scholarly article | Q13442814 |
P2093 | author name string | Rasheed Gbadegesin | |
Peter Lavin | |||
John Foreman | |||
Michelle Winn | |||
P2860 | cites work | Non-corticosteroid treatment for nephrotic syndrome in children | Q24242865 |
Podocin localizes in the kidney to the slit diaphragm area | Q24292141 | ||
Mutations in the formin gene INF2 cause focal segmental glomerulosclerosis | Q24292947 | ||
Regeneration of glomerular podocytes by human renal progenitors | Q24308871 | ||
Positional cloning uncovers mutations in PLCE1 responsible for a nephrotic syndrome variant that may be reversible | Q24312154 | ||
Congenital nephrotic syndrome in mice lacking CD2-associated protein | Q57962338 | ||
Rituximab treatment for severe steroid- or cyclosporine-dependent nephrotic syndrome: a multicentric series of 22 cases | Q61849364 | ||
Mutations in phospholipase C epsilon 1 are not sufficient to cause diffuse mesangial sclerosis | Q62644413 | ||
Nephrin mutations cause childhood- and adult-onset focal segmental glomerulosclerosis | Q63434594 | ||
Nephrotic syndrome in children: prediction of histopathology from clinical and laboratory characteristics at time of diagnosis. A report of the International Study of Kidney Disease in Children | Q67406480 | ||
Treatment of childhood nephrotic syndrome | Q67948621 | ||
Clinicopathological study of nephrotic syndrome in childhood | Q70138834 | ||
Circulating factor associated with increased glomerular permeability to albumin in recurrent focal segmental glomerulosclerosis | Q70989780 | ||
The pathogenesis of 'classic' focal segmental glomerulosclerosis-lessons from rat models | Q73894982 | ||
A hitherto undescribed vulnerability of the juxtamedullary glomeruli in lipoid nephrosis | Q74487217 | ||
Mycophenolate mofetil prevents the induction of active Heymann nephritis: association with Th2 cytokine inhibition | Q77665859 | ||
Cellular focal segmental glomerulosclerosis: Clinical and pathologic features | Q79190024 | ||
Induction of TRPC6 channel in acquired forms of proteinuric kidney disease | Q79431457 | ||
FK506 in the treatment of children with nephrotic syndrome of different pathological types | Q80181461 | ||
Pathological variants of focal segmental glomerulosclerosis in an adult Dutch population--epidemiology and outcome | Q80807199 | ||
Focal segmental glomerulosclerosis in a patient homozygous for a CD2AP mutation | Q80829546 | ||
Positionally cloned gene for a novel glomerular protein--nephrin--is mutated in congenital nephrotic syndrome | Q24314612 | ||
Mutations in LMX1B cause abnormal skeletal patterning and renal dysplasia in nail patella syndrome | Q24317185 | ||
Podocin, a raft-associated component of the glomerular slit diaphragm, interacts with CD2AP and nephrin | Q24564913 | ||
African ancestry allelic variation at the MYH9 gene contributes to increased susceptibility to non-diabetic end-stage kidney disease in Hispanic Americans | Q24602407 | ||
Association of trypanolytic ApoL1 variants with kidney disease in African Americans | Q24606581 | ||
MYH9 is a major-effect risk gene for focal segmental glomerulosclerosis | Q24643421 | ||
MYH9 is associated with nondiabetic end-stage renal disease in African Americans | Q24653027 | ||
Alpha-actinin-4-mediated FSGS: an inherited kidney disease caused by an aggregated and rapidly degraded cytoskeletal protein | Q24793347 | ||
Advances in nephrology from the Necker Hospital | Q27708863 | ||
Bulletin of the Johns Hopkins Hospital | Q27718094 | ||
NPHS2, encoding the glomerular protein podocin, is mutated in autosomal recessive steroid-resistant nephrotic syndrome | Q28116208 | ||
Lipid rafts and signal transduction | Q28131735 | ||
Mutations in ACTN4, encoding alpha-actinin-4, cause familial focal segmental glomerulosclerosis | Q28145757 | ||
CD2-associated protein haploinsufficiency is linked to glomerular disease susceptibility | Q28206197 | ||
Podocin inactivation in mature kidneys causes focal segmental glomerulosclerosis and nephrotic syndrome | Q28256662 | ||
WT1 and glomerular diseases | Q28259503 | ||
Array-based gene discovery with three unrelated subjects shows SCARB2/LIMP-2 deficiency causes myoclonus epilepsy and glomerulosclerosis | Q28270717 | ||
Human laminin beta2 deficiency causes congenital nephrosis with mesangial sclerosis and distinct eye abnormalities | Q28282108 | ||
Mutation in the key enzyme of sialic acid biosynthesis causes severe glomerular proteinuria and is rescued by N-acetylmannosamine | Q28305085 | ||
Involvement of lipid rafts in nephrin phosphorylation and organization of the glomerular slit diaphragm | Q28350870 | ||
FSGS-associated alpha-actinin-4 (K256E) impairs cytoskeletal dynamics in podocytes | Q28513677 | ||
Nephrin and Neph1 co-localize at the podocyte foot process intercellular junction and form cis hetero-oligomers | Q28577451 | ||
Neph1 cooperates with nephrin to transduce a signal that induces actin polymerization | Q28594662 | ||
TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function | Q28594959 | ||
CD2AP mutations are associated with sporadic nephrotic syndrome and focal segmental glomerulosclerosis (FSGS). | Q30157406 | ||
A novel TRPC6 mutation that causes childhood FSGS. | Q30913201 | ||
Twenty-one-year trend in ESRD due to focal segmental glomerulosclerosis in the United States | Q31121770 | ||
Trends in the epidemiology of focal segmental glomerulosclerosis | Q31139397 | ||
Mycophenolate mofetil inhibits rat and human mesangial cell proliferation by guanosine depletion. | Q33177527 | ||
A randomized trial of cyclosporine in patients with steroid-resistant focal segmental glomerulosclerosis. North America Nephrotic Syndrome Study Group. | Q33179291 | ||
Glucocorticoids protect and enhance recovery of cultured murine podocytes via actin filament stabilization | Q40346220 | ||
Mycophenolic acid and brequinar, inhibitors of purine and pyrimidine synthesis, block the glycosylation of adhesion molecules | Q40919952 | ||
Treatment of the idiopathic nephrotic syndrome: regimens and outcomes in children and adults | Q41492359 | ||
Pathogenesis of lipoid nephrosis: a disorder of T-cell function | Q41509804 | ||
Regulation of podocyte structure during the development of nephrotic syndrome | Q41735840 | ||
Mutations in the Wilms' tumor 1 gene cause isolated steroid resistant nephrotic syndrome and occur in exons 8 and 9. | Q42678945 | ||
Induction of T regulatory cells attenuates idiopathic nephrotic syndrome | Q43210229 | ||
Clinical and epidemiological assessment of steroid-resistant nephrotic syndrome associated with the NPHS2 R229Q variant | Q43501079 | ||
Sieve plugs in fenestrae of glomerular capillaries--site of the filtration barrier? | Q43814600 | ||
Novel therapy of focal glomerulosclerosis with mycophenolate and angiotensin blockade | Q44481330 | ||
Patients with mutations in NPHS2 (podocin) do not respond to standard steroid treatment of nephrotic syndrome. | Q44772814 | ||
FSGS permeability factor-associated nephrotic syndrome: remission after oral galactose therapy | Q45757311 | ||
Single-centre experience with cyclosporin in 106 children with idiopathic focal segmental glomerulosclerosis | Q46384319 | ||
Galactose binds to focal segmental glomerulosclerosis permeability factor and inhibits its activity | Q46566229 | ||
2007 Young Investigator Award: TRP'ing into a new era for glomerular disease | Q48323086 | ||
Presenting features and short-term outcome according to pathologic variant in childhood primary focal segmental glomerulosclerosis. | Q51741626 | ||
Bone marrow-derived cells contribute to podocyte regeneration and amelioration of renal disease in a mouse model of Alport syndrome. | Q51793488 | ||
Recurrence of nephrotic syndrome after transplantation in a mixed population of children and adults: course of glomerular lesions and value of the Columbia classification of histological variants of focal and segmental glomerulosclerosis (FSGS). | Q51808864 | ||
Pathology of the nephrotic syndrome in children: a report for the International Study of Kidney Disease in Children. | Q53452950 | ||
Podocyte depletion causes glomerulosclerosis: diphtheria toxin-induced podocyte depletion in rats expressing human diphtheria toxin receptor transgene. | Q53661064 | ||
Effects of cyclosporin A on glomerular barrier function in the nephrotic syndrome | Q54269913 | ||
Recessive missense mutations in LAMB2 expand the clinical spectrum of LAMB2-associated disorders | Q55671186 | ||
Pathologic classification of focal segmental glomerulosclerosis: a working proposal | Q56008651 | ||
Mutational analysis of the PLCE1 gene in steroid resistant nephrotic syndrome | Q57203717 | ||
VEGF-C and VEGF-D expression in neuroendocrine cells and their receptor, VEGFR-3, in fenestrated blood vessels in human tissues | Q57382249 | ||
Genetics, clinical and pathological features of glomerulonephrites associated with mutations of nonmuscle myosin IIA (Fechtner syndrome) | Q57589515 | ||
Mutations in PLCE1 are a major cause of isolated diffuse mesangial sclerosis (IDMS) | Q57740824 | ||
Mycophenolate mofetil in the treatment of focal segmental glomerulosclerosis. | Q33210575 | ||
Cyclosporin in idiopathic glomerular disease associated with the nephrotic syndrome : workshop recommendations | Q33300551 | ||
Genotype-phenotype correlation in MYH9-related thrombocytopenia | Q33367809 | ||
Follow-up of phase I trial of adalimumab and rosiglitazone in FSGS: III. Report of the FONT study group | Q33527568 | ||
Phase 1 trial of adalimumab in Focal Segmental Glomerulosclerosis (FSGS): II. Report of the FONT (Novel Therapies for Resistant FSGS) study group | Q33584368 | ||
The actin cytoskeleton of kidney podocytes is a direct target of the antiproteinuric effect of cyclosporine A. | Q33943247 | ||
The association of the MYH9 gene and kidney outcomes in American Indians: the Strong Heart Family Study | Q34092120 | ||
Mutant chromatin remodeling protein SMARCAL1 causes Schimke immuno-osseous dysplasia. | Q34110155 | ||
The TRP ion channel family. | Q34271602 | ||
A mutation in the TRPC6 cation channel causes familial focal segmental glomerulosclerosis | Q34416768 | ||
How do mesangial and endothelial cells form the glomerular tuft? | Q34733658 | ||
Focal segmental glomerulosclerosis in adults. | Q35210693 | ||
CD2-associated protein and glomerular disease | Q35595375 | ||
PLC-epsilon: a shared effector protein in Ras-, Rho-, and G alpha beta gamma-mediated signaling | Q35679578 | ||
Lipid raft domains and protein networks in T-cell receptor signal transduction | Q35768915 | ||
Urinary excretion of viable podocytes in health and renal disease | Q36012253 | ||
Early glomerular filtration defect and severe renal disease in podocin-deficient mice | Q36222666 | ||
Clinical impact of research on the podocyte slit diaphragm | Q36577165 | ||
The structure and function of the endothelial glycocalyx layer | Q36764891 | ||
The spectrum of podocytopathies: a unifying view of glomerular diseases. | Q36780741 | ||
Mycophenolic acid: a new approach to the therapy of experimental mesangial proliferative glomerulonephritis. | Q36899338 | ||
Resolved: capillary endothelium is a major contributor to the glomerular filtration barrier. | Q36921162 | ||
Phase I trial of rosiglitazone in FSGS: I. Report of the FONT Study Group | Q37042128 | ||
Recruitment of podocytes from glomerular parietal epithelial cells | Q37086632 | ||
Properties of the glomerular barrier and mechanisms of proteinuria | Q37129340 | ||
Urine podocyte mRNAs mark progression of renal disease | Q37182003 | ||
Therapeutic targets in focal and segmental glomerulosclerosis | Q37228164 | ||
Rituximab treatment of adult patients with steroid-resistant focal segmental glomerulosclerosis | Q37297636 | ||
Monoclonal antibodies for podocytopathies: rationale and clinical responses | Q37498290 | ||
New insights into the role of podocytes in proteinuria | Q37542923 | ||
Is there such a thing as a renal stem cell? | Q37588328 | ||
The structural basis of glomerular filtration | Q39882777 | ||
Identification and functional analysis of a novel TRPC6 mutation associated with late onset familial focal segmental glomerulosclerosis in Chinese patients | Q39898583 | ||
Direct effects of dexamethasone on human podocytes | Q40256235 | ||
P433 | issue | 7 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | pathogenesis | Q372016 |
focal segmental glomerulosclerosis | Q1435223 | ||
P304 | page(s) | 1001-1015 | |
P577 | publication date | 2010-11-26 | |
P1433 | published in | Pediatric Nephrology | Q15749796 |
P1476 | title | Pathogenesis and therapy of focal segmental glomerulosclerosis: an update | |
P478 | volume | 26 |
Q51695170 | (1) H NMR-based metabolomics exploring urinary biomarkers correlated with proteinuria in focal segmental glomerulosclerosis: a pilot study. |
Q41778140 | A case of the "cellular variant" of focal segmental glomerulosclerosis with acute renal failure that remitted completely with oral steroid administration |
Q35379384 | A circulating antibody panel for pretransplant prediction of FSGS recurrence after kidney transplantation |
Q37704986 | A multicolor podocyte reporter highlights heterogeneous podocyte changes in focal segmental glomerulosclerosis |
Q53813439 | Analysis of the genes responsible for steroid-resistant nephrotic syndrome and/or focal segmental glomerulosclerosis in Japanese patients by whole-exome sequencing analysis. |
Q35168576 | BDNF repairs podocyte damage by microRNA-mediated increase of actin polymerization |
Q59329757 | Bone marrow mesenchymal stem cells attenuate the progression of focal segmental glomerulosclerosis in rat models |
Q42698311 | Clinical and genetic heterogeneity in familial steroid-sensitive nephrotic syndrome |
Q36131458 | Clinical and histopathological features resembling those of human focal segmental glomerulosclerosis in a cat with nonimmune-mediated glomerulonephropathy. |
Q37297472 | Distinct metabolic profile of primary focal segmental glomerulosclerosis revealed by NMR-based metabolomics |
Q46896504 | Dysfunction of the PGC-1α-mitochondria axis confers adriamycin-induced podocyte injury |
Q34730562 | Dysregulated autophagy contributes to podocyte damage in Fabry's disease |
Q55400170 | Effect of human very low-density lipoproteins on cardiotrophin-like cytokine factor 1 (CLCF1) activity. |
Q38938623 | Focal Segmental Glomerulosclerosis |
Q87592423 | Glomerular nestin expression: possible predictor of outcome of focal segmental glomerulosclerosis in children |
Q38094055 | Is a legacy effect possible in IgA nephropathy? |
Q38840895 | Lessons from genetics: is it time to revise the therapeutic approach to children with steroid-resistant nephrotic syndrome? |
Q38113310 | Mycophenolate mofetil for primary focal segmental glomerulosclerosis: systematic review |
Q84242626 | Nephrotic syndrome |
Q42096387 | Nonfunction of the ECT2 gene may cause renal tubulointerstitial injury leading to focal segmental glomerulosclerosis |
Q38738516 | Permeability factors in nephrotic syndrome and focal segmental glomerulosclerosis |
Q47215250 | Plasma and urine proteomic profiles in childhood idiopathic nephrotic syndrome |
Q56342197 | Recurrence of nephrotic syndrome following kidney transplantation is associated with initial native kidney biopsy findings |
Q34455143 | Relationship between serum soluble urokinase plasminogen activator receptor level and steroid responsiveness in FSGS |
Q53675239 | Rituximab treatment for adults with refractory nephrotic syndrome: a single-center experience and review of the literature. |
Q47147627 | Rituximab treatment in adults with refractory minimal change disease or focal segmental glomerulosclerosis |
Q35611793 | Secondary focal and segmental glomerulosclerosis associated with single-nucleotide polymorphisms in the genes encoding complement factor H and C3. |
Q59791112 | Serum Metabolic Profiling in a Mouse Model of Adriamycin-Induced Focal Segmental Glomerulosclerosis |
Q49469264 | Steroid-resistant nephrotic syndrome caused by co-inheritance of mutations at NPHS1 and ADCK4 genes in two Chinese siblings |
Q92991647 | The Role of Glucocorticoid Receptors in Podocytes and Nephrotic Syndrome |
Q47611276 | The natural compound 2,3,5,4'-tetrahydroxystilbene-2-O-β-d glucoside protects against adriamycin-induced nephropathy through activating the Nrf2-Keap1 antioxidant pathway |
Q34182007 | The population genetics of chronic kidney disease: insights from the MYH9-APOL1 locus |
Q28088653 | Translating genetic findings in hereditary nephrotic syndrome: the missing loops |
Q36811343 | Treatment of Focal Segmental Glomerulosclerosis Recurrence in the Renal Allograft: A Report of Two Cases |
Q35869275 | Trends in the histopathology of childhood nephrotic syndrome in Ibadan Nigeria: preponderance of idiopathic focal segmental glomerulosclerosis |
Q34630984 | Two new families with hereditary minimal change disease. |
Q44401470 | Unique X-linked familial FSGS with co-segregating heart block disorder is associated with a mutation in the NXF5 gene. |
Q26751470 | Update on the treatment of focal segmental glomerulosclerosis in renal transplantation |
Q37182778 | Urinary IgG and α2-macroglobulin are powerful predictors of outcome and responsiveness to steroids and cyclophosphamide in idiopathic focal segmental glomerulosclerosis with nephrotic syndrome |
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