| P50 | author | Jennifer Luebke | Q56524312 |
| | Amy M Pooler | Q74787777 |
| | Tara L Spires-Jones | Q42586954 |
| P2093 | author name string | Manuela Polydoro | |
| | Johanna L Crimins | |
| P2860 | cites work | Amyloid plaque core protein in Alzheimer disease and Down syndrome | Q24568384 |
| | S-Nitrosylation activates Cdk5 and contributes to synaptic spine loss induced by beta-amyloid peptide | Q24612303 |
| | Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory | Q24645499 |
| | Soluble oligomers of the amyloid beta-protein impair synaptic plasticity and behavior | Q24645994 |
| | Soluble oligomers of amyloid Beta protein facilitate hippocampal long-term depression by disrupting neuronal glutamate uptake | Q24656613 |
| | Synapses and Alzheimer's disease | Q26269868 |
| | Spines, plasticity, and cognition in Alzheimer's model mice | Q27691328 |
| | Synapse loss and microglial activation precede tangles in a P301S tauopathy mouse model | Q28115206 |
| | Tau accumulation causes mitochondrial distribution deficits in neurons in a mouse model of tauopathy and in human Alzheimer's disease brain | Q28116593 |
| | Dendritic function of tau mediates amyloid-beta toxicity in Alzheimer's disease mouse models | Q28119018 |
| | Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo | Q28131779 |
| | Alzheimer's disease: genes, proteins, and therapy | Q28131806 |
| | Staging of neurofibrillary degeneration caused by human tau overexpression in a unique cellular model of human tauopathy | Q28345670 |
| | Regulation of NMDA receptor trafficking by amyloid-beta | Q28506085 |
| | Trafficking and surface expression of hyperpolarization-activated cyclic nucleotide-gated channels in hippocampal neurons | Q28572166 |
| | Differential regulation of dynein and kinesin motor proteins by tau | Q28589618 |
| | Natural oligomers of the Alzheimer amyloid-beta protein induce reversible synapse loss by modulating an NMDA-type glutamate receptor-dependent signaling pathway | Q28854318 |
| | Physical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment | Q29547294 |
| | Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins | Q29547593 |
| | Alzheimer's disease is a synaptic failure | Q29547670 |
| | Alzheimer's disease: Initial report of the purification and characterization of a novel cerebrovascular amyloid protein | Q29547803 |
| | AMPAR removal underlies Abeta-induced synaptic depression and dendritic spine loss | Q29614850 |
| | Natural oligomers of the amyloid-beta protein specifically disrupt cognitive function | Q29614851 |
| | The amyloid cascade hypothesis for Alzheimer's disease: an appraisal for the development of therapeutics | Q29614972 |
| | Tau suppression in a neurodegenerative mouse model improves memory function | Q29615831 |
| | Reducing endogenous tau ameliorates amyloid beta-induced deficits in an Alzheimer's disease mouse model | Q29617937 |
| | Tau-mediated neurodegeneration in Alzheimer's disease and related disorders | Q29618148 |
| | The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptor | Q29618322 |
| | Alzheimer's disease-like tau neuropathology leads to memory deficits and loss of functional synapses in a novel mutated tau transgenic mouse without any motor deficits | Q30478477 |
| | Early axonopathy preceding neurofibrillary tangles in mutant tau transgenic mice | Q30480000 |
| | Oligomeric amyloid beta associates with postsynaptic densities and correlates with excitatory synapse loss near senile plaques | Q30486499 |
| | Age-dependent impairment of cognitive and synaptic function in the htau mouse model of tau pathology | Q30490880 |
| | Soluble Aβ oligomers inhibit long-term potentiation through a mechanism involving excessive activation of extrasynaptic NR2B-containing NMDA receptors | Q42738430 |
| | Prominent axonopathy in the brain and spinal cord of transgenic mice overexpressing four-repeat human tau protein | Q42795195 |
| | Aβ Oligomers Cause Localized Ca2+Elevation, Missorting of Endogenous Tau into Dendrites, Tau Phosphorylation, and Destruction of Microtubules and Spines | Q42913207 |
| | Abeta plaques lead to aberrant regulation of calcium homeostasis in vivo resulting in structural and functional disruption of neuronal networks | Q43246347 |
| | Early selective vulnerability of synapses and synaptic mitochondria in the hippocampal CA1 region of the Tg2576 mouse model of Alzheimer's disease | Q43440335 |
| | Homeostatic responses by surviving cortical pyramidal cells in neurodegenerative tauopathy | Q44199130 |
| | Early Abeta accumulation and progressive synaptic loss, gliosis, and tangle formation in AD brain | Q44810166 |
| | Dendrite and dendritic spine alterations in Alzheimer models | Q45099584 |
| | Soluble beta-amyloid1-40 induces NMDA-dependent degradation of postsynaptic density-95 at glutamatergic synapses. | Q46828851 |
| | Dendritic spine loss in the hippocampus of young PDAPP and Tg2576 mice and its prevention by the ApoE2 genotype | Q47711160 |
| | Ultrastructural neuronal pathology in transgenic mice expressing mutant (P301L) human tau. | Q47878702 |
| | Dendritic hyperpolarization-activated currents modify the integrative properties of hippocampal CA1 pyramidal neurons. | Q48393633 |
| | Improved long-term potentiation and memory in young tau-P301L transgenic mice before onset of hyperphosphorylation and tauopathy. | Q48600800 |
| | Alzheimer's disease: transiently developing dendritic changes in pyramidal cells of sector CA1 of the Ammon's horn | Q48744714 |
| | Linking amyloid-β and tau: amyloid-β induced synaptic dysfunction via local wreckage of the neuronal cytoskeleton | Q48788749 |
| | Caspase-3 triggers early synaptic dysfunction in a mouse model of Alzheimer's disease | Q48948068 |
| | Synapse loss in frontal cortex biopsies in Alzheimer's disease: correlation with cognitive severity | Q48983820 |
| | Tau-induced defects in synaptic plasticity, learning, and memory are reversible in transgenic mice after switching off the toxic Tau mutant. | Q51019667 |
| | Fibrillar amyloid deposition leads to local synaptic abnormalities and breakage of neuronal branches. | Q52560574 |
| | Soluble oligomers of beta amyloid (1-42) inhibit long-term potentiation but not long-term depression in rat dentate gyrus. | Q53245545 |
| | Age-Dependent Emergence and Progression of a Tauopathy in Transgenic Mice Overexpressing the Shortest Human Tau Isoform | Q54965151 |
| | Neurofibrillary tangle-related synaptic alterations of spinal motor neurons of P301L tau transgenic mice | Q57306382 |
| | The beta-propensity of Tau determines aggregation and synaptic loss in inducible mouse models of tauopathy | Q60603646 |
| | Axonopathy and amyotrophy in mice transgenic for human four-repeat tau protein | Q73774228 |
| | Amyloid-β/Fyn-induced synaptic, network, and cognitive impairments depend on tau levels in multiple mouse models of Alzheimer's disease. | Q30513293 |
| | Electrophysiological changes precede morphological changes to frontal cortical pyramidal neurons in the rTg4510 mouse model of progressive tauopathy | Q30528756 |
| | Synaptic targeting by Alzheimer's-related amyloid beta oligomers. | Q33288786 |
| | Dendritic vulnerability in neurodegenerative disease: insights from analyses of cortical pyramidal neurons in transgenic mouse models | Q33533576 |
| | Amyloid beta induces the morphological neurodegenerative triad of spine loss, dendritic simplification, and neuritic dystrophies through calcineurin activation | Q33740340 |
| | Structural and functional changes in tau mutant mice neurons are not linked to the presence of NFTs | Q33829000 |
| | Polarized and compartment-dependent distribution of HCN1 in pyramidal cell dendrites. | Q34155306 |
| | Tau mislocalization to dendritic spines mediates synaptic dysfunction independently of neurodegeneration | Q34518577 |
| | Soluble amyloid beta-protein dimers isolated from Alzheimer cortex directly induce Tau hyperphosphorylation and neuritic degeneration | Q34804911 |
| | Thirty years of Alzheimer's disease genetics: the implications of systematic meta-analyses | Q34831241 |
| | Dendritic degeneration, neurovascular defects, and inflammation precede neuronal loss in a mouse model for tau-mediated neurodegeneration | Q35235749 |
| | Tau oligomers impair memory and induce synaptic and mitochondrial dysfunction in wild-type mice. | Q35574384 |
| | Synaptic slaughter in Alzheimer's disease | Q35595427 |
| | Abnormal phosphorylation of tau and the mechanism of Alzheimer neurofibrillary degeneration: sequestration of microtubule-associated proteins 1 and 2 and the disassembly of microtubules by the abnormal tau | Q35911885 |
| | Apolipoprotein E4 effects in Alzheimer's disease are mediated by synaptotoxic oligomeric amyloid-β | Q36054191 |
| | Soluble forms of tau are toxic in Alzheimer's disease | Q36286360 |
| | The synaptic accumulation of hyperphosphorylated tau oligomers in Alzheimer disease is associated with dysfunction of the ubiquitin-proteasome system | Q36295481 |
| | A century of Alzheimer's disease | Q36643414 |
| | Synaptic alterations in the rTg4510 mouse model of tauopathy | Q37051901 |
| | Balancing structure and function at hippocampal dendritic spines | Q37088448 |
| | Structural abnormalities in the cortex of the rTg4510 mouse model of tauopathy: a light and electron microscopy study | Q37107652 |
| | Cytosolic abnormally hyperphosphorylated tau but not paired helical filaments sequester normal MAPs and inhibit microtubule assembly | Q37238256 |
| | Phosphorylated Tau interacts with c-Jun N-terminal kinase-interacting protein 1 (JIP1) in Alzheimer disease | Q37344062 |
| | Synaptic degeneration in Alzheimer's disease | Q37458270 |
| | Amyloid-β and tau--a toxic pas de deux in Alzheimer's disease | Q37824813 |
| | Alzheimer's disease: synapses gone cold | Q37922808 |
| | Structural plasticity upon learning: regulation and functions. | Q38020090 |
| | From mitochondrial dysfunction to amyloid beta formation: novel insights into the pathogenesis of Alzheimer's disease. | Q38029575 |
| | Tau inhibits anterograde axonal transport and perturbs stability in growing axonal neurites in part by displacing kinesin cargo: neurofilaments attenuate tau-mediated neurite instability | Q40052235 |
| | Missorting of tau in neurons causes degeneration of synapses that can be rescued by the kinase MARK2/Par-1. | Q40158901 |
| | Accumulation of vesicle-associated human tau in distal dendrites drives degeneration and tau secretion in an in situ cellular tauopathy model | Q40796484 |
| | Tau blocks traffic of organelles, neurofilaments, and APP vesicles in neurons and enhances oxidative stress | Q41830251 |
| | Soluble tau species, not neurofibrillary aggregates, disrupt neural system integration in a tau transgenic model | Q41868802 |
| | Dendritic spine abnormalities in amyloid precursor protein transgenic mice demonstrated by gene transfer and intravital multiphoton microscopy | Q42087146 |
| | Microtubular reorganization and dendritic growth response in Alzheimer's disease | Q42500791 |
| | Calcineurin inhibition with FK506 ameliorates dendritic spine density deficits in plaque-bearing Alzheimer model mice | Q42586885 |
| P433 | issue | 3 | |
| P921 | main subject | Alzheimer's disease | Q11081 |
| P304 | page(s) | 757-763 | |
| P577 | publication date | 2013-03-22 | |
| P1433 | published in | Ageing Research Reviews | Q762802 |
| P1476 | title | The intersection of amyloid β and tau in glutamatergic synaptic dysfunction and collapse in Alzheimer's disease | |
| P478 | volume | 12 | |