| P2093 | author name string | E Rhéaume | |
| | Y Shi | |
| | M Lebel | |
| | J-C Tardif | |
| | D Busseuil | |
| | M Mecteau | |
| | R Mongrain | |
| | D Shustik | |
| | I El-Hamamsy | |
| | J Trapeaux | |
| | S Nobari | |
| P2860 | cites work | Effect of recombinant ApoA-I Milano on coronary atherosclerosis in patients with acute coronary syndromes: a randomized controlled trial | Q28181984 |
| | A deletion within the murine Werner syndrome helicase induces sensitivity to inhibitors of topoisomerase and loss of cellular proliferative capacity | Q28589633 |
| | Calcific aortic valve stenosis: methods, models, and mechanisms | Q30503246 |
| | Extracellular matrix remodelling in human aortic valve disease: the role of matrix metalloproteinases and their tissue inhibitors | Q81644115 |
| | Aortic stenosis | Q83386744 |
| | Aortic stenosis: diagnosis and treatment | Q33156648 |
| | Hemodynamics and axial strain additively increase matrix remodeling and MMP-9, but not MMP-2, expression in arteries engineered by directed remodeling | Q33606446 |
| | Arterial and aortic valve calcification inversely correlates with osteoporotic bone remodelling: a role for inflammation | Q34065287 |
| | Divergent effects of matrix metalloproteinases 3, 7, 9, and 12 on atherosclerotic plaque stability in mouse brachiocephalic arteries | Q34098485 |
| | Animal research: reporting in vivo experiments: the ARRIVE guidelines | Q34116755 |
| | Evidence for Active Regulation of Pro-Osteogenic Signaling in Advanced Aortic Valve Disease | Q34378569 |
| | Effect of very high-intensity statin therapy on regression of coronary atherosclerosis: the ASTEROID trial | Q34501729 |
| | Mechanisms of cardiovascular disease in accelerated aging syndromes | Q34646841 |
| | Calcific aortic valve disease: not simply a degenerative process: A review and agenda for research from the National Heart and Lung and Blood Institute Aortic Stenosis Working Group. Executive summary: Calcific aortic valve disease-2011 update. | Q35836894 |
| | Pharmacologic augmentation of high-density lipoproteins: mechanisms of currently available and emerging therapies | Q36163410 |
| | Spectrum of calcific aortic valve disease: pathogenesis, disease progression, and treatment strategies | Q36169078 |
| | Aortic valve stenosis: an active atheroinflammatory process | Q36947972 |
| | Models of aortic valve calcification | Q36982749 |
| | Calcific aortic stenosis: lessons learned from experimental and clinical studies | Q37331207 |
| | High-density lipoproteins, inflammation and oxidative stress | Q37348323 |
| | Medical therapy for calcific aortic stenosis | Q37914518 |
| | Effects of reconstituted high-density lipoprotein infusions on coronary atherosclerosis: a randomized controlled trial | Q38398332 |
| | Guidelines for reporting experiments involving animals: the ARRIVE guidelines | Q39512979 |
| | Regression of aortic valve stenosis by ApoA-I mimetic peptide infusions in rabbits. | Q39573172 |
| | Multimodality molecular imaging identifies proteolytic and osteogenic activities in early aortic valve disease. | Q40251686 |
| | Arterial and aortic valve calcification abolished by elastolytic cathepsin S deficiency in chronic renal disease. | Q41999917 |
| | Therapeutic vascular compliance change may cause significant variation in coronary perfusion: a numerical study. | Q42582342 |
| | Recombinant apolipoprotein A-I Milano rapidly reverses aortic valve stenosis and decreases leaflet inflammation in an experimental rabbit model. | Q43122856 |
| | Experimental aortic valve stenosis in rabbits | Q44392664 |
| | Increased insulin, triglycerides, reactive oxygen species, and cardiac fibrosis in mice with a mutation in the helicase domain of the Werner syndrome gene homologue | Q46837223 |
| | A high fat/high carbohydrate diet induces aortic valve disease in C57BL/6J mice | Q46951764 |
| | Age-associated aortic stenosis in apolipoprotein E-deficient mice. | Q50762984 |
| | Further insights into normal aortic valve function: role of a compliant aortic root on leaflet opening and valve orifice area. | Q51707285 |
| | Increased matrix metalloproteinase-9 activity in unstable carotid plaques. A potential role in acute plaque disruption. | Q52537589 |
| | Targeted disruption of the matrix metalloproteinase-9 gene impairs smooth muscle cell migration and geometrical arterial remodeling. | Q52548108 |
| | Role of lung apolipoprotein A-I in idiopathic pulmonary fibrosis: antiinflammatory and antifibrotic effect on experimental lung injury and fibrosis. | Q53470982 |
| | High-density lipoproteins (HDL) are present in stenotic aortic valves and may interfere with the mechanisms of valvular calcification. | Q54348949 |
| | Effects of Rosuvastatin on Progression of Stenosis in Adult Patients With Congenital Aortic Stenosis (PROCAS Trial) | Q57916583 |
| | Extracellular Matrix Remodeling and Organization in Developing and Diseased Aortic Valves | Q63341951 |
| | Dilated cardiomyopathy is associated with an increase in the type I/type III collagen ratio: a quantitative assessment | Q72168553 |
| | Severe heart valve calcification in a young patient with Werner syndrome | Q73598994 |
| | [Calcified aortic valvular disease associated with adult progeria] | Q74547908 |
| | Dilated cardiomyopathy is associated with significant changes in collagen type I/III ratio | Q77822656 |
| | Cardiovascular manifestations in progeria. Report of clinical and pathologic findings in a patient with severe arteriosclerotic heart disease and aortic stenosis | Q78998940 |
| | Morphological changes of the aortic valve leaflets in non-compliant aortic roots: in-vivo experiments | Q80253499 |
| | Reduction of coronary flow reserve in patients with increased aortic stiffness | Q81365809 |
| P433 | issue | 7 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | aorta | Q101004 |
| | aortic valve stenosis | Q531019 |
| P304 | page(s) | 1587-1599 | |
| P577 | publication date | 2013-08-01 | |
| P1433 | published in | British Journal of Pharmacology | Q919631 |
| P1476 | title | Improvement of aortic valve stenosis by ApoA-I mimetic therapy is associated with decreased aortic root and valve remodelling in mice | |
| P478 | volume | 169 | |