scholarly article | Q13442814 |
P2093 | author name string | Li Zhao | |
Dong Wook Kim | |||
Sheue-Yann Cheng | |||
Mark C Willingham | |||
Xuguang Zhu | |||
P2860 | cites work | Phase 2 trial of oral vorinostat (suberoylanilide hydroxamic acid, SAHA) for refractory cutaneous T-cell lymphoma (CTCL) | Q24681480 |
Convergent loss of PTEN leads to clinical resistance to a PI(3)Kα inhibitor | Q27853093 | ||
PTEN, a putative protein tyrosine phosphatase gene mutated in human brain, breast, and prostate cancer | Q27860985 | ||
Histone acetylation in chromatin structure and transcription | Q28131749 | ||
Lost in transcription: p21 repression, mechanisms, and consequences | Q28251319 | ||
Dimethyl sulfoxide to vorinostat: development of this histone deacetylase inhibitor as an anticancer drug | Q28282479 | ||
The landscape of somatic copy-number alteration across human cancers | Q29547648 | ||
Phase IIb multicenter trial of vorinostat in patients with persistent, progressive, or treatment refractory cutaneous T-cell lymphoma | Q33375600 | ||
FDA approval summary: vorinostat for treatment of advanced primary cutaneous T-cell lymphoma | Q33377159 | ||
Lack of therapeutic effect of the histone deacetylase inhibitor vorinostat in patients with metastatic radioiodine-refractory thyroid carcinoma | Q33381498 | ||
Romidepsin: a new therapy for cutaneous T-cell lymphoma and a potential therapy for solid tumors. | Q33390876 | ||
Phase I study of vorinostat in patients with advanced solid tumors and hepatic dysfunction: a National Cancer Institute Organ Dysfunction Working Group study | Q33391851 | ||
Induction of thyroid gene expression and radioiodine uptake in thyroid cancer cells by targeting major signaling pathways | Q33736109 | ||
Src-dependent ezrin phosphorylation in adhesion-mediated signaling | Q33914506 | ||
Activation of phosphatidylinositol 3-kinase signaling by a mutant thyroid hormone beta receptor | Q34478795 | ||
Synergistic signaling of KRAS and thyroid hormone receptor β mutants promotes undifferentiated thyroid cancer through MYC up-regulation | Q34528284 | ||
Histone deacetylases and cancer: causes and therapies | Q34570082 | ||
Aberrant accumulation of PTTG1 induced by a mutated thyroid hormone beta receptor inhibits mitotic progression | Q35070405 | ||
TSH-activated signaling pathways in thyroid tumorigenesis | Q35736825 | ||
PTEN deficiency accelerates tumour progression in a mouse model of thyroid cancer | Q36278234 | ||
Role of TSH in the spontaneous development of asymmetrical thyroid carcinoma in mice with a targeted mutation in a single allele of the thyroid hormone-β receptor | Q36439404 | ||
Robust Thyroid Gene Expression and Radioiodine Uptake Induced by Simultaneous Suppression of BRAF V600E and Histone Deacetylase in Thyroid Cancer Cells | Q36715993 | ||
Regulation of beta-catenin by a novel nongenomic action of thyroid hormone beta receptor | Q36748109 | ||
Evaluation of Romidepsin for Clinical Activity and Radioactive Iodine Reuptake in Radioactive Iodine–Refractory Thyroid Carcinoma | Q36813563 | ||
Histone deacetylase inhibitors: molecular mechanisms of action | Q36908440 | ||
Mice with a targeted mutation in the thyroid hormone beta receptor gene exhibit impaired growth and resistance to thyroid hormone | Q37288003 | ||
Phase I Trial of a New Schedule of Romidepsin in Patients with Advanced Cancers | Q37667088 | ||
Genetic alterations in the phosphatidylinositol-3 kinase/Akt pathway in thyroid cancer | Q37767709 | ||
Radioiodine for remnant ablation and therapy of metastatic disease | Q37913996 | ||
HDAC inhibitors in cancer biology: emerging mechanisms and clinical applications | Q37962333 | ||
Mechanisms of Resistance to Histone Deacetylase Inhibitors | Q38054220 | ||
p21WAF1 and tumourigenesis: 20 years after | Q38060451 | ||
Ezrin is key regulator of Src-induced malignant phenotype in three-dimensional environment | Q39525953 | ||
Activity of deacetylase inhibitor panobinostat (LBH589) in cutaneous T-cell lymphoma models: Defining molecular mechanisms of resistance | Q39744464 | ||
Histone deacetylase inhibitors induce thyroid cancer-specific apoptosis through proteasome-dependent inhibition of TRAIL degradation. | Q39791600 | ||
Constitutive activation of signal transducers and activators of transcription predicts vorinostat resistance in cutaneous T-cell lymphoma | Q39981886 | ||
Akt- and MAPK-mediated activation and secretion of MMP-9 into stroma in breast cancer cells upon heregulin treatment. | Q40029865 | ||
The histone deacetylase inhibitors suberoylanilide hydroxamic (Vorinostat) and valproic acid induce irreversible and MDR1-independent resistance in human colon cancer cells | Q40098928 | ||
Antitumor activity of suberoylanilide hydroxamic acid against thyroid cancer cell lines in vitro and in vivo | Q40227787 | ||
Novel histone deacetylase inhibitors in the treatment of thyroid cancer | Q40422202 | ||
Crosstalk between the Rb pathway and AKT signaling forms a quiescence-senescence switch | Q46909608 | ||
Mice with a mutation in the thyroid hormone receptor beta gene spontaneously develop thyroid carcinoma: a mouse model of thyroid carcinogenesis | Q52548664 | ||
Inhibition of phosphatidylinositol 3-kinase delays tumor progression and blocks metastatic spread in a mouse model of thyroid cancer | Q53330585 | ||
P433 | issue | 7 | |
P921 | main subject | carcinogenesis | Q1637543 |
P304 | page(s) | 521-533 | |
P577 | publication date | 2016-06-07 | |
P1433 | published in | Endocrine-Related Cancer | Q3054004 |
P1476 | title | SAHA-induced loss of tumor suppressor Pten gene promotes thyroid carcinogenesis in a mouse model | |
P478 | volume | 23 |
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