scholarly article | Q13442814 |
P50 | author | Tom Misteli | Q7816902 |
Vassilis Roukos | Q59696826 | ||
Bharat Burman | Q125310126 | ||
P2860 | cites work | Chromosome territories, nuclear architecture and gene regulation in mammalian cells | Q22122379 |
The genomic complexity of primary human prostate cancer | Q24617949 | ||
Tissue-specific spatial organization of genomes | Q24805135 | ||
The plant homeodomain finger of RAG2 recognizes histone H3 methylated at both lysine-4 and arginine-2 | Q27649107 | ||
RAG2 PHD finger couples histone H3 lysine 4 trimethylation with V(D)J recombination | Q27649126 | ||
Colocalization of multiple DNA double-strand breaks at a single Rad52 repair centre | Q27940019 | ||
Analysis of the V(D)J recombination efficiency at lymphoid chromosomal translocation breakpoints | Q28199398 | ||
Structural organization of the bcr gene and its role in the Ph' translocation | Q28286576 | ||
A chromatin-wide transition to H4K20 monomethylation impairs genome integrity and programmed DNA rearrangements in the mouse | Q28289637 | ||
Recombination centres and the orchestration of V(D)J recombination | Q28307066 | ||
Letter: A new consistent chromosomal abnormality in chronic myelogenous leukaemia identified by quinacrine fluorescence and Giemsa staining | Q29614339 | ||
Changes in chromatin structure and mobility in living cells at sites of DNA double-strand breaks | Q30480374 | ||
Intermingling of chromosome territories in interphase suggests role in translocations and transcription-dependent associations | Q33240304 | ||
Over half of breakpoints in gene pairs involved in cancer-specific recurrent translocations are mapped to human chromosomal fragile sites | Q33404454 | ||
Evidence for direct involvement of epirubicin in the formation of chromosomal translocations in t(15;17) therapy-related acute promyelocytic leukemia | Q33597526 | ||
DNA breaks at fragile sites generate oncogenic RET/PTC rearrangements in human thyroid cells | Q33792630 | ||
Higher-order genome organization in human disease | Q34014679 | ||
Histone3 lysine4 trimethylation regulated by the facilitates chromatin transcription complex is critical for DNA cleavage in class switch recombination | Q34438355 | ||
Long-range directional movement of an interphase chromosome site | Q34516449 | ||
Mechanism and regulation of class switch recombination | Q34764888 | ||
The role of Alu repeat clusters as mediators of recurrent chromosomal aberrations in tumors | Q34804028 | ||
Chromosomal translocations involving the MLL gene: molecular mechanisms | Q35128590 | ||
Translocation-capture sequencing reveals the extent and nature of chromosomal rearrangements in B lymphocytes | Q35304991 | ||
Spatial genome organization in the formation of chromosomal translocations | Q35650635 | ||
The dangers of transcription. | Q52906715 | ||
Increased mobility of double-strand breaks requires Mec1, Rad9 and the homologous recombination machinery | Q53175321 | ||
The nuclear topography of ABL, BCR, PML, and RARalpha genes: evidence for gene proximity in specific phases of the cell cycle and stages of hematopoietic differentiation | Q57758627 | ||
Proximity ofTPRandNTRK1Rearranging Loci in Human Thyrocytes | Q58813385 | ||
Increased chromosome mobility facilitates homology search during recombination | Q63965533 | ||
Proximity of chromosomal loci that participate in radiation-induced rearrangements in human cells | Q64388367 | ||
Chromosomal translocation mechanisms at intronic alu elements in mammalian cells | Q64388774 | ||
Chromosomal breakpoint positions suggest a direct role for radiation in inducing illegitimate recombination between the ELE1 and RET genes in radiation-induced thyroid carcinomas | Q73270725 | ||
Cancer. Proximity matters | Q73450100 | ||
Spatial proximity of translocation-prone gene loci in human lymphomas | Q73531485 | ||
Localisation and distance between ABL and BCR genes in interphase nuclei of bone marrow cells of control donors and patients with chronic myeloid leukaemia | Q73814969 | ||
A brief survey of aberration origin theories | Q77208276 | ||
On the contribution of spatial genome organization to cancerous chromosome translocations | Q81686023 | ||
Potential G-quadruplex formation at breakpoint regions of chromosomal translocations in cancer may explain their fragility | Q84279386 | ||
Nonimmunoglobulin target loci of activation-induced cytidine deaminase (AID) share unique features with immunoglobulin genes | Q35786869 | ||
Spatial organization of the mouse genome and its role in recurrent chromosomal translocations | Q35874015 | ||
The constitutional t(11;22): implications for a novel mechanism responsible for gross chromosomal rearrangements. | Q35913621 | ||
Combinatorial complexity in chromatin structure and function: revisiting the histone code | Q35936019 | ||
DNA damage defines sites of recurrent chromosomal translocations in B lymphocytes | Q36282872 | ||
Mating type-dependent constraints on the mobility of the left arm of yeast chromosome III. | Q36322219 | ||
Human c-myc onc gene is located on the region of chromosome 8 that is translocated in Burkitt lymphoma cells | Q36327307 | ||
Translocation of the c-myc gene into the immunoglobulin heavy chain locus in human Burkitt lymphoma and murine plasmacytoma cells | Q36327408 | ||
Common fragile sites as targets for chromosome rearrangements | Q36522587 | ||
Topoisomerase II and the etiology of chromosomal translocations | Q36542253 | ||
Close proximity to Igh is a contributing factor to AID-mediated translocations. | Q36608103 | ||
Positional stability of single double-strand breaks in mammalian cells | Q36742792 | ||
Beyond the sequence: cellular organization of genome function | Q36744492 | ||
The impact of translocations and gene fusions on cancer causation | Q36760027 | ||
Role of AID in tumorigenesis | Q36845685 | ||
A plant homeodomain in RAG-2 that binds Hypermethylated lysine 4 of histone H3 is necessary for efficient antigen-receptor-gene rearrangement | Q37013832 | ||
DNA structure-induced genomic instability in vivo | Q37090816 | ||
Live cell microscopy analysis of radiation-induced DNA double-strand break motion. | Q37096613 | ||
Gene deregulation and spatial genome reorganization near breakpoints prior to formation of translocations in anaplastic large cell lymphoma | Q37153719 | ||
AID is required for the chromosomal breaks in c-myc that lead to c-myc/IgH translocations | Q37269390 | ||
The complex basis underlying common fragile site instability in cancer | Q37998801 | ||
Genome-wide translocation sequencing reveals mechanisms of chromosome breaks and rearrangements in B cells | Q38626707 | ||
V(D)J-mediated translocations in lymphoid neoplasms: a functional assessment of genomic instability by cryptic sites. | Q39017691 | ||
Signatures of mutation and selection in the cancer genome | Q39737975 | ||
Nuclear receptor-induced chromosomal proximity and DNA breaks underlie specific translocations in cancer | Q39766844 | ||
A topoisomerase IIbeta-mediated dsDNA break required for regulated transcription. | Q40263593 | ||
A non-B-DNA structure at the Bcl-2 major breakpoint region is cleaved by the RAG complex | Q40582495 | ||
Conservation of relative chromosome positioning in normal and cancer cells | Q40700082 | ||
In Situ Visualization of DNA Double-Strand Break Repair in Human Fibroblasts | Q41045622 | ||
Androgen-induced TOP2B-mediated double-strand breaks and prostate cancer gene rearrangements | Q41823310 | ||
53BP1 promotes non-homologous end joining of telomeres by increasing chromatin mobility | Q41919899 | ||
A model of oncogenic rearrangements: differences between chromosomal translocation mechanisms and simple double-strand break repair | Q42115543 | ||
Chromosomal instability mediated by non-B DNA: cruciform conformation and not DNA sequence is responsible for recurrent translocation in humans. | Q42150838 | ||
Human chromosomal translocations at CpG sites and a theoretical basis for their lineage and stage specificity | Q42260473 | ||
H3K4me3 stimulates the V(D)J RAG complex for both nicking and hairpinning in trans in addition to tethering in cis: implications for translocations | Q42536440 | ||
Activation-induced cytidine deaminase induces reproducible DNA breaks at many non-Ig Loci in activated B cells | Q42630118 | ||
Two levels of protection for the B cell genome during somatic hypermutation. | Q46755254 | ||
Influences of chromosome size, gene density and nuclear position on the frequency of constitutional translocations in the human population | Q47173079 | ||
RAG-dependent recombination at cryptic RSSs within TEL–AML1 t(12;21)(p13;q22) chromosomal translocation region | Q51553699 | ||
Dynamics of DNA double-strand breaks revealed by clustering of damaged chromosome domains | Q51830570 | ||
Chromatin motion is constrained by association with nuclear compartments in human cells | Q52545632 | ||
P433 | issue | 3 | |
P304 | page(s) | 357-364 | |
P577 | publication date | 2013-03-14 | |
P1433 | published in | Current Opinion in Cell Biology | Q13505682 |
P1476 | title | The cellular etiology of chromosome translocations | |
P478 | volume | 25 |
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Q55421355 | Comparative analysis of 2D and 3D distance measurements to study spatial genome organization. |
Q35879048 | Histone modifications predispose genome regions to breakage and translocation |
Q37259269 | Identification of kinase fusion oncogenes in post-Chernobyl radiation-induced thyroid cancers. |
Q58429078 | Mechanical Coupling of the Endothelial Cytoskeleton and Nucleus with VEGF Stimulation |
Q34040940 | Mechanisms and principles of homology search during recombination |
Q95941505 | Spatial inter-centromeric interactions facilitated the emergence of evolutionary new centromeres |
Q51017426 | Studies of the DNA Damage Response by Using the Lac Operator/Repressor (LacO/LacR) Tethering System. |
Q38201247 | The biogenesis of chromosome translocations. |
Q90242213 | The nucleoskeleton protein IFFO1 immobilizes broken DNA and suppresses chromosome translocation during tumorigenesis |
Q90291147 | The role of 3D genome organization in disease: From compartments to single nucleotides |
Q37076289 | Translocation and deletion breakpoints in cancer genomes are associated with potential non-B DNA-forming sequences |
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