scholarly article | Q13442814 |
P50 | author | Balakrishnan Ganapathy | Q84649467 |
P2093 | author name string | Stuart Walsh | |
Brian D Polizzotti | |||
Bernhard Kühn | |||
Shima Arab | |||
Sangita Choudhury | |||
Kevin Bersell | |||
Brian Wadugu | |||
Mariya Mollova | |||
P2860 | cites work | Cardiac-Specific Loss of N-Cadherin Leads to Alteration in Connexins With Conduction Slowing and Arrhythmogenesis | Q57396516 |
Transgenic expression of green fluorescence protein can cause dilatedcardiomyopathy | Q61995637 | ||
Formation of Neu/ErbB2-induced mammary tumors is unaffected by loss of ErbB4 | Q83230378 | ||
Switch from caspase-dependent to caspase-independent death during heart development: essential role of endonuclease G in ischemia-induced DNA processing of differentiated cardiomyocytes | Q83929474 | ||
Intrapericardial delivery of gelfoam enables the targeted delivery of Periostin peptide after myocardial infarction by inducing fibrin clot formation | Q27303167 | ||
A new mathematical model for relative quantification in real-time RT-PCR | Q27860781 | ||
Generalized lacZ expression with the ROSA26 Cre reporter strain | Q27860837 | ||
Cre recombinase: the universal reagent for genome tailoring | Q28145361 | ||
Neuregulin1/ErbB4 signaling induces cardiomyocyte proliferation and repair of heart injury | Q28252995 | ||
Hypoxia and acidosis activate cardiac myocyte death through the Bcl-2 family protein BNIP3. | Q28577311 | ||
DNA targeting specificity of RNA-guided Cas9 nucleases | Q29615793 | ||
High-frequency off-target mutagenesis induced by CRISPR-Cas nucleases in human cells | Q29616045 | ||
Growth inhibition and DNA damage induced by Cre recombinase in mammalian cells | Q33930407 | ||
Lineage tracing | Q34248722 | ||
Evidence from a genetic fate-mapping study that stem cells refresh adult mammalian cardiomyocytes after injury | Q34657173 | ||
Systolic dysfunction in cardiac-specific ligand-inducible MerCreMer transgenic mice | Q35087050 | ||
Illegitimate Cre-dependent chromosome rearrangements in transgenic mouse spermatids | Q35573350 | ||
Cre-mediated recombination can induce apoptosis in vivo by activating the p53 DNA damage-induced pathway | Q35740259 | ||
Cryptic loxP sites in mammalian genomes: genome-wide distribution and relevance for the efficiency of BAC/PAC recombineering techniques. | Q35781787 | ||
RIP-Cre revisited, evidence for impairments of pancreatic beta-cell function | Q36330860 | ||
Vagaries of conditional gene targeting | Q36853628 | ||
Role of caspase-independent apoptosis in cardiovascular diseases | Q36992481 | ||
With great power comes great responsibility: using mouse genetics to study cardiac hypertrophy and failure | Q37101602 | ||
High-throughput profiling of off-target DNA cleavage reveals RNA-programmed Cas9 nuclease specificity | Q37196274 | ||
Cardiac fibrosis in mice expressing an inducible myocardial-specific Cre driver. | Q37287472 | ||
Role of AIF in cardiac apoptosis in hypertrophic cardiomyocytes from Dahl salt-sensitive rats | Q37467834 | ||
Unsuspected effects of a lung-specific Cre deleter mouse line | Q39594755 | ||
Inducible site-directed recombination in mouse embryonic stem cells | Q39715564 | ||
Cre activity causes widespread apoptosis and lethal anemia during embryonic development. | Q40146225 | ||
Self-excising retroviral vectors encoding the Cre recombinase overcome Cre-mediated cellular toxicity | Q40785247 | ||
Mammalian genomes contain active recombinase recognition sites | Q40898133 | ||
High levels of Cre expression in neuronal progenitors cause defects in brain development leading to microencephaly and hydrocephaly. | Q41919230 | ||
Avoidance of transient cardiomyopathy in cardiomyocyte-targeted tamoxifen-induced MerCreMer gene deletion models | Q42560803 | ||
Cre-loxP DNA recombination is possible with only minimal unspecific transcriptional changes and without cardiomyopathy in Tg(alphaMHC-MerCreMer) mice | Q42923082 | ||
Temporally regulated and tissue-specific gene manipulations in the adult and embryonic heart using a tamoxifen-inducible Cre protein | Q43664631 | ||
Direct hematological toxicity and illegitimate chromosomal recombination caused by the systemic activation of CreERT2. | Q46043692 | ||
Tamoxifen modulates apoptosis in multiple modes of action in CreER mice | Q46185200 | ||
Behavioral characteristics of a nervous system-specific erbB4 knock-out mouse | Q46523281 | ||
Effect of caspase inhibitors on myocardial infarct size and myocyte DNA fragmentation in the ischemia-reperfused rat heart | Q47243708 | ||
Toxicity of ligand-dependent Cre recombinases and generation of a conditional Cre deleter mouse allowing mosaic recombination in peripheral tissues | Q48189802 | ||
Dilated cardiomyopathy resulting from high-level myocardial expression of Cre-recombinase. | Q48505009 | ||
P275 | copyright license | Creative Commons Attribution 2.0 Generic | Q19125117 |
P6216 | copyright status | copyrighted | Q50423863 |
P433 | issue | 6 | |
P921 | main subject | heart failure | Q181754 |
tamoxifen | Q412178 | ||
DNA damage | Q5205747 | ||
P304 | page(s) | 1459-1469 | |
P577 | publication date | 2013-08-07 | |
P1433 | published in | Disease Models & Mechanisms | Q1524006 |
P1476 | title | Moderate and high amounts of tamoxifen in αMHC-MerCreMer mice induce a DNA damage response, leading to heart failure and death | |
P478 | volume | 6 |