Cerebellar hypoplasia in mice lacking selenoprotein biosynthesis in neurons

scientific article published on 06 March 2014

Cerebellar hypoplasia in mice lacking selenoprotein biosynthesis in neurons is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1007/S12011-014-9920-Z
P932PMC publication ID3984410
P698PubMed publication ID24599700
P5875ResearchGate publication ID260558792

P50authorMarcus ConradQ56517307
Eva K WirthQ40281864
Ulrich SchweizerQ40281900
P2093author name stringMarkus Brielmeier
Dolph Hatfield
B Suman Bharathi
P2860cites workSelenium and brain function: a poorly recognized liaisonQ56611276
Whole-mount immunohistochemistry: a high-throughput screen for patterning defects in the mouse cerebellumQ77513338
Essential role for mitochondrial thioredoxin reductase in hematopoiesis, heart development, and heart functionQ24563493
Characterization of mammalian selenoproteomes.Q27863319
Neurological dysfunction occurs in mice with targeted deletion of the selenoprotein P geneQ28508731
The level of sonic hedgehog signaling regulates the complexity of cerebellar foliationQ28508934
Ablation of TrkA function in the immune system causes B cell abnormalitiesQ28509375
Deletion of selenoprotein P alters distribution of selenium in the mouseQ28512024
Efficient selenium transfer from mother to offspring in selenoprotein-P-deficient mice enables dose-dependent rescue of phenotypes associated with selenium deficiencyQ28512221
Neuronal selenoprotein expression is required for interneuron development and prevents seizures and neurodegenerationQ28591775
The role of thioredoxin reductases in brain developmentQ33324392
12/15-lipoxygenase-derived lipid peroxides control receptor tyrosine kinase signaling through oxidation of protein tyrosine phosphatasesQ34115614
Mutations Disrupting Selenocysteine Formation Cause Progressive Cerebello-Cerebral AtrophyQ34169430
Glutathione peroxidase 4 senses and translates oxidative stress into 12/15-lipoxygenase dependent- and AIF-mediated cell deathQ34818662
Selective removal of the selenocysteine tRNA [Ser]Sec gene (Trsp) in mouse mammary epitheliumQ34820264
Progressive cerebellocerebral atrophy: a new syndrome with microcephaly, mental retardation, and spastic quadriplegia.Q35443213
Patterned Purkinje cell death in the cerebellumQ35563957
Post-transcriptional control of selenoprotein biosynthesisQ38019545
Hepatically derived selenoprotein P is a key factor for kidney but not for brain selenium supplyQ41870693
Gene disruption discloses role of selenoprotein P in selenium delivery to target tissuesQ42127289
Development of a serum-free supplement for primary neuron culture reveals the interplay of selenium and vitamin E in neuronal survivalQ43088552
Specific excision of the selenocysteine tRNA[Ser]Sec (Trsp) gene in mouse liver demonstrates an essential role of selenoproteins in liver functionQ44683592
Neurodegeneration in mice resulting from loss of functional selenoprotein P or its receptor apolipoprotein E receptor 2.Q46825393
Synthesis and metabolism of thyroid hormones is preferentially maintained in selenium-deficient transgenic miceQ46830920
Comparative analysis of selenocysteine machinery and selenoproteome gene expression in mouse brain identifies neurons as key functional sites of selenium in mammalsQ46886702
Hepatic selenoprotein P (SePP) expression restores selenium transport and prevents infertility and motor-incoordination in Sepp-knockout miceQ46931534
Deletion of apolipoprotein E receptor-2 in mice lowers brain selenium and causes severe neurological dysfunction and death when a low-selenium diet is fed.Q48144238
Embryonic development of the rat cerebellum. III. Regional differences in the time of origin, migration, and settling of Purkinje cellsQ48557609
P275copyright licenseCreative Commons Attribution 4.0 InternationalQ20007257
P6216copyright statuscopyrightedQ50423863
P433issue2
P921main subjectcerebellar hypoplasiaQ2214869
P304page(s)203-210
P577publication date2014-03-06
P1433published inBiological Trace Element ResearchQ15716661
P1476titleCerebellar hypoplasia in mice lacking selenoprotein biosynthesis in neurons
P478volume158