Genetic subgrouping of melanoma reveals new opportunities for targeted therapy

scientific article published on 07 April 2009

Genetic subgrouping of melanoma reveals new opportunities for targeted therapy is …
instance of (P31):
scholarly articleQ13442814
review articleQ7318358

External links are
P356DOI10.1158/0008-5472.CAN-08-4305
P698PubMed publication ID19351826
P5875ResearchGate publication ID24262768

P50authorKatherine L NathansonQ37369859
Keiran S M SmalleyQ61525717
Keith FlahertyQ6384357
P433issue8
P407language of work or nameEnglishQ1860
P304page(s)3241-3244
P577publication date2009-04-07
P1433published inCancer ResearchQ326097
P1476titleGenetic subgrouping of melanoma reveals new opportunities for targeted therapy
P478volume69

Reverse relations

cites work (P2860)
Q56898613Q56898613
Q27851611A Variant in a MicroRNA complementary site in the 3' UTR of the KIT oncogene increases risk of acral melanoma
Q34259205A brief history of melanoma: from mummies to mutations
Q37815139A new era: melanoma genetics and therapeutics
Q28817145A novel compound which sensitizes BRAF wild-type melanoma cells to vemurafenib in a TRIM16-dependent manner
Q34986199Adjuvant Immunotherapy and Radiation in the Management of High-risk Resected Melanoma.
Q41360812Autocrine insulin-like growth factor 1 and stem cell factor but not interleukin 6 support self-renewal of human myeloma cells
Q34217755Canine malignant melanoma alpha-3 integrin binding peptides
Q64007381Carbazole Derivatives' Binding to c-KIT G-Quadruplex DNA
Q27349020Combined targeting of BRAF and CRAF or BRAF and PI3K effector pathways is required for efficacy in NRAS mutant tumors
Q94564084Comprehensive Genomic Profiling of Rare Tumors: Routes to Targeted Therapies
Q39712781Differential sensitivity of melanoma cell lines with BRAFV600E mutation to the specific Raf inhibitor PLX4032.
Q34020065Electrochemotherapy as an adjunct or alternative to other treatments for unresectable or in-transit melanoma
Q34104555Essential requirement for PP2A inhibition by the oncogenic receptor c-KIT suggests PP2A reactivation as a strategy to treat c-KIT+ cancers
Q42785249G-quadruplex-binding benzo[a]phenoxazines down-regulate c-KIT expression in human gastric carcinoma cells
Q38500731Inflammatory signaling compromises cell responses to interferon alpha
Q28475549Integrated ligand-receptor bioinformatic and in vitro functional analysis identifies active TGFA/EGFR signaling loop in papillary thyroid carcinomas
Q40009179Management of melanoma brain metastases in the era of targeted therapy
Q37727077Measurement of constitutive MAPK and PI3K/AKT signaling activity in human cancer cell lines
Q37605787Melanoma biomarkers: current status and utility in diagnosis, prognosis, and response to therapy
Q24606438PLX4032, a selective BRAF(V600E) kinase inhibitor, activates the ERK pathway and enhances cell migration and proliferation of BRAF melanoma cells
Q37799845PLX4032: does it keep its promise for metastatic melanoma treatment?
Q34761941PTEN loss confers BRAF inhibitor resistance to melanoma cells through the suppression of BIM expression
Q35320304Phosphoproteomic analysis of basal and therapy-induced adaptive signaling networks in BRAF and NRAS mutant melanoma
Q37863222Preparing pathology for personalized medicine: possibilities for improvement of the pre-analytical phase.
Q54497681Sinonasal mucosal melanoma: Molecular profile and therapeutic implications from a series of 32 cases.
Q37873463State of the art of diagnostic technology for early-stage melanoma
Q37245787The Conundrum of Genetic "Drivers" in Benign Conditions
Q35878248The current state of targeted therapy in melanoma: this time it's personal
Q37731451The glutamate release inhibitor Riluzole decreases migration, invasion, and proliferation of melanoma cells
Q38337903Towards the lean lab: the industry challenge
Q34802932Tumor heterogeneity and strategies to overcome kinase inhibitor resistance in cancer: lessons from melanoma
Q82623062[Advances in molecular biology and their application in the diagnosis and treatment of melanoma]
Q39719008c-Abl and Arg are activated in human primary melanomas, promote melanoma cell invasion via distinct pathways, and drive metastatic progression

Search more.