| scholarly article | Q13442814 |
| P356 | DOI | 10.1172/JCI8561 |
| P8608 | Fatcat ID | release_fnxwo63pwnbihduceazy3vt33a |
| P932 | PMC publication ID | 377433 |
| P698 | PubMed publication ID | 10642601 |
| P2093 | author name string | S D Miller | |
| L A Matis | |||
| M C Dal Canto | |||
| G C Suvannavejh | |||
| P2860 | cites work | Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis | Q30080034 |
| Lpr and gld: single gene models of systemic autoimmunity and lymphoproliferative disease | Q34602392 | ||
| Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide | Q36297378 | ||
| The involvement of T cell receptor peptide-specific regulatory CD4+ T cells in recovery from antigen-induced autoimmune disease | Q36362274 | ||
| Cytokine-induced immune deviation as a therapy for inflammatory autoimmune disease | Q36364080 | ||
| Functional evidence for epitope spreading in the relapsing pathology of experimental autoimmune encephalomyelitis | Q36365105 | ||
| A predictable sequential determinant spreading cascade invariably accompanies progression of experimental autoimmune encephalomyelitis: a basis for peptide-specific therapy after onset of clinical disease | Q36366545 | ||
| Inactivation of T cell receptor peptide-specific CD4 regulatory T cells induces chronic experimental autoimmune encephalomyelitis (EAE). | Q36367554 | ||
| Dual role for Fas ligand in the initiation of and recovery from experimental allergic encephalomyelitis | Q36368058 | ||
| Local delivery of interleukin 4 by retrovirus-transduced T lymphocytes ameliorates experimental autoimmune encephalomyelitis. | Q36377181 | ||
| Multiple sclerosis: Fas signaling in oligodendrocyte cell death | Q36377383 | ||
| Myelin basic protein-specific T helper 2 (Th2) cells cause experimental autoimmune encephalomyelitis in immunodeficient hosts rather than protect them from the disease | Q36380402 | ||
| Requirement of Fas for the development of autoimmune diabetes in nonobese diabetic mice | Q36380580 | ||
| Treatment of experimental encephalomyelitis with a novel chimeric fusion protein of myelin basic protein and proteolipid protein | Q37360410 | ||
| Loci predisposing to autoimmunity in MRL-Fas lpr and C57BL/6-Faslpr mice | Q37378097 | ||
| Evidence for Fas-dependent and Fas-independent mechanisms in the pathogenesis of experimental autoimmune encephalomyelitis | Q40951076 | ||
| IL-10 is critical in the regulation of autoimmune encephalomyelitis as demonstrated by studies of IL-10- and IL-4-deficient and transgenic mice. | Q41005196 | ||
| Apoptosis mediated by the TNF-related cytokine and receptor families | Q41126177 | ||
| Interleukin-2 programs mouse alpha beta T lymphocytes for apoptosis | Q43539775 | ||
| Vaccination against experimental autoimmune encephalomyelitis using a subencephalitogenic dose of autoimmune effector T cells. (2). Induction of a protective anti-idiotypic response | Q44539084 | ||
| Immunization with a synthetic T-cell receptor V-region peptide protects against experimental autoimmune encephalomyelitis | Q45351794 | ||
| Astroglial overproduction of TGF-beta 1 enhances inflammatory central nervous system disease in transgenic mice . | Q46004205 | ||
| Apoptotic elimination of V beta 8.2+ cells from the central nervous system during recovery from experimental autoimmune encephalomyelitis induced by the passive transfer of V beta 8.2+ encephalitogenic T cells | Q46225637 | ||
| Microglia are more susceptible than macrophages to apoptosis in the central nervous system in experimental autoimmune encephalomyelitis through a mechanism not involving Fas (CD95). | Q46386507 | ||
| Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas. | Q46925453 | ||
| Mechanisms of recovery from experimental autoimmune encephalomyelitis: T cell deletion and immune deviation in myelin basic protein T cell receptor transgenic mice | Q48467858 | ||
| Fas expression on human fetal astrocytes without susceptibility to fas-mediated cytotoxicity | Q48491692 | ||
| Myelin oligodendrocyte glycoprotein-induced autoimmune encephalomyelitis is chronic/relapsing in perforin knockout mice, but monophasic in Fas- and Fas ligand-deficient lpr and gld mice | Q48535553 | ||
| Adoptively transferred chronic relapsing experimental autoimmune encephalomyelitis in the mouse. Neuropathologic analysis | Q48609064 | ||
| Successful treatment of experimental allergic encephalomyelitis with transforming growth factor-beta 1 | Q48655440 | ||
| Cell death during autoimmune demyelination: effector but not target cells are eliminated by apoptosis. | Q51995260 | ||
| Interleukin-10 prevents experimental allergic encephalomyelitis in rats. | Q54205210 | ||
| Suppression of experimentally induced autoimmune encephalomyelitis by cytolytic T-T cell interactions. | Q54385154 | ||
| Fas- and FasL-deficient mice are resistant to induction of autoimmune encephalomyelitis. | Q64999806 | ||
| Induction of active and adoptive relapsing experimental autoimmune encephalomyelitis (EAE) using an encephalitogenic epitope of proteolipid protein | Q67537170 | ||
| Evidence of endogenous regulatory function of transforming growth factor-beta 1 in experimental allergic encephalomyelitis | Q67539413 | ||
| Apoptosis of alpha beta T lymphocytes in the nervous system in experimental autoimmune encephalomyelitis: its possible implications for recovery and acquired tolerance | Q67544544 | ||
| Inhibition of murine relapsing experimental autoimmune encephalomyelitis by immune tolerance to proteolipid protein and its encephalitogenic peptides | Q67653169 | ||
| Prevention and treatment of chronic relapsing experimental allergic encephalomyelitis by transforming growth factor-beta 1 | Q67687123 | ||
| Analysis of cytokine mRNA expression in the central nervous system of mice with experimental autoimmune encephalomyelitis reveals that IL-10 mRNA expression correlates with recovery | Q68047768 | ||
| CD4+ suppressor cells inhibit the function of effector cells of experimental autoimmune encephalomyelitis through a mechanism involving transforming growth factor-beta | Q68291930 | ||
| Chronologic neuropathology of relapsing experimental allergic encephalomyelitis in the mouse | Q70293154 | ||
| In vivo administration of IL-4 induces TGF-beta-producing cells and protects animals from experimental autoimmune encephalomyelitis | Q71026292 | ||
| The roles of costimulation and Fas in T cell apoptosis and peripheral tolerance | Q71066518 | ||
| Transforming growth factor-beta 1 inhibits tumor necrosis factor-alpha/lymphotoxin production and adoptive transfer of disease by effector cells of autoimmune encephalomyelitis | Q71618908 | ||
| The fas antigen is involved in peripheral but not thymic deletion of T lymphocytes in T cell receptor transgenic mice | Q71678868 | ||
| IL-10 fails to abrogate experimental autoimmune encephalomyelitis | Q71726961 | ||
| Cytokine production in the central nervous system of Lewis rats with experimental autoimmune encephalomyelitis: dynamics of mRNA expression for interleukin-10, interleukin-12, cytolysin, tumor necrosis factor α and tumor necrosis factor β | Q71825997 | ||
| Neuroantigen-specific Th2 cells are inefficient suppressors of experimental autoimmune encephalomyelitis induced by effector Th1 cells | Q71829117 | ||
| Characteristic T helper 2 T cell cytokine abnormalities in autoimmune lymphoproliferative syndrome, a syndrome marked by defective apoptosis and humoral autoimmunity | Q73064451 | ||
| Competitive PCR quantification of pro- and anti-inflammatory cytokine mRNA in the central nervous system during autoimmune encephalomyelitis | Q73128591 | ||
| The role of Fas in autoimmune diabetes | Q73205341 | ||
| Fas and Fas ligand enhance the pathogenesis of experimental allergic encephalomyelitis, but are not essential for immune privilege in the central nervous system | Q73747870 | ||
| Adoptively transferred EAE in mice bearing the lpr mutation | Q73944984 | ||
| The roles of Fas, Fas ligand and Bcl-2 in T cell apoptosis in the central nervous system in experimental autoimmune encephalomyelitis | Q74396844 | ||
| Presentation of proteolipid protein epitopes and B7-1-dependent activation of encephalitogenic T cells by IFN-gamma-activated SJL/J astrocytes | Q74515347 | ||
| Differential expression of inflammatory cytokines parallels progression of central nervous system pathology in two clinically distinct models of multiple sclerosis | Q77437086 | ||
| Beta-cell destruction in NOD mice correlates with Fas (CD95) expression on beta-cells and proinflammatory cytokine expression in islets | Q77804879 | ||
| Cytokines as intrinsic and exogenous regulators of pathogenesis in experimental autoimmune encephalomyelitis | Q77922196 | ||
| P433 | issue | 2 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | apoptotic process | Q14599311 |
| experimental autoimmune encephalomyelitis | Q494066 | ||
| P304 | page(s) | 223-231 | |
| P577 | publication date | 2000-01-01 | |
| P1433 | published in | Journal of Clinical Investigation | Q3186904 |
| P1476 | title | Fas-mediated apoptosis in clinical remissions of relapsing experimental autoimmune encephalomyelitis | |
| P478 | volume | 105 |
| Q35868821 | A public T cell clonotype within a heterogeneous autoreactive repertoire is dominant in driving EAE. |
| Q37267473 | A targeted DNA vaccine encoding fas ligand defines its dual role in the regulation of experimental autoimmune encephalomyelitis |
| Q33882444 | Antigen-specific CD25- Foxp3- IFN-gamma(high) CD4+ T cells restrain the development of experimental allergic encephalomyelitis by suppressing Th17. |
| Q34398617 | Apoptosis of inflammatory cells in immune control of the nervous system: role of glia |
| Q36922991 | Apoptotic signal transduction and T cell tolerance |
| Q48453594 | Astrocyte-induced T cell elimination is CD95 ligand dependent. |
| Q34768674 | Autoreactive CD8 T cells in organ-specific autoimmunity: emerging targets for therapeutic intervention. |
| Q34398602 | Basic principles of immunological surveillance of the normal central nervous system |
| Q79291232 | Beneficial autoimmunity to proinflammatory mediators restrains the consequences of self-destructive immunity |
| Q37061386 | CD4 T cells: Balancing the coming and going of autoimmune-mediated inflammation in the CNS. |
| Q50937116 | CRYAB modulates the activation of CD4+ T cells from relapsing-remitting multiple sclerosis patients. |
| Q56905834 | CTLA-4 downregulates epitope spreading and mediates remission in relapsing experimental autoimmune encephalomyelitis |
| Q34993208 | Cell death and immune privilege |
| Q52042442 | Clearance of apoptotic beta-cells is reduced in neonatal autoimmune diabetes-prone rats. |
| Q33859990 | Comparison of sensitivity of Th1, Th2, and Th17 cells to Fas-mediated apoptosis |
| Q33654218 | Constitutive expression of murine c-FLIPR causes autoimmunity in aged mice |
| Q38995923 | Control of the Survival and Growth of Human Glioblastoma Grafted Into the Spinal Cord of Mice by Taking Advantage of Immunorejection |
| Q36755403 | Cultured murine thyroid epithelial cells expressing transgenic Fas-associated death domain-like interleukin-1beta converting enzyme inhibitory protein are protected from fas-mediated apoptosis |
| Q36535669 | Death ligands and autoimmune demyelination |
| Q36198607 | Dendritic cell-based immunotherapy for malignant gliomas |
| Q52593498 | Disease activity in multiple sclerosis correlates with T lymphocyte expression of the inhibitor of apoptosis proteins. |
| Q36529963 | Distinct different sensitivity of Treg and Th17 cells to Fas-mediated apoptosis signaling in patients with acute coronary syndrome. |
| Q46036987 | Dual role of Response gene to complement-32 in multiple sclerosis. |
| Q35168021 | Effect of transgenic overexpression of FLIP on lymphocytes on development and resolution of experimental autoimmune thyroiditis |
| Q34418038 | Effector lymphocytes in autoimmunity |
| Q43436517 | Effects of peptide fraction and counter ion on the development of clinical signs in experimental autoimmune encephalomyelitis |
| Q79073479 | FLIP and FasL expression by inflammatory cells vs thyrocytes can be predictive of chronic inflammation or resolution of autoimmune thyroiditis |
| Q34230777 | Fas (CD95, Apo-1) ligand gene transfer |
| Q63408388 | Fas-Ligand and Immune Privilege: The Eyes Have It |
| Q34571353 | Functional epistasis on a common MHC haplotype associated with multiple sclerosis |
| Q45993800 | Functional polymorphisms in cell death pathway genes FAS and FAS ligand and risk of alopecia areata. |
| Q73056498 | Genetic control of pathogenic mechanisms in autoimmune demyelinating disease |
| Q36898699 | Glioma stem cells and immunotherapy for the treatment of malignant gliomas |
| Q36126949 | Hippocampal protection in mice with an attenuated inflammatory monocyte response to acute CNS picornavirus infection |
| Q43213851 | Incomplete Killing And Enhanced Activation of Islet-Reactive CD8+ T Cells by FasL-Expressing Dendritic Cells Limits Protection from Diabetes |
| Q73153762 | Increased cellular expression of the caspase inhibitor FLIP in intrathecal lymphocytes from patients with multiple sclerosis |
| Q33806619 | MK2 and Fas receptor contribute to the severity of CNS demyelination |
| Q35781415 | Murine FLIP transgene expressed on thyroid epithelial cells promotes resolution of granulomatous experimental autoimmune thyroiditis in DBA/1 mice. |
| Q54022484 | Overexpression of the apoptosis inhibitor FLIP in T cells correlates with disease activity in multiple sclerosis. |
| Q48628780 | Peripheral LPS administrations up-regulate Fas and FasL on brain microglial cells: a brain protective or pathogenic event? |
| Q78952331 | Regulatory role of p53 in experimental autoimmune encephalomyelitis |
| Q47873731 | Resistance to activation-induced cell death and elevated FLIPL expression of CD4+ T cells in a polyI:C-induced primary biliary cirrhosis mouse model |
| Q26800155 | Role of Tumor Necrosis Factor Superfamily in Neuroinflammation and Autoimmunity |
| Q34432395 | Role of passive T-cell death in chronic experimental autoimmune encephalomyelitis |
| Q37754572 | T-cell response dynamics in animal models of multiple sclerosis: implications for immunotherapies |
| Q34230758 | The Fas signaling connection between autoimmunity and embryonic lethality |
| Q36938320 | The Peyer's patch is a critical immunoregulatory site for mucosal tolerance in experimental autoimmune encephalomylelitis (EAE) |
| Q47919335 | The development of clinical activity in relapsing-remitting MS is associated with a decrease of FasL mRNA and an increase of Fas mRNA in peripheral blood |
| Q34458562 | The docosatriene protectin D1 is produced by TH2 skewing and promotes human T cell apoptosis via lipid raft clustering |
| Q28139188 | The multifaceted role of Fas signaling in immune cell homeostasis and autoimmunity |
| Q34457197 | The role of Fas ligand in immune privilege |
| Q34268029 | The role of antigen presenting cells in multiple sclerosis |
| Q33898921 | The role of fas ligand in vivo as a cause and regulator of pathogenesis |
| Q34217243 | Unlike Th1, Th17 cells mediate sustained autoimmune inflammation and are highly resistant to restimulation-induced cell death |
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