scholarly article | Q13442814 |
P2093 | author name string | Lei Li | |
Jiadong Wang | |||
Junjie Chen | |||
Wenqi Wang | |||
Gargi Ghosal | |||
Xi Shen | |||
Justin Wai-Chung Leung | |||
P2860 | cites work | Chk1 inhibits replication factory activation but allows dormant origin firing in existing factories. | Q39617777 |
Loss of ATRX leads to chromosome cohesion and congression defects | Q41825660 | ||
The MRX complex stabilizes the replisome independently of the S phase checkpoint during replication stress | Q43125340 | ||
ATR-X syndrome protein targets tandem repeats and influences allele-specific expression in a size-dependent manner. | Q47407852 | ||
Synchronization of HeLa cells. | Q53213847 | ||
Identification of acquired somatic mutations in the gene encoding chromatin-remodeling factor ATRX in the α-thalassemia myelodysplasia syndrome (ATMDS) | Q63248767 | ||
Promyelocytic leukemia nuclear bodies are predetermined processing sites for damaged DNA | Q79988209 | ||
Daxx is an H3.3-specific histone chaperone and cooperates with ATRX in replication-independent chromatin assembly at telomeres | Q24292850 | ||
Proliferating cell nuclear antigen (PCNA)-binding protein C1orf124 is a regulator of translesion synthesis | Q24296912 | ||
RNF8 transduces the DNA-damage signal via histone ubiquitylation and checkpoint protein assembly | Q24300428 | ||
Fanconi anemia (FA) binding protein FAAP20 stabilizes FA complementation group A (FANCA) and participates in interstrand cross-link repair | Q24306790 | ||
Mutations in a putative global transcriptional regulator cause X-linked mental retardation with alpha-thalassemia (ATR-X syndrome) | Q24311995 | ||
The death-associated protein DAXX is a novel histone chaperone involved in the replication-independent deposition of H3.3 | Q24315621 | ||
CDK targeting of NBS1 promotes DNA-end resection, replication restart and homologous recombination | Q24318939 | ||
The annealing helicase HARP is recruited to DNA repair sites via an interaction with RPA | Q24319860 | ||
Polyubiquitinated PCNA recruits the ZRANB3 translocase to maintain genomic integrity after replication stress | Q24338351 | ||
The HARP-like domain-containing protein AH2/ZRANB3 binds to PCNA and participates in cellular response to replication stress | Q24338440 | ||
ZRANB3 is a structure-specific ATP-dependent endonuclease involved in replication stress response | Q24339475 | ||
Loss of maternal ATRX results in centromere instability and aneuploidy in the mammalian oocyte and pre-implantation embryo | Q27345576 | ||
ATRX ADD domain links an atypical histone methylation recognition mechanism to human mental-retardation syndrome | Q27670417 | ||
Sensing DNA damage through ATRIP recognition of RPA-ssDNA complexes | Q27860662 | ||
ATRX interacts with H3.3 in maintaining telomere structural integrity in pluripotent embryonic stem cells | Q28114801 | ||
Distinct factors control histone variant H3.3 localization at specific genomic regions | Q28275277 | ||
ATRX encodes a novel member of the SNF2 family of proteins: mutations point to a common mechanism underlying the ATR-X syndrome | Q28299644 | ||
Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpoints | Q29617405 | ||
Cell cycle checkpoint signaling through the ATM and ATR kinases | Q29617837 | ||
Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication | Q29617915 | ||
The annealing helicase SMARCAL1 maintains genome integrity at stalled replication forks | Q33507753 | ||
The annealing helicase HARP protects stalled replication forks | Q33507756 | ||
Altered telomeres in tumors with ATRX and DAXX mutations | Q34196696 | ||
Hydroxyurea-stalled replication forks become progressively inactivated and require two different RAD51-mediated pathways for restart and repair. | Q34218130 | ||
Involvement of a chromatin remodeling complex in damage tolerance during DNA replication | Q34280451 | ||
Loss of ATRX, genome instability, and an altered DNA damage response are hallmarks of the alternative lengthening of telomeres pathway | Q34350929 | ||
ATR-X mutations cause impaired nuclear location and altered DNA binding properties of the XNP/ATR-X protein | Q35559646 | ||
Role of ATRX in chromatin structure and function: implications for chromosome instability and human disease | Q35661042 | ||
Replicon clusters are stable units of chromosome structure: evidence that nuclear organization contributes to the efficient activation and propagation of S phase in human cells | Q36255115 | ||
Understanding alpha-globin gene regulation: Aiming to improve the management of thalassemia | Q36337001 | ||
Compromised genomic integrity impedes muscle growth after Atrx inactivation. | Q36498031 | ||
PARP is activated at stalled forks to mediate Mre11-dependent replication restart and recombination. | Q37334922 | ||
Probing PML body function in ALT cells reveals spatiotemporal requirements for telomere recombination | Q37354145 | ||
SWI/SNF nucleosome remodellers and cancer. | Q37886668 | ||
P433 | issue | 9 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | Alpha-thalassemia mental retardation syndrome | Q2613277 |
P304 | page(s) | 6342-6350 | |
P577 | publication date | 2013-01-16 | |
P1433 | published in | Journal of Biological Chemistry | Q867727 |
P1476 | title | Alpha thalassemia/mental retardation syndrome X-linked gene product ATRX is required for proper replication restart and cellular resistance to replication stress | |
P478 | volume | 288 |
Q92375621 | ATRX affects the repair of telomeric DSBs by promoting cohesion and a DAXX-dependent activity |
Q39071428 | ATRX and DAXX: Mechanisms and Mutations |
Q28116488 | ATRX binds to atypical chromatin domains at the 3' exons of zinc finger genes to preserve H3K9me3 enrichment |
Q24339453 | ATRX dysfunction induces replication defects in primary mouse cells |
Q41565938 | ATRX is a regulator of therapy induced senescence in human cells. |
Q37738658 | ATRX loss promotes tumor growth and impairs nonhomologous end joining DNA repair in glioma |
Q37008597 | ATRX mutations and glioblastoma: Impaired DNA damage repair, alternative lengthening of telomeres, and genetic instability. |
Q50302756 | ATRX promotes gene expression by facilitating transcriptional elongation through guanine-rich coding regions |
Q36140856 | ATRX represses alternative lengthening of telomeres |
Q47552952 | Alpha Thalassemia/Mental Retardation Syndrome X-Linked, the Alternative Lengthening of Telomere Phenotype, and Gliomagenesis: Current Understandings and Future Potential |
Q89622479 | Alternative Lengthening of Telomeres in Pediatric Cancer: Mechanisms to Therapies |
Q33558285 | Alternative lengthening of telomeres can be maintained by preferential elongation of lagging strands |
Q90346173 | Alternative lengthening of telomeres: from molecular mechanisms to therapeutic outlooks |
Q33983659 | Analysis of neonatal brain lacking ATRX or MeCP2 reveals changes in nucleosome density, CTCF binding and chromatin looping |
Q38690384 | Arabidopsis ATRX Modulates H3.3 Occupancy and Fine-Tunes Gene Expression. |
Q35171587 | CHK1-driven histone H3.3 serine 31 phosphorylation is important for chromatin maintenance and cell survival in human ALT cancer cells |
Q38240348 | Chromatin regulation of DNA damage repair and genome integrity in the central nervous system |
Q47095909 | Diagnosis and treatment of ALT tumors: is Trabectedin a new therapeutic option? |
Q38662646 | Emerging roles of ATRX in cancer |
Q64102392 | G-quadruplex DNA drives genomic instability and represents a targetable molecular abnormality in ATRX-deficient malignant glioma |
Q40292496 | Identification of epigenetic signature associated with alpha thalassemia/mental retardation X-linked syndrome |
Q90224942 | Mechanisms of DNA Replication and Repair: Insights from the Study of G-Quadruplexes |
Q37644439 | Mosaic expression of Atrx in the mouse central nervous system causes memory deficits. |
Q55456861 | Mutant ATRX: uncovering a new therapeutic target for glioma. |
Q98735817 | Neurodevelopmental Disorders Caused by Defective Chromatin Remodeling: Phenotypic Complexity Is Highlighted by a Review of ATRX Function |
Q91843058 | PML is recruited to heterochromatin during S phase and represses DAXX-mediated histone H3.3 chromatin assembly |
Q94048487 | Proteomic characterization of chromosomal common fragile site (CFS)-associated proteins uncovers ATRX as a regulator of CFS stability |
Q26785908 | Replication Stress: A Lifetime of Epigenetic Change |
Q64266001 | Rescue of collapsed replication forks is dependent on NSMCE2 to prevent mitotic DNA damage |
Q47806032 | Retinal interneuron survival requires non-cell-autonomous Atrx activity |
Q41924768 | Stalled replication forks within heterochromatin require ATRX for protection |
Q38856558 | Suppression of the alternative lengthening of telomere pathway by the chromatin remodelling factor ATRX. |
Q26751191 | The DNA fibre technique - tracking helicases at work |
Q42369808 | The Role of ATRX in Glioma Biology |
Q28071562 | The Role of ATRX in the Alternative Lengthening of Telomeres (ALT) Phenotype |
Q38795281 | The chromatin remodelling factor ATRX suppresses R-loops in transcribed telomeric repeats |
Q64387662 | The macroH2A1.2 histone variant links ATRX loss to alternative telomere lengthening |
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