Histone H2AX is integral to hypoxia-driven neovascularization

scientific article published on 19 April 2009

Histone H2AX is integral to hypoxia-driven neovascularization is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1038/NM.1947
P8608Fatcat IDrelease_c6dkl37jgzbeljxz73gw66o6sa
P932PMC publication ID2713773
P698PubMed publication ID19377486
P5875ResearchGate publication ID24308423

P50authorTriantafyllos ChavakisQ28054037
P2093author name stringValeria V Orlova
Athanassios Vassilopoulos
Eun Young Choi
Manfred Boehm
Andre Nussenzweig
Arkady Celeste
Mingchao Ma
Harald F Langer
Craig H Bassing
Chuxia Deng
Elsa Callen
Matina Economopoulou
P2860cites workThe phosphorylation status of PAS-B distinguishes HIF-1alpha from HIF-2alpha in NBS1 repressionQ24306098
Sensing DNA damage through ATRIP recognition of RPA-ssDNA complexesQ27860662
ATM and ATR: networking cellular responses to DNA damageQ28143424
H2AX: the histone guardian of the genomeQ28274279
Genomic instability in mice lacking histone H2AXQ28589826
Angiogenesis in life, disease and medicineQ29614539
Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaksQ29617463
VEGF guides angiogenic sprouting utilizing endothelial tip cell filopodiaQ29617501
The DNA replication checkpoint response stabilizes stalled replication forksQ29618306
Increased ionizing radiation sensitivity and genomic instability in the absence of histone H2AXQ34031720
Revascularization of ischemic tissues by PlGF treatment, and inhibition of tumor angiogenesis, arthritis and atherosclerosis by anti-Flt1.Q34136366
Hypoxia links ATR and p53 through replication arrestQ34324309
Interfaces between the detection, signaling, and repair of DNA damageQ34762844
Histone H2AX in DNA damage and repair.Q35184065
Ocular neovascularization: a valuable model systemQ35551128
H2AX may function as an anchor to hold broken chromosomal DNA ends in close proximityQ35623763
Inhibition of pathologic retinal neovascularization by alpha-defensinsQ35847765
The role of ATM and ATR in the cellular response to hypoxia and re-oxygenationQ35848530
The DNA damage response: sensing and signalingQ35941800
Junctional adhesion molecule-C regulates vascular endothelial permeability by modulating VE-cadherin-mediated cell-cell contactsQ36228150
Turning the replication checkpoint on and off.Q36343227
Oxygen-dependent diseases in the retina: role of hypoxia-inducible factorsQ36496079
Hypoxia-induced genetic instability--a calculated mechanism underlying tumor progressionQ36688644
ATM and ATR: components of an integrated circuitQ36741134
Oxygen sensing and the DNA-damage responseQ37006525
Cell cycle-dependent variations in the distribution of the nuclear protein cyclin proliferating cell nuclear antigen in cultured cells: subdivision of S phaseQ37688155
Blockade of Dll4 inhibits tumour growth by promoting non-productive angiogenesisQ40191493
Hypoxia-induced phosphorylation of Chk2 in an ataxia telangiectasia mutated-dependent mannerQ40345113
Inhibition of ATR leads to increased sensitivity to hypoxia/reoxygenationQ40514725
ATR/ATM targets are phosphorylated by ATR in response to hypoxia and ATM in response to reoxygenationQ40678693
VE-Cadherin-Cre-recombinase transgenic mouse: a tool for lineage analysis and gene deletion in endothelial cells.Q45973228
Caveolin-1-deficient mice have increased tumor microvascular permeability, angiogenesis, and growth.Q50470839
Oxygen-induced retinopathy in the mouseQ71608431
P433issue5
P407language of work or nameEnglishQ1860
P921main subjecthypoxiaQ105688
neovascularizationQ1281049
P304page(s)553-558
P577publication date2009-04-19
P1433published inNature MedicineQ1633234
P1476titleHistone H2AX is integral to hypoxia-driven neovascularization
P478volume15

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