| human | Q5 |
| P6178 | Dimensions author ID | 01370076050.75 |
| P856 | official website | https://research.qut.edu.au/translationalgenomicsgroup/people/tony-kenna/ |
| P496 | ORCID iD | 0000-0001-6844-3463 |
| P1153 | Scopus author ID | 6602781126 |
| P10861 | Springer Nature person ID | 01370076050.75 |
| P69 | educated at | University College Dublin | Q1068258 |
| Dublin City University School of Biotechnology | Q101012436 | ||
| P108 | employer | University College Dublin | Q1068258 |
| Queensland University of Technology | Q1144750 | ||
| P734 | family name | Kenna | Q37032336 |
| Kenna | Q37032336 | ||
| Kenna | Q37032336 | ||
| P735 | given name | Tony | Q15238167 |
| Tony | Q15238167 | ||
| P106 | occupation | researcher | Q1650915 |
| P21 | sex or gender | male | Q6581097 |
| Q42403489 | A combination of local inflammation and central memory T cells potentiates immunotherapy in the skin |
| Q24650483 | A potent nonporphyrin class of photodynamic therapeutic agent: cellular localisation, cytotoxic potential and influence of hypoxia |
| Q34412203 | A promoter-level mammalian expression atlas |
| Q51887160 | Antigen-specific CD4 cells assist CD8 T-effector cells in eliminating keratinocytes. |
| Q35151883 | Blockade of PD-1/PD-L1 promotes adoptive T-cell immunotherapy in a tolerogenic environment |
| Q41652119 | Brief Report: Intestinal Dysbiosis in Ankylosing Spondylitis |
| Q54586481 | CD1 expression and CD1-restricted T cell activity in normal and tumour-bearing human liver. |
| Q40177092 | Chemotherapy and zoledronate sensitize solid tumour cells to Vgamma9Vdelta2 T cell cytotoxicity |
| Q40286194 | Chemotherapy pretreatment sensitizes solid tumor-derived cell lines to V alpha 24+ NKT cell-mediated cytotoxicity |
| Q57667207 | Disease-associated polymorphisms in ERAP1 do not alter endoplasmic reticulum stress in patients with ankylosing spondylitis |
| Q57667278 | Distinct subpopulations of ?? T cells are present in normal and tumor-bearing human liver |
| Q57667281 | Diverse populations of T cells with NK cell receptors accumulate in the human intestine in health and in colorectal cancer |
| Q35165514 | Does the microbiome play a causal role in spondyloarthritis? |
| Q48122051 | ERAP2 functional knockout in humans does not alter surface heavy chains or HLA-B27, inflammatory cytokines or endoplasmic reticulum stress markers |
| Q38526791 | Endoplasmic reticulum aminopeptidases in the pathogenesis of ankylosing spondylitis. |
| Q38432390 | Epigenetic and gene expression analysis of ankylosing spondylitis-associated loci implicate immune cells and the gut in the disease pathogenesis |
| Q98508242 | Epistatic interactions between killer immunoglobulin-like receptors and human leukocyte antigen ligands are associated with ankylosing spondylitis |
| Q38604042 | FANTOM5 CAGE profiles of human and mouse samples |
| Q38958165 | Functional Genomics and Its Bench-to-Bedside Translation Pertaining to the Identified Susceptibility Alleles and Loci in Ankylosing Spondylitis. |
| Q53614088 | Genetic association of ankylosing spondylitis with TBX21 influences T-bet and pro-inflammatory cytokine expression in humans and SKG mice as a model of spondyloarthritis. |
| Q38599986 | Genetics of ankylosing spondylitis--insights into pathogenesis |
| Q64077628 | Genome-wide association study in Turkish and Iranian populations identify rare familial Mediterranean fever gene (MEFV) polymorphisms associated with ankylosing spondylitis |
| Q41526747 | Human Blood CD1c+ Dendritic Cells Promote Th1 and Th17 Effector Function in Memory CD4+ T Cells |
| Q30417517 | Identification of multiple risk variants for ankylosing spondylitis through high-density genotyping of immune-related loci |
| Q33206210 | In vitro demonstration of the heavy-atom effect for photodynamic therapy. |
| Q36525920 | Inflammation-driven bone formation in a mouse model of ankylosing spondylitis: sequential not parallel processes. |
| Q29417082 | Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility |
| Q33765449 | Microbes, the gut and ankylosing spondylitis |
| Q50521211 | Mutations in LTBP3 cause acromicric dysplasia and geleophysic dysplasia. |
| Q40119340 | Severe viral respiratory infections in children with IFIH1 loss-of-function mutations. |
| Q57667259 | Steady-state dendritic cells expressing cognate antigen terminate memory CD8+ T-cell responses |
| Q44234403 | Targeting antigen to diverse APCs inactivates memory CD8+ T cells without eliciting tissue-destructive effector function |
| Q57667231 | The role of IL-17-secreting mast cells in inflammatory joint disease |
| Q40081875 | Transcriptome analysis of ankylosing spondylitis patients before and after TNF-α inhibitor therapy reveals the pathways affected. |
| Q21144959 | Whole-exome re-sequencing in a family quartet identifies POP1 mutations as the cause of a novel skeletal dysplasia |
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