| scholarly article | Q13442814 |
| P6179 | Dimensions Publication ID | 1002698945 |
| P356 | DOI | 10.1038/NN1857 |
| P698 | PubMed publication ID | 17387329 |
| P5875 | ResearchGate publication ID | 6420907 |
| P2093 | author name string | Stephen G Waxman | |
| P2860 | cites work | Sodium channel mutations in epilepsy and other neurological disorders | Q36216288 |
| Erythermalgia: molecular basis for an inherited pain syndrome | Q36309445 | ||
| Nav1.3 sodium channels: rapid repriming and slow closed-state inactivation display quantitative differences after expression in a mammalian cell line and in spinal sensory neurons. | Q40788199 | ||
| Slow closed-state inactivation: a novel mechanism underlying ramp currents in cells expressing the hNE/PN1 sodium channel. | Q42686664 | ||
| Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain | Q44805364 | ||
| Open-channel block by the cytoplasmic tail of sodium channel beta4 as a mechanism for resurgent sodium current | Q45233496 | ||
| Abnormal Purkinje cell activity in vivo in experimental allergic encephalomyelitis | Q47670118 | ||
| Structure and function of a novel voltage-gated, tetrodotoxin-resistant sodium channel specific to sensory neurons. | Q48065579 | ||
| Expression of Nav1.8 sodium channels perturbs the firing patterns of cerebellar Purkinje cells | Q48422070 | ||
| Rescue of α-SNS Sodium Channel Expression in Small Dorsal Root Ganglion Neurons After Axotomy by Nerve Growth Factor In Vivo | Q48941318 | ||
| A gain-of-function mutation in the sodium channel gene Scn2a results in seizures and behavioral abnormalities | Q49049294 | ||
| mRNA for NGF and p75 in the central nervous system of rats affected by experimental allergic encephalomyelitis | Q49127983 | ||
| The TTX-resistant sodium channel Nav1.8 (SNS/PN3): expression and correlation with membrane properties in rat nociceptive primary afferent neurons | Q51295679 | ||
| alpha-SNS produces the slow TTX-resistant sodium current in large cutaneous afferent DRG neurons | Q51386667 | ||
| Downregulation of tetrodotoxin-resistant sodium currents and upregulation of a rapidly repriming tetrodotoxin-sensitive sodium current in small spinal sensory neurons after nerve injury. | Q51542388 | ||
| Ionic mechanisms of anoxic injury in mammalian CNS white matter: role of Na+ channels and Na(+)-Ca2+ exchanger | Q51697656 | ||
| Upregulation and colocalization of p75 and Nav1.8 in Purkinje neurons in experimental autoimmune encephalomyelitis | Q53731135 | ||
| Genetic modifiers affecting severity of epilepsy caused by mutation of sodium channel Scn2a | Q54634416 | ||
| A new molecular mechanism for severe myoclonic epilepsy of infancy: Exonic deletions in SCN1A | Q58417059 | ||
| Sensory and electrophysiological properties of guinea-pig sensory neurones expressing Nav 1.7 (PN1) Na+ channel alpha subunit protein | Q78793355 | ||
| Mutations of SCN1A, encoding a neuronal sodium channel, in two families with GEFS+2 | Q22253421 | ||
| A quantitative description of membrane current and its application to conduction and excitation in nerve | Q22337072 | ||
| Fibroblast growth factor homologous factor 2B: association with Nav1.6 and selective colocalization at nodes of Ranvier of dorsal root axons | Q24300238 | ||
| De novo mutations in the sodium-channel gene SCN1A cause severe myoclonic epilepsy of infancy | Q24533495 | ||
| A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons | Q24546007 | ||
| Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain | Q24562888 | ||
| Mutations in SCN9A, encoding a sodium channel alpha subunit, in patients with primary erythermalgia | Q24675844 | ||
| Epilepsy-associated dysfunction in the voltage-gated neuronal sodium channel SCN1A | Q28191292 | ||
| Mutations of sodium channel alpha subunit type 1 (SCN1A) in intractable childhood epilepsies with frequent generalized tonic-clonic seizures | Q28207332 | ||
| Molecular basis of an inherited epilepsy | Q28207595 | ||
| Pathomechanisms in channelopathies of skeletal muscle and brain | Q28246271 | ||
| A nonsense mutation of the sodium channel gene SCN2A in a patient with intractable epilepsy and mental decline | Q28251184 | ||
| An SCN9A channelopathy causes congenital inability to experience pain | Q28278844 | ||
| International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels | Q28289125 | ||
| A novel epilepsy mutation in the sodium channel SCN1A identifies a cytoplasmic domain for beta subunit interaction | Q28291611 | ||
| An epilepsy mutation in the sodium channel SCN1A that decreases channel excitability | Q28300929 | ||
| Identification of PN1, a predominant voltage-dependent sodium channel expressed principally in peripheral neurons | Q28569751 | ||
| A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons | Q28570486 | ||
| Reduced sodium current in GABAergic interneurons in a mouse model of severe myoclonic epilepsy in infancy | Q28585126 | ||
| Impaired neurogenic control of skin perfusion in erythromelalgia | Q32068468 | ||
| Erythromelalgia: vasculopathy, neuropathy, or both? A prospective study of vascular and neurophysiologic studies in erythromelalgia | Q33194068 | ||
| A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of chronic pain | Q33679477 | ||
| Contribution of Na(v)1.8 sodium channels to action potential electrogenesis in DRG neurons | Q34086296 | ||
| Roles of tetrodotoxin (TTX)-sensitive Na+ current, TTX-resistant Na+ current, and Ca2+ current in the action potentials of nociceptive sensory neurons. | Q34160850 | ||
| Electrophysiological properties of mutant Nav1.7 sodium channels in a painful inherited neuropathy. | Q34350866 | ||
| Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons | Q34426737 | ||
| Sensory neuron-specific sodium channel SNS is abnormally expressed in the brains of mice with experimental allergic encephalomyelitis and humans with multiple sclerosis | Q35325861 | ||
| Inherited Channelopathies Associated with Epilepsy | Q35962079 | ||
| P433 | issue | 4 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | phenotype | Q104053 |
| P304 | page(s) | 405-409 | |
| P577 | publication date | 2007-04-01 | |
| P13046 | publication type of scholarly work | review article | Q7318358 |
| P1433 | published in | Nature Neuroscience | Q1535359 |
| P1476 | title | Channel, neuronal and clinical function in sodium channelopathies: from genotype to phenotype | |
| P478 | volume | 10 |
| Q54373490 | Arachidonic acid modulates Na+ currents by non-metabolic and metabolic pathways in rat cerebellar granule cells |
| Q41933122 | Autism and Epilepsy: Exploring the Relationship Using Experimental Models |
| Q33889459 | Blockade of Persistent Sodium Currents Contributes to the Riluzole-Induced Inhibition of Spontaneous Activity and Oscillations in Injured DRG Neurons |
| Q33563907 | Calpain mediates proteolysis of the voltage-gated sodium channel alpha-subunit |
| Q35434773 | Channelopathies: Summary of the hot topic keynotes session |
| Q41968217 | Colonic inflammation up‐regulates voltage‐gated sodium channels in bladder sensory neurons via activation of peripheral transient potential vanilloid 1 receptors |
| Q38722862 | Conservation of alternative splicing in sodium channels reveals evolutionary focus on release from inactivation and structural insights into gating |
| Q30855778 | DDESC: Dragon database for exploration of sodium channels in human |
| Q48108091 | Distribution and function of voltage-gated sodium channels in the nervous system |
| Q39696103 | Effects of ranolazine on wild-type and mutant hNav1.7 channels and on DRG neuron excitability |
| Q34562987 | Exposure to extremely low-frequency electromagnetic fields modulates Na+ currents in rat cerebellar granule cells through increase of AA/PGE2 and EP receptor-mediated cAMP/PKA pathway. |
| Q33723915 | Genetic basis of pediatric epilepsy syndromes |
| Q37784196 | Genetic disorders of ion channels |
| Q37439341 | HSV vector-mediated modification of primary nociceptor afferents: an approach to inhibit chronic pain |
| Q38182564 | Herpes simplex virus-based nerve targeting gene therapy in pain management |
| Q37511682 | Inherited pain: sodium channel Nav1.7 A1632T mutation causes erythromelalgia due to a shift of fast inactivation. |
| Q30457731 | Ion channels and schizophrenia: a gene set-based analytic approach to GWAS data for biological hypothesis testing |
| Q36565208 | Lack of transient receptor potential vanilloid 1 channel modulates the development of neurogenic bladder dysfunction induced by cross-sensitization in afferent pathways |
| Q28540363 | Locating the route of entry and binding sites of benzocaine and phenytoin in a bacterial voltage gated sodium channel |
| Q84747700 | Membrane-select regulation of cardiac Nav channel isoforms |
| Q34585790 | Mutations in sodium-channel gene SCN9A cause a spectrum of human genetic pain disorders |
| Q37955681 | Natural and artificial ion channels for biosensing platforms |
| Q37254618 | Nociceptor sensitization in pain pathogenesis |
| Q36916035 | Paroxysmal extreme pain disorder M1627K mutation in human Nav1.7 renders DRG neurons hyperexcitable |
| Q35707037 | Role of calcium and calpain in the downregulation of voltage-gated sodium channel expression by the pyrethroid pesticide deltamethrin |
| Q28116090 | SCN5A variant that blocks fibroblast growth factor homologous factor regulation causes human arrhythmia |
| Q36908837 | Seizure termination by acidosis depends on ASIC1a |
| Q37576848 | Severe epilepsy syndromes of early childhood: the link between genetics and pathophysiology with a focus on SCN1A mutations |
| Q37605350 | Sodium channel blockers for the treatment of neuropathic pain |
| Q42153853 | Strength-Duration Time Constant in Peripheral Nerve: No Abnormality in Multiple Sclerosis |
| Q34013724 | Systems approach to understanding electromechanical activity in the human heart: a national heart, lung, and blood institute workshop summary. |
| Q37614747 | The beta-secretase enzyme BACE in health and Alzheimer's disease: regulation, cell biology, function, and therapeutic potential |
| Q39081430 | The influence of sodium on pathophysiology of multiple sclerosis. |
| Q27002550 | Towards a Unified Theory of Calmodulin Regulation (Calmodulation) of Voltage-Gated Calcium and Sodium Channels |
| Q36995561 | Trafficking and cellular distribution of voltage-gated sodium channels. |
| Q84255216 | Transfection of rat or mouse neurons by biolistics or electroporation |
| Q38239812 | Translational pain research: Lessons from genetics and genomics |
| Q61866177 | Voltage-gated Na+ channels: potential for beta subunits as therapeutic targets |
| Q35814531 | cAMP/PKA Pathways and S56 Phosphorylation Are Involved in AA/PGE2-Induced Increases in rNaV1.4 Current |
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