scholarly article | Q13442814 |
P2093 | author name string | Masumi Abe | |
Yao Xu | |||
Katheryn Meek | |||
Eric Hendrickson | |||
Jessica A Neal | |||
P2860 | cites work | A vector based on the SV40 origin of replication and chromosomal S/MARs replicates episomally in CHO cells | Q24548417 |
ATM damage response and XLF repair factor are functionally redundant in joining DNA breaks | Q24596775 | ||
RNA-guided human genome engineering via Cas9 | Q24598394 | ||
A human XRCC4-XLF complex bridges DNA | Q24630031 | ||
XRCC4's interaction with XLF is required for coding (but not signal) end joining | Q24630098 | ||
Molecular Mechanism of V(D)J Recombination from Synaptic RAG1-RAG2 Complex Structures | Q27321738 | ||
Non-homologous end-joining partners in a helical dance: structural studies of XLF-XRCC4 interactions | Q27674365 | ||
Autophosphorylation of the catalytic subunit of the DNA-dependent protein kinase is required for efficient end processing during DNA double-strand break repair | Q27919641 | ||
DNA-PK autophosphorylation facilitates Artemis endonuclease activity | Q27919702 | ||
The catalytic subunit of DNA-dependent protein kinase regulates proliferation, telomere length, and genomic stability in human somatic cells | Q28115800 | ||
DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation | Q28206029 | ||
Roles of nonhomologous DNA end joining, V(D)J recombination, and class switch recombination in chromosomal translocations | Q28247857 | ||
The response to and repair of RAG-mediated DNA double-strand breaks | Q28256889 | ||
A severe combined immunodeficiency mutation in the mouse | Q28274531 | ||
The ataxia telangiectasia mutated kinase controls Igκ allelic exclusion by inhibiting secondary Vκ-to-Jκ rearrangements | Q36603553 | ||
53BP1 facilitates long-range DNA end-joining during V(D)J recombination | Q36682376 | ||
Complementary functions of ATM and H2AX in development and suppression of genomic instability | Q36756926 | ||
A DNA-PKcs mutation in a radiosensitive T-B- SCID patient inhibits Artemis activation and nonhomologous end-joining. | Q37036037 | ||
RAG: a recombinase diversified | Q37171099 | ||
MRN complex function in the repair of chromosomal Rag-mediated DNA double-strand breaks | Q37234327 | ||
Formation of dynamic gamma-H2AX domains along broken DNA strands is distinctly regulated by ATM and MDC1 and dependent upon H2AX densities in chromatin. | Q37347174 | ||
Aberrantly resolved RAG-mediated DNA breaks in Atm-deficient lymphocytes target chromosomal breakpoints in cis. | Q37416485 | ||
The RAG2 C-terminus and ATM protect genome integrity by controlling antigen receptor gene cleavage | Q37519459 | ||
DNA double-strand break repair in a cellular context | Q38196085 | ||
Functional overlaps between XLF and the ATM-dependent DNA double strand break response | Q38199848 | ||
V(D)J Recombination: Mechanism, Errors, and Fidelity | Q38535955 | ||
PRKDC mutations in a SCID patient with profound neurological abnormalities | Q38955848 | ||
DNA-PK is essential only for coding joint formation in V(D)J recombination | Q39724861 | ||
Deficiency in the catalytic subunit of DNA-dependent protein kinase causes down-regulation of ATM. | Q40449393 | ||
Unraveling the complexities of DNA-dependent protein kinase autophosphorylation | Q40603571 | ||
DNA uptake and repair enzyme access to transfected DNA is under reported by gene expression | Q40640079 | ||
SCID in Jack Russell terriers: a new animal model of DNA-PKcs deficiency | Q40787847 | ||
Signal joint formation is also impaired in DNA-dependent protein kinase catalytic subunit knockout cells | Q40848620 | ||
Analyses of TCRB rearrangements substantiate a profound deficit in recombination signal sequence joining in SCID foals: implications for the role of DNA-dependent protein kinase in V(D)J recombination | Q40905044 | ||
A kinase-negative mutation of DNA-PK(CS) in equine SCID results in defective coding and signal joint formation | Q41116024 | ||
V(D)J recombination in mammalian cell mutants defective in DNA double-strand break repair | Q41549015 | ||
Impairment of V(D)J recombination in double-strand break repair mutants | Q41560292 | ||
Not All SCID Pigs Are Created Equally: Two Independent Mutations in the Artemis Gene Cause SCID in Pigs. | Q41666062 | ||
DNA-PKcs and ATM co-regulate DNA double-strand break repair | Q42007528 | ||
Extrachromosomal DNA substrates in pre-B cells undergo inversion or deletion at immunoglobulin V-(D)-J joining signals | Q45246384 | ||
H2AX prevents DNA breaks from progressing to chromosome breaks and translocations | Q46904251 | ||
The defect in murine severe combined immune deficiency: joining of signal sequences but not coding segments in V(D)J recombination | Q48313594 | ||
DNA-PKcs mutations in dogs and horses: allele frequency and association with neoplasia | Q48316202 | ||
Severe combined immunodeficiency in Frisian Water Dogs caused by a RAG1 mutation | Q58243743 | ||
Rag mutations reveal robust alternative end joining | Q59098597 | ||
V(D)J recombination: a functional definition of the joining signals | Q69721561 | ||
Characterization of coding ends in thymocytes of scid mice: Implications for the mechanism of V(D)J recombination | Q71843533 | ||
A targeted DNA-PKcs-null mutation reveals DNA-PK-independent functions for KU in V(D)J recombination | Q77398644 | ||
RAG1 and RAG2 form a stable postcleavage synaptic complex with DNA containing signal ends in V(D)J recombination | Q28307236 | ||
Targeted disruption of the catalytic subunit of the DNA-PK gene in mice confers severe combined immunodeficiency and radiosensitivity | Q28506163 | ||
RAG-1 and ATM coordinate monoallelic recombination and nuclear positioning of immunoglobulin loci | Q28508203 | ||
Crystal structure of the V(D)J recombinase RAG1-RAG2 | Q30622421 | ||
Molecular and biochemical characterisation of DNA-dependent protein kinase-defective rodent mutant irs-20 | Q32082716 | ||
ATM prevents the persistence and propagation of chromosome breaks in lymphocytes | Q33289263 | ||
RAG2's acidic hinge restricts repair-pathway choice and promotes genomic stability | Q33558920 | ||
Histone H2AX stabilizes broken DNA strands to suppress chromosome breaks and translocations during V(D)J recombination | Q33591110 | ||
Analysis of variable (diversity) joining recombination in DNAdependent protein kinase (DNA-PK)-deficient mice reveals DNA-PK-independent pathways for both signal and coding joint formation | Q33597709 | ||
Both V(D)J recombination and radioresistance require DNA-PK kinase activity, though minimal levels suffice for V(D)J recombination | Q33616301 | ||
Equine severe combined immunodeficiency: a defect in V(D)J recombination and DNA-dependent protein kinase activity. | Q33702830 | ||
Intermediates in V(D)J recombination: a stable RAG1/2 complex sequesters cleaved RSS ends. | Q33948953 | ||
Requirement for the kinase activity of human DNA-dependent protein kinase catalytic subunit in DNA strand break rejoining | Q33958394 | ||
ATM-deficient thymic lymphoma is associated with aberrant tcrd rearrangement and gene amplification | Q33979941 | ||
The ataxia telangiectasia mutated and cyclin D3 proteins cooperate to help enforce TCRβ and IgH allelic exclusion | Q34146558 | ||
Defective DNA-dependent protein kinase activity is linked to V(D)J recombination and DNA repair defects associated with the murine scid mutation. | Q34320792 | ||
XLF regulates filament architecture of the XRCC4·ligase IV complex | Q34433470 | ||
ATM stabilizes DNA double-strand-break complexes during V(D)J recombination. | Q34541828 | ||
Ataxia telangiectasia-mutated protein and DNA-dependent protein kinase have complementary V(D)J recombination functions | Q34549990 | ||
Ataxia telangiectasia mutated (Atm) and DNA-PKcs kinases have overlapping activities during chromosomal signal joint formation | Q34550115 | ||
V(D)J recombination: RAG proteins, repair factors, and regulation | Q34667417 | ||
XR-C1, a new CHO cell mutant which is defective in DNA-PKcs, is impaired in both V(D)J coding and signal joint formation | Q34669732 | ||
Aberrant V(D)J recombination in ataxia telangiectasia mutated-deficient lymphocytes is dependent on nonhomologous DNA end joining. | Q34805011 | ||
DNA-dependent protein kinase activity is absent in xrs-6 cells: implications for site-specific recombination and DNA double-strand break repair | Q34868235 | ||
Catalytic subunit of DNA-dependent protein kinase: impact on lymphocyte development and tumorigenesis | Q34992049 | ||
Inhibition of homologous recombination by DNA-dependent protein kinase requires kinase activity, is titratable, and is modulated by autophosphorylation | Q35077127 | ||
H2AX prevents CtIP-mediated DNA end resection and aberrant repair in G1-phase lymphocytes. | Q35148396 | ||
Unique and redundant functions of ATM and DNA-PKcs during V(D)J recombination | Q35159001 | ||
PRKDC mutations associated with immunodeficiency, granuloma, and autoimmune regulator-dependent autoimmunity. | Q35806314 | ||
N-terminal constraint activates the catalytic subunit of the DNA-dependent protein kinase in the absence of DNA or Ku. | Q35888831 | ||
Linking double-stranded DNA breaks to the recombination activating gene complex directs repair to the nonhomologous end-joining pathway | Q36089263 | ||
Redundant and nonredundant functions of ATM and H2AX in αβ T-lineage lymphocytes | Q36106641 | ||
Defects in coding joint formation in vivo in developing ATM-deficient B and T lymphocytes. | Q36229444 | ||
Biochemistry of V(D)J recombination. | Q36397181 | ||
P433 | issue | 7 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 3032-3042 | |
P577 | publication date | 2016-02-26 | |
P1433 | published in | Journal of Immunology | Q3521441 |
P1476 | title | Restoration of ATM Expression in DNA-PKcs-Deficient Cells Inhibits Signal End Joining | |
P478 | volume | 196 |