ||
| Myeloid derived suppressor cells and their role in diseases. | Q38081474 | ||
| Circulating cytokine/inhibitor profiles reshape the understanding of the SIRS/CARS continuum in sepsis and predict mortality. | Q38483410 | ||
| MicroRNA-494 is required for the accumulation and functions of tumor-expanded myeloid-derived suppressor cells via targeting of PTEN. | Q39356550 | ||
| A deuterated analog of dasatinib disrupts cell cycle progression and displays anti-non-small cell lung cancer activity in vitro and in vivo | Q39391009 | ||
| PI3K/Akt inhibition modulates multidrug resistance and activates NF-kappaB in murine lymphoma cell lines. | Q39959297 | ||
| All-trans-retinoic acid eliminates immature myeloid cells from tumor-bearing mice and improves the effect of vaccination. | Q42605441 | ||
| After Xigris, researchers look to new targets to combat sepsis | Q44161242 | ||
| Gr-1+ myeloid cells derived from tumor-bearing mice inhibit primary T cell activation induced through CD3/CD28 costimulation | Q45869019 | ||
| p53-dependent inhibition of cyclin-dependent kinase activities in human fibroblasts during radiation-induced G1 arrest. | Q45993768 | ||
| Interaction between the innate and adaptive immune systems is required to survive sepsis and control inflammation after injury | Q48971002 | ||
| Matrine inhibits breast cancer growth via miR-21/PTEN/Akt pathway in MCF-7 cells. | Q53154822 | ||
| The search for effective therapy for sepsis: back to the drawing board? | Q57151020 | ||
| Sepsis studies need new direction | Q64132695 | ||
| P433 | issue | 1 | |
| P921 | main subject | sepsis | Q183134 |
| cell differentiation | Q210861 | ||
| P304 | page(s) | 201-211 | |
| P577 | publication date | 2015-08-10 | |
| P1433 | published in | Journal of Leukocyte Biology | Q1524048 |
| P1476 | title | NFI-A disrupts myeloid cell differentiation and maturation in septic mice | |
| P478 | volume | 99 |
| Q40365847 | A natural food sweetener with anti-pancreatic cancer properties |
| Q40078180 | Circulating miRNAs in sepsis-A network under attack: An in-silico prediction of the potential existence of miRNA sponges in sepsis. |
| Q47766404 | Expression of C/EBPβ in myeloid progenitors during sepsis promotes immunosuppression |
| Q92031701 | Ferumoxytol Attenuates the Function of MDSCs to Ameliorate LPS-Induced Immunosuppression in Sepsis |
| Q50669666 | Frontline Science: Myeloid cell-specific deletion of Cebpb decreases sepsis-induced immunosuppression in mice. |
| Q47331514 | Intracellular S100A9 Promotes Myeloid-Derived Suppressor Cells during Late Sepsis. |
| Q40342586 | Myeloid Cell-Specific Knockout of NFI-A Improves Sepsis Survival |
| Q64094878 | Myeloid-Derived Suppressor Cells in Sepsis |
| Q49666322 | Nfia deletion in myeloid cells blocks expansion of myeloid-derived suppressor cells during sepsis |
| Q37553132 | Stat3 and C/EBPβ synergize to induce miR-21 and miR-181b expression during sepsis |
| Q92650586 | The CCAAT/Enhancer-Binding Protein Family: Its Roles in MDSC Expansion and Function |
| Q37444472 | The involvement of regulatory non-coding RNAs in sepsis: a systematic review. |
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