scholarly article | Q13442814 |
P50 | author | E. Cristy Ruteshouser | Q64848353 |
Qianghua Hu | Q104615614 | ||
Vicki Huff | Q59617066 | ||
P2093 | author name string | M John Hicks | |
Sharada Mokkapati | |||
Le Huang | |||
P2860 | cites work | Intestinal polyposis in mice with a dominant stable mutation of the beta-catenin gene | Q24529898 |
Disruption of imprinting caused by deletion of the H19 gene region in mice | Q28287765 | ||
Homeobox genes and connective tissue patterning | Q28300079 | ||
WT-1 is required for early kidney development | Q28512266 | ||
Glycogen synthase kinase-3 inactivation and stabilization of beta-catenin induce nephron differentiation in isolated mouse and rat kidney mesenchymes | Q28584354 | ||
Nephric lineage specification by Pax2 and Pax8 | Q28585002 | ||
Six2 is required for suppression of nephrogenesis and progenitor renewal in the developing kidney | Q28586205 | ||
Six2 defines and regulates a multipotent self-renewing nephron progenitor population throughout mammalian kidney development | Q28592487 | ||
Cited1 and Cited2 are differentially expressed in the developing kidney but are not required for nephrogenesis | Q28593339 | ||
Osr1 expression demarcates a multi-potent population of intermediate mesoderm that undergoes progressive restriction to an Osr1-dependent nephron progenitor compartment within the mammalian kidney | Q28594297 | ||
Six2 and Wnt regulate self-renewal and commitment of nephron progenitors through shared gene regulatory networks | Q28594605 | ||
A robust and high-throughput Cre reporting and characterization system for the whole mouse brain | Q29616609 | ||
Efficient recombination in diverse tissues by a tamoxifen-inducible form of Cre: a tool for temporally regulated gene activation/inactivation in the mouse | Q29620355 | ||
Fate tracing reveals the pericyte and not epithelial origin of myofibroblasts in kidney fibrosis | Q33556248 | ||
Frequent association of beta-catenin and WT1 mutations in Wilms tumors. | Q33927092 | ||
Single cell dissection of early kidney development: multilineage priming | Q34341652 | ||
Wt1 ablation and Igf2 upregulation in mice result in Wilms tumors with elevated ERK1/2 phosphorylation | Q34428960 | ||
Identification of a multipotent self-renewing stromal progenitor population during mammalian kidney organogenesis | Q34469453 | ||
The Wilms tumor gene, Wt1, is required for Sox9 expression and maintenance of tubular architecture in the developing testis | Q35033140 | ||
Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation | Q35279994 | ||
β-Catenin and K-RAS synergize to form primitive renal epithelial tumors with features of epithelial Wilms' tumors | Q35679310 | ||
Clinically relevant subsets identified by gene expression patterns support a revised ontogenic model of Wilms tumor: a Children's Oncology Group Study | Q36200428 | ||
Recurrent DGCR8, DROSHA, and SIX homeodomain mutations in favorable histology Wilms tumors. | Q36709112 | ||
Role for compartmentalization in nephron progenitor differentiation | Q36712738 | ||
Fate mapping using Cited1-CreERT2 mice demonstrates that the cap mesenchyme contains self-renewing progenitor cells and gives rise exclusively to nephronic epithelia. | Q37235452 | ||
High-resolution gene expression analysis of the developing mouse kidney defines novel cellular compartments within the nephron progenitor population | Q37356678 | ||
β-catenin activation in a novel liver progenitor cell type is sufficient to cause hepatocellular carcinoma and hepatoblastoma | Q38993024 | ||
Sequential WT1 and CTNNB1 mutations and alterations of beta-catenin localisation in intralobar nephrogenic rests and associated Wilms tumours: two case studies | Q41809711 | ||
Msg1 and Mrg1, founding members of a gene family, show distinct patterns of gene expression during mouse embryogenesis. | Q48038317 | ||
Mutations in the SIX1/2 pathway and the DROSHA/DGCR8 miRNA microprocessor complex underlie high-risk blastemal type Wilms tumors. | Q50601697 | ||
P433 | issue | 2 | |
P304 | page(s) | 71-81 | |
P577 | publication date | 2016-02-01 | |
P1433 | published in | Neoplasia | Q2962042 |
P1476 | title | Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation | |
P478 | volume | 18 |
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Q47280626 | Haploinsufficiency for the Six2 gene increases nephron progenitor proliferation promoting branching and nephron number |
Q89295025 | Loss of Dis3l2 partially phenocopies Perlman syndrome in mice and results in up-regulation of Igf2 in nephron progenitor cells |
Q64970878 | Mutations in microRNA processing genes in Wilms tumors derepress the IGF2 regulator PLAG1. |
Q90697631 | Overgrowth syndromes and pediatric cancers: how many roads lead to IGF2? |
Q47110433 | Pharmacologic Inhibition of β-Catenin With Pyrvinium Inhibits Murine and Human Models of Wilms Tumor |
Q64236688 | Platforms of in vivo genome editing with inducible Cas9 for advanced cancer modeling |
Q55318193 | Wilms' Tumor Protein 1 and Enzymatic Oxidation of 5-Methylcytosine in Brain Tumors: Potential Perspectives. |
Q47948619 | Wilms' tumour 1 (WT1) in development, homeostasis and disease. |
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