| scholarly article | Q13442814 |
| P2093 | author name string | J A Metcalf | |
| R K Root | |||
| R Isturiz | |||
| P2860 | cites work | Myeloperoxidase, hydrogen peroxide, chloride antimicrobial system: nitrogen-chlorine derivatives of bacterial components in bactericidal action against Escherichia coli | Q24564090 |
| Potentiation of opsonization and phagocytosis of Streptococcus pyogenes following growth in the presence of clindamycin | Q24601968 | ||
| Abnormal bactericidal, metabolic, and lysosomal functions of Chediak-Higashi Syndrome leukocytes | Q28363269 | ||
| Catalase, superoxide dismutase, and virulence of Staphylococcus aureus. In vitro and in vivo studies with emphasis on staphylococcal--leukocyte interaction | Q30449755 | ||
| Mutants of Diplococcus pneumoniae that lack deoxyribonucleases and other activities possibly pertinent to genetic transformation | Q33768056 | ||
| Oxidation of Escherichia coli iron centers by the myeloperoxidase-mediated microbicidal system | Q34253860 | ||
| Complement and immunoglobulins stimulate superoxide production by human leukocytes independently of phagocytosis | Q34484070 | ||
| Generation of nitrogen-chlorine oxidants by human phagocytes | Q34617174 | ||
| Mechanism of action of penicillin: triggering of the pneumococcal autolytic enzyme by inhibitors of cell wall synthesis | Q35092585 | ||
| Hypersusceptibility of penicillin-treated group B streptococci to bactericidal activity of human polymorphonuclear leukocytes | Q35669554 | ||
| Tolerant response of Streptococcus sanguis to beta-lactams and other cell wall inhibitors | Q36463053 | ||
| Secretion of Cell Wall Polymers into the Growth Medium of Lysis-Defective Pneumococci During Treatment with Penicillin and Other Inhibitors of Cell Wall Synthesis | Q36466799 | ||
| Characterization of Cell Wall Polymers Secreted into the Growth Medium of Lysis-Defective Pneumococci During Treatment with Penicillin and Other Inhibitors of Cell Wall Synthesis | Q36466841 | ||
| Cytochalasin B reversibly inhibits phagocytosis: functional, metabolic, and ultrastructural effects in human blood leukocytes and rabbit alveolar macrophages | Q36991610 | ||
| H2O2 release from human granulocytes during phagocytosis. Relationship to superoxide anion formation and cellular catabolism of H2O2: studies with normal and cytochalasin B-treated cells | Q37048880 | ||
| From penicillin-binding proteins to the lysis and death of bacteria: a 1979 view | Q39307860 | ||
| The microbicidal mechanisms of human neutrophils and eosinophils | Q40315026 | ||
| Interactions between antibiotics and human neutrophils in the killing of staphylococci. | Q40687700 | ||
| Effect of sublethal concentrations of penicillins on the lysis of bacteria by lysozyme and trypsin | Q43489572 | ||
| Role of myeloperoxidase-mediated antimicrobial systems in intact leukocytes | Q44323452 | ||
| A study of the genetic material determining an enzyme activity in Pneumococcus | Q44711463 | ||
| Fungal infection in chronic granulomatous disease | Q57908583 | ||
| Role of myeloperoxidase in the respiratory burst of human neutrophils | Q67264676 | ||
| Effects of cytochalasin B on polymorphonuclear leucocyte locomotion, phagocytosis and glycolysis | Q70439654 | ||
| Myeloperoxidase deficiency: prevalence and clinical significance | Q72904937 | ||
| P433 | issue | 2 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | gram-positive bacteria | Q857288 |
| P304 | page(s) | 133-143 | |
| P577 | publication date | 1985-03-01 | |
| P1433 | published in | Yale Journal of Biology and Medicine | Q15709982 |
| P1476 | title | Enhanced killing of penicillin-treated gram-positive cocci by human granulocytes: role of bacterial autolysins, catalase, and granulocyte oxidative pathways | |
| P478 | volume | 58 |
| Q39881971 | A new method to determine postantibiotic effect and effects of subinhibitory antibiotic concentrations |
| Q39484244 | Cationic polyelectrolytes: a new look at their possible roles as opsonins, as stimulators of respiratory burst in leukocytes, in bacteriolysis, and as modulators of immune-complex diseases (a review hypothesis). |
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