scholarly article | Q13442814 |
P2093 | author name string | Joshua M Thurman | |
Brandon Renner | |||
James W McCullough | |||
P2860 | cites work | Rapid induction of neutrophil–endothelial adhesion by endothelial complement fixation | Q59054348 |
C4b-binding Protein and Factor H Compensate for the Loss of Membrane-bound Complement Inhibitors to Protect Apoptotic Cells against Excessive Complement Attack | Q60250539 | ||
Complement activation retards resolution of acute ischemic renal failure in the rat | Q67804884 | ||
Soluble human complement receptor type 1: in vivo inhibitor of complement suppressing post-ischemic myocardial inflammation and necrosis | Q68856319 | ||
Initiation of the alternative complement pathway due to spontaneous hydrolysis of the thioester of C3 | Q70939345 | ||
Anaphylatoxin formation during hemodialysis: effects of different dialyzer membranes | Q71006733 | ||
Regulated expression of complement factor B in the human kidney | Q71612640 | ||
Localization of the complement regulatory proteins in the normal human kidney | Q71651834 | ||
Ischemic acute tubular necrosis induces an extensive local cytokine response. Evidence for induction of interferon-gamma, transforming growth factor-beta 1, granulocyte-macrophage colony-stimulating factor, interleukin-2, and interleukin-10 | Q72601437 | ||
Biocompatible membranes in acute renal failure: prospective case-controlled study | Q72688835 | ||
The C5a receptor is expressed in normal renal proximal tubular but not in normal pulmonary or hepatic epithelial cells | Q73395775 | ||
Evaluation of haemodialysis membrane biocompatibility by parallel assessment in an ex vivo model in healthy volunteers | Q73718941 | ||
Pathophysiological role of T lymphocytes in renal ischemia-reperfusion injury in mice | Q74247708 | ||
Blockade of antibody-induced glomerulonephritis with Crry-Ig, a soluble murine complement inhibitor | Q74515425 | ||
Ischemia/reperfusion-induced IFN-gamma up-regulation: involvement of IL-12 and IL-18 | Q77399412 | ||
Up-regulation of intracellular calcium, cyclic adenosine monophosphate and fibronectin synthesis in tubuar epithelial cells by complement | Q77402483 | ||
Complement regulatory proteins in glomerular diseases | Q77650589 | ||
Mannose-binding lectin binds IgM to activate the lectin complement pathway in vitro and in vivo | Q79369849 | ||
The differing roles of the classical and mannose-binding lectin complement pathways in the events following skeletal muscle ischemia-reperfusion | Q79370206 | ||
Acute renal failure after bilateral nephrectomy is associated with cytokine-mediated pulmonary injury | Q79431461 | ||
Unrestricted C3 activation occurs in Crry-deficient kidneys and rapidly leads to chronic renal failure | Q79578913 | ||
C3a is required for the production of CXC chemokines by tubular epithelial cells after renal ishemia/reperfusion | Q79609757 | ||
Intravenous immunoglobulin attenuates mesenteric ischemia-reperfusion injury | Q81245282 | ||
Acute tubular necrosis is characterized by activation of the alternative pathway of complement | Q81324722 | ||
Local extravascular pool of C3 is a determinant of postischemic acute renal failure | Q82436381 | ||
Endothelial oxidative stress activates the lectin complement pathway: role of cytokeratin 1 | Q24291669 | ||
Acute renal failure: definitions, diagnosis, pathogenesis, and therapy | Q24563291 | ||
C5a-induced expression of P-selectin in endothelial cells | Q24633672 | ||
Expression of complement components differs between kidney allografts from living and deceased donors | Q24654702 | ||
C-Reactive protein binds to apoptotic cells, protects the cells from assembly of the terminal complement components, and sustains an antiinflammatory innate immune response: implications for systemic autoimmunity | Q24675774 | ||
Structure and biology of complement protein C3, a connecting link between innate and acquired immunity | Q28202590 | ||
C3d of complement as a molecular adjuvant: bridging innate and acquired immunity | Q28272810 | ||
The internal thioester and the covalent binding properties of the complement proteins C3 and C4 | Q28304143 | ||
Requirement and role of C5a in acute lung inflammatory injury in rats | Q28569055 | ||
Role of membrane cofactor protein (CD46) in regulation of C4b and C3b deposited on cells | Q28610892 | ||
Initiation of the alternative pathway of complement: recognition of activators by bound C3b and assembly of the entire pathway from six isolated proteins | Q28610965 | ||
Formation of the initial C3 convertase of the alternative complement pathway. Acquisition of C3b-like activities by spontaneous hydrolysis of the putative thioester in native C3 | Q28610996 | ||
Macrophages, dendritic cells, and kidney ischemia-reperfusion injury | Q30432565 | ||
Human serum IgM glycosylation: identification of glycoforms that can bind to mannan-binding lectin | Q33217116 | ||
The simple design of complement factor H: Looks can be deceiving | Q33372312 | ||
Differential impact of complement mutations on clinical characteristics in atypical hemolytic uremic syndrome | Q33375728 | ||
Translational mini-review series on complement factor H: renal diseases associated with complement factor H: novel insights from humans and animals. | Q33378236 | ||
Complement regulators and inhibitory proteins | Q33386110 | ||
Atypical hemolytic-uremic syndrome | Q33386736 | ||
Eculizumab for atypical hemolytic uremic syndrome recurrence in renal transplantation. | Q33403196 | ||
Use of eculizumab for atypical haemolytic uraemic syndrome and C3 glomerulopathies | Q33403607 | ||
Complement genes strongly predict recurrence and graft outcome in adult renal transplant recipients with atypical hemolytic and uremic syndrome | Q33405522 | ||
Pathogenic natural antibodies recognizing annexin IV are required to develop intestinal ischemia-reperfusion injury | Q33646877 | ||
Targeted and restricted complement activation on acrosome-reacted spermatozoa | Q33743670 | ||
Therapeutic targeting of classical and lectin pathways of complement protects from ischemia-reperfusion-induced renal damage | Q33745120 | ||
Expression of the anaphylatoxin C5a receptor in non-myeloid cells | Q33852657 | ||
Targeted complement inhibition by C3d recognition ameliorates tissue injury without apparent increase in susceptibility to infection | Q33925353 | ||
Renal-associated TLR2 mediates ischemia/reperfusion injury in the kidney | Q33948196 | ||
Genetic and therapeutic targeting of properdin in mice prevents complement-mediated tissue injury | Q34162118 | ||
Targeting of mannan-binding lectin-associated serine protease-2 confers protection from myocardial and gastrointestinal ischemia/reperfusion injury. | Q34179243 | ||
Interleukin 2 mediates stimulation of complement C3 biosynthesis in human proximal tubular epithelial cells | Q34191473 | ||
AKI in the ICU: definition, epidemiology, risk stratification, and outcomes | Q34221657 | ||
Complement-targeted therapeutics | Q34255011 | ||
Altered renal tubular expression of the complement inhibitor Crry permits complement activation after ischemia/reperfusion. | Q34315819 | ||
Pathophysiology of chronic tubulo-interstitial disease in rats. Interactions of dietary acid load, ammonia, and complement component C3. | Q34548269 | ||
Deficiency of C4 from donor or recipient mouse fails to prevent renal allograft rejection | Q35088132 | ||
Complement receptor 2-mediated targeting of complement inhibitors to sites of complement activation | Q35095565 | ||
B cell subsets contribute to renal injury and renal protection after ischemia/reperfusion | Q35097477 | ||
Binding of factor H to tubular epithelial cells limits interstitial complement activation in ischemic injury | Q35097512 | ||
The complement inhibitors Crry and factor H are critical for preventing autologous complement activation on renal tubular epithelial cells. | Q35097528 | ||
Cellular pathophysiology of ischemic acute kidney injury | Q35484725 | ||
Alternative complement pathway in the pathogenesis of disease mediated by anti-neutrophil cytoplasmic autoantibodies | Q35570402 | ||
Tubulointerstitial injury induced in rats by a monoclonal antibody that inhibits function of a membrane inhibitor of complement | Q35766363 | ||
Acute renal failure and sepsis | Q35833149 | ||
Ischemic acute renal failure: an inflammatory disease? | Q35836380 | ||
Potentiation of the anaphylatoxins in vivo using an inhibitor of serum carboxypeptidase N (SCPN). I. Lethality and pathologic effects on pulmonary tissue | Q35885799 | ||
TLR4 activation mediates kidney ischemia/reperfusion injury | Q35987574 | ||
Innate autoimmunity | Q36037749 | ||
C3a and C5a promote renal ischemia-reperfusion injury | Q36200905 | ||
Identification of the target self-antigens in reperfusion injury | Q36227933 | ||
Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement | Q36366602 | ||
The central role of the alternative complement pathway in human disease | Q36372905 | ||
Transgenic mice overexpressing the complement inhibitor crry as a soluble protein are protected from antibody-induced glomerular injury | Q36401591 | ||
Mannan binding lectin and its interaction with immunoglobulins in health and in disease. | Q36524747 | ||
Blocking properdin, the alternative pathway, and anaphylatoxin receptors ameliorates renal ischemia-reperfusion injury in decay-accelerating factor and CD59 double-knockout mice | Q36718295 | ||
C3a mediates epithelial-to-mesenchymal transition in proteinuric nephropathy | Q37123256 | ||
Complement and renal transplantation: from donor to recipient | Q37137507 | ||
Immunoglobulin and complement deposition in glomeruli of 756 subjects who had committed suicide or met with a violent death | Q37237720 | ||
The tick-over theory revisited: formation and regulation of the soluble alternative complement C3 convertase (C3(H2O)Bb) | Q37240329 | ||
A novel targeted inhibitor of the alternative pathway of complement and its therapeutic application in ischemia/reperfusion injury | Q37434667 | ||
Dynamic control of the complement system by modulated expression of regulatory proteins | Q37797869 | ||
The mannose-binding lectin-pathway is involved in complement activation in the course of renal ischemia-reperfusion injury | Q38782859 | ||
Quantitative analysis of adenine nucleotides during the prelytic phase of cell death mediated by C5b-9. | Q39250858 | ||
Structure and specificity of complement receptors. | Q39764682 | ||
The cytokine-adhesion molecule cascade in ischemia/reperfusion injury of the rat kidney. Inhibition by a soluble P-selectin ligand | Q39785346 | ||
Complement activation by tubular cells is mediated by properdin binding | Q39946407 | ||
CD46 plays a key role in tailoring innate immune recognition of apoptotic and necrotic cells | Q40387246 | ||
Predominant role for C5b-9 in renal ischemia/reperfusion injury. | Q40437461 | ||
The C5a receptor is expressed by human renal proximal tubular epithelial cells | Q40863588 | ||
Transforming growth factor-beta 1 regulates chemokine and complement production by human proximal tubular epithelial cells | Q41058011 | ||
Nontransgenic Hyperexpression of a Complement Regulator in Donor Kidney Modulates Transplant Ischemia/Reperfusion Damage, Acute Rejection, and Chronic Nephropathy | Q41090414 | ||
The cytolytically inactive terminal complement complex activates endothelial cells to express adhesion molecules and tissue factor procoagulant activity | Q41923727 | ||
Specificity of the thioester-containing reactive site of human C3 and its significance to complement activation | Q42828955 | ||
Inhibiting the complement system does not reduce injury in renal ischemia reperfusion | Q43651985 | ||
Detection of the complement degradation product C4d in renal allografts: diagnostic and therapeutic implications. | Q43830010 | ||
Injury in renal ischemia-reperfusion is independent from immunoglobulins and T lymphocytes | Q43852773 | ||
The effect of pH and nucleophiles on complement activation by human proximal tubular epithelial cells | Q43974278 | ||
Classical pathway of complement activation in normal and diseased human glomeruli | Q44026716 | ||
A small molecule C5a receptor antagonist protects kidneys from ischemia/reperfusion injury in rats | Q44242757 | ||
Lack of a functional alternative complement pathway ameliorates ischemic acute renal failure in mice | Q44285426 | ||
Inhibition of complement factor C5 protects against renal ischemia-reperfusion injury: inhibition of late apoptosis and inflammation. | Q44317230 | ||
Properdin can initiate complement activation by binding specific target surfaces and providing a platform for de novo convertase assembly | Q44441855 | ||
Changes in neutrophil surface-receptor expression after stimulation with FMLP, endotoxin, interleukin-8 and activated complement compared to degranulation | Q44728179 | ||
Role of Toll-like receptor 4 in endotoxin-induced acute renal failure | Q44757046 | ||
Critical protection from renal ischemia reperfusion injury by CD55 and CD59. | Q44788674 | ||
Renal expression of the C3a receptor and functional responses of primary human proximal tubular epithelial cells | Q45050747 | ||
Mannan-binding lectin recognizes structures on ischaemic reperfused mouse kidneys and is implicated in tissue injury | Q46480573 | ||
Signaling through up-regulated C3a receptor is key to the development of experimental lupus nephritis | Q46610901 | ||
Treatment with an inhibitory monoclonal antibody to mouse factor B protects mice from induction of apoptosis and renal ischemia/reperfusion injury. | Q46935293 | ||
Complement factor C5a mediates renal ischemia-reperfusion injury independent from neutrophils. | Q48024813 | ||
Renal ischemia/reperfusion leads to macrophage-mediated increase in pulmonary vascular permeability. | Q50518455 | ||
Design of clinical trials in acute kidney injury: report from an NIDDK workshop on trial methodology. | Q50955754 | ||
Biocompatibility of hemodialysis membranes: a study in healthy subjects. | Q51597719 | ||
Nucleophilic modification of human complement protein C3: correlation of conformational changes with acquisition of C3b-like functional properties | Q52735864 | ||
Local renal complement C3 induction by donor brain death is associated with reduced renal allograft function after transplantation. | Q54399040 | ||
Cytolytically inactive terminal complement complex causes transendothelial migration of polymorphonuclear leukocytes in vitro and in vivo | Q56943166 | ||
Mice Deficient in Complement Receptors 1 and 2 Lack a Tissue Injury-Inducing Subset of the Natural Antibody Repertoire | Q57331400 | ||
Local synthesis of complement component C3 regulates acute renal transplant rejection | Q57369617 | ||
P433 | issue | 6 | |
P304 | page(s) | 543-556 | |
P577 | publication date | 2013-11-01 | |
P1433 | published in | Seminars in Nephrology | Q15761561 |
P1476 | title | The role of the complement system in acute kidney injury | |
P478 | volume | 33 |
Q88050198 | A zebrafish model of infection-associated acute kidney injury |
Q48055653 | Anti-LG3 Antibodies Aggravate Renal Ischemia-Reperfusion Injury and Long-Term Renal Allograft Dysfunction |
Q90705177 | Complement Inhibitors in Clinical Trials for Glomerular Diseases |
Q33432423 | Complement in disease: a defence system turning offensive |
Q97070834 | Complement levels in patients with bloodstream infection due to Staphylococcus aureus or Gram-negative bacteria |
Q34501875 | Complementing the inflammasome. |
Q35406436 | Crescentic acute glomerulonephritis with isolated C3 deposition: a case report and review of literature |
Q38187566 | Delayed kidney graft function: from mechanism to translation |
Q53665622 | Effects of melatonin on the serum levels of pro-inflammatory cytokines and tissue injury after renal ischemia reperfusion in rats. |
Q56341973 | Endothelial Microparticles and Systemic Complement Activation in Patients With Chronic Kidney Disease |
Q55219729 | Epithelial C5aR1 Signaling Enhances Uropathogenic Escherichia coli Adhesion to Human Renal Tubular Epithelial Cells. |
Q41106073 | Human Tamm-Horsfall protein, a renal specific protein, serves as a cofactor in complement 3b degradation. |
Q90076810 | Human mesenchymal stromal cells ameliorate complement induced inflammatory cascade and improve renal functions in a rat model of ischemia-reperfusion induced acute kidney injury |
Q34484801 | Immunosuppressive Medications |
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Q46776795 | Oxidative Stress and Acute Kidney Injury in Critical Illness: Pathophysiologic Mechanisms-Biomarkers-Interventions, and Future Perspectives. |
Q38645385 | Potential Roles for C1 Inhibitor in Transplantation |
Q91711957 | Trib1 Contributes to Recovery From Ischemia/Reperfusion-Induced Acute Kidney Injury by Regulating the Polarization of Renal Macrophages |
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