scholarly article | Q13442814 |
P50 | author | Paul B Yu | Q42413083 |
Nicholas W Morrell | Q60960702 | ||
Panagis Filippakopoulos | Q28320317 | ||
Petra Knaus | Q28324737 | ||
P2093 | author name string | Hideyuki Beppu | |
Alex N Bullock | |||
Petra Knaus | |||
Yuji Mishina | |||
Donna Y Deng | |||
Ivan Alfano | |||
Ashley J Vonner | |||
Carol S C Lai | |||
Jan Börgermann | |||
Jana Bagarova | |||
Kelli A Armstrong | |||
P2860 | cites work | Constitutively active receptors as a disease-causing mechanism | Q40686419 |
Hyperactive BMP signaling induced by ALK2(R206H) requires type II receptor function in a Drosophila model for classic fibrodysplasia ossificans progressiva | Q41903017 | ||
The mode of bone morphogenetic protein (BMP) receptor oligomerization determines different BMP-2 signaling pathways. | Q42823613 | ||
High frequency of BMPR2 exonic deletions/duplications in familial pulmonary arterial hypertension | Q43173796 | ||
Bone morphogenetic protein (BMP) type II receptor deletion reveals BMP ligand-specific gain of signaling in pulmonary artery smooth muscle cells | Q46481363 | ||
Bone morphogenetic protein (BMP) type II receptor is required for BMP-mediated growth arrest and differentiation in pulmonary artery smooth muscle cells | Q46879441 | ||
Generation of a mouse with conditionally activated signaling through the BMP receptor, ALK2. | Q50734070 | ||
Generation of a floxed allele of the mouse BMP type II receptor gene. | Q51535561 | ||
Stoichiometric imbalance in the receptor complex contributes to dysfunctional BMPR-II mediated signalling in pulmonary arterial hypertension | Q57148975 | ||
Identification of type I receptors for osteogenic protein-1 and bone morphogenetic protein-4 | Q72000906 | ||
Functional modeling of the ACVR1 (R206H) mutation in FOP | Q80486365 | ||
Functional analysis of bone morphogenetic protein type II receptor mutations underlying primary pulmonary hypertension | Q24298704 | ||
A recurrent mutation in the BMP type I receptor ACVR1 causes inherited and sporadic fibrodysplasia ossificans progressiva | Q24321505 | ||
Bone morphogenetic protein receptor complexes on the surface of live cells: a new oligomerization mode for serine/threonine kinase receptors | Q24548576 | ||
BMP type I receptor inhibition reduces heterotopic [corrected] ossification | Q24611307 | ||
Structure of the bone morphogenetic protein receptor ALK2 and implications for fibrodysplasia ossificans progressiva | Q24623325 | ||
Size-Distribution Analysis of Macromolecules by Sedimentation Velocity Ultracentrifugation and Lamm Equation Modeling | Q27860847 | ||
Specific activation of Smad1 signaling pathways by the BMP7 type I receptor, ALK2 | Q28283345 | ||
BMP signaling is required for septation of the outflow tract of the mammalian heart | Q28592358 | ||
The type II activin receptors are essential for egg cylinder growth, gastrulation, and rostral head development in mice | Q28592388 | ||
Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts | Q29547605 | ||
Classic and atypical fibrodysplasia ossificans progressiva (FOP) phenotypes are caused by mutations in the bone morphogenetic protein (BMP) type I receptor ACVR1 | Q30373949 | ||
Kinase mutations in cancer: chinks in the enemy's armour? | Q34477061 | ||
Somatic mutations of the EGF receptor and their signal transducers affect the efficacy of EGF receptor-specific tyrosine kinase inhibitors | Q34987162 | ||
Fibrodysplasia ossificans progressiva | Q37105402 | ||
Constitutively activated ALK2 and increased SMAD1/5 cooperatively induce bone morphogenetic protein signaling in fibrodysplasia ossificans progressiva. | Q37119764 | ||
Constitutive activation of G protein-coupled receptors and diseases: insights into mechanisms of activation and therapeutics | Q37157903 | ||
The fibrodysplasia ossificans progressiva R206H ACVR1 mutation activates BMP-independent chondrogenesis and zebrafish embryo ventralization | Q37403100 | ||
P433 | issue | 12 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | cooperation | Q380962 |
P304 | page(s) | 2413-2424 | |
P577 | publication date | 2013-04-09 | |
P1433 | published in | Molecular and Cellular Biology | Q3319478 |
P1476 | title | Constitutively active ALK2 receptor mutants require type II receptor cooperation | |
P478 | volume | 33 |
Q54248766 | ACVR1R206H receptor mutation causes fibrodysplasia ossificans progressiva by imparting responsiveness to activin A. |
Q28066886 | Activin receptor-like kinases: a diverse family playing an important role in cancer |
Q35035999 | Adult-specific systemic over-expression reveals novel in vivo effects of the soluble forms of ActRIIA, ActRIIB and BMPRII. |
Q58543870 | An Adult Zebrafish Model of Fibrodysplasia Ossificans Progressiva |
Q48302210 | Animal models of fibrodysplasia ossificans progressiva. |
Q39439904 | Application of human induced pluripotent stem cells to model fibrodysplasia ossificans progressiva |
Q35625198 | Apyrase as a novel therapeutic inhibitor of heterotopic ossification |
Q34293026 | BMP type II receptors have redundant roles in the regulation of hepatic hepcidin gene expression and iron metabolism |
Q91879175 | BMPR2 acts as a gatekeeper to protect endothelial cells from increased TGFβ responses and altered cell mechanics |
Q46612046 | BmpR1A is a major type 1 BMP receptor for BMP-Smad signaling during skull development |
Q34512444 | Bone Morphogenetic Protein (BMP) signaling in development and human diseases |
Q36578816 | Common mutations in ALK2/ACVR1, a multi-faceted receptor, have roles in distinct pediatric musculoskeletal and neural orphan disorders |
Q39271127 | Conserved signaling pathways underlying heterotopic ossification |
Q36981608 | Granting immunity to FOP and catching heterotopic ossification in the Act. |
Q57161028 | Injury of Adult Zebrafish Expressing Acvr1l Does Not Result in Heterotopic Ossification |
Q64058749 | Insights into the biology of fibrodysplasia ossificans progressiva using patient-derived induced pluripotent stem cells |
Q35212915 | Molecular and cellular mechanisms of heterotopic ossification |
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Q36394399 | Neofunction of ACVR1 in fibrodysplasia ossificans progressiva |
Q41215881 | New Protocol to Optimize iPS Cells for Genome Analysis of Fibrodysplasia Ossificans Progressiva. |
Q58543834 | Pharmacologic Strategies for Assaying BMP Signaling Function |
Q58543855 | Phenotypic Analyses of Genetically Modified Mice for BMP Receptors |
Q38666840 | Recent advances in understanding contextual TGFβ signaling |
Q47378323 | Regulation of continuous but complex expression pattern of Six1 during early sensory development. |
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Q50152313 | Stem cells and heterotopic ossification: Lessons from animal models |
Q34313807 | Structure-activity relationship of 3,5-diaryl-2-aminopyridine ALK2 inhibitors reveals unaltered binding affinity for fibrodysplasia ossificans progressiva causing mutants |
Q26798172 | TGF-β/BMP signaling and other molecular events: regulation of osteoblastogenesis and bone formation |
Q33751426 | The genomic landscape of diffuse intrinsic pontine glioma and pediatric non-brainstem high-grade glioma |
Q38669186 | The obligatory role of Activin A in the formation of heterotopic bone in Fibrodysplasia Ossificans Progressiva. |
Q52343928 | The orphan GPR50 receptor promotes constitutive TGFβ receptor signaling and protects against cancer development. |
Q92152712 | The role of Activin A in fibrodysplasia ossificans progressiva: a prominent mediator |
Q40459563 | Two tissue-resident progenitor lineages drive distinct phenotypes of heterotopic ossification. |
Q36561184 | Unique genetic and epigenetic mechanisms driving paediatric diffuse high-grade glioma |
Q47447929 | Variable signaling activity by FOP ACVR1 mutations |
Q50144496 | Variant BMP receptor mutations causing fibrodysplasia ossificans progressiva (FOP) in humans show BMP ligand-independent receptor activation in zebrafish |
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