review article | Q7318358 |
scholarly article | Q13442814 |
P356 | DOI | 10.1097/MIB.0000000000000193 |
P698 | PubMed publication ID | 25248001 |
P50 | author | Libor Vitek | Q42714402 |
P2093 | author name string | Libor Vítek | |
P2860 | cites work | Vitamin D receptor as an intestinal bile acid sensor | Q24297643 |
Definition of a novel growth factor-dependent signal cascade for the suppression of bile acid biosynthesis | Q24305561 | ||
Primary bile acid malabsorption caused by mutations in the ileal sodium-dependent bile acid transporter gene (SLC10A2) | Q24314316 | ||
A G protein-coupled receptor responsive to bile acids | Q24338558 | ||
Bile acid malabsorption in Crohn's disease and indications for its assessment using SeHCAT | Q24538029 | ||
Colonic secretion of water and electrolytes induced by bile acids: perfusion studies in man | Q24561896 | ||
Methods for diagnosis of bile acid malabsorption in clinical practice | Q24618216 | ||
Bile acids: regulation of synthesis | Q24658139 | ||
New pathophysiological concepts underlying pathogenesis of pigment gallstones | Q26826934 | ||
Pregnane X receptor as a target for treatment of inflammatory bowel disorders | Q26998347 | ||
Farnesoid X receptor activation inhibits inflammation and preserves the intestinal barrier in inflammatory bowel disease | Q28116241 | ||
The bile acid receptor FXR is a modulator of intestinal innate immunity | Q28116925 | ||
Gastrointestinal effects of low-digestible carbohydrates | Q28235996 | ||
Farnesoid X receptor agonists attenuate colonic epithelial secretory function and prevent experimental diarrhoea in vivo | Q28296042 | ||
Regulation of antibacterial defense in the small intestine by the nuclear bile acid receptor | Q28508358 | ||
The bile acid sensor FXR is required for immune-regulatory activities of TLR-9 in intestinal inflammation | Q28585275 | ||
The bile acid receptor GPBAR-1 (TGR5) modulates integrity of intestinal barrier and immune response to experimental colitis | Q28590901 | ||
Changes in expression and distribution of claudin 2, 5 and 8 lead to discontinuous tight junctions and barrier dysfunction in active Crohn's disease | Q33249005 | ||
Bile acids modulate tight junction structure and barrier function of Caco-2 monolayers via EGFR activation. | Q33317737 | ||
Farnesoid X receptor (FXR) activation and FXR genetic variation in inflammatory bowel disease | Q34009566 | ||
Cytoskeletal regulation of epithelial barrier function during inflammation | Q34033538 | ||
Fibroblast growth factor 19 in patients with bile acid diarrhoea: a prospective comparison of FGF19 serum assay and SeHCAT retention | Q34366988 | ||
Systematic review: the management of chronic diarrhoea due to bile acid malabsorption. | Q34408666 | ||
Advances in understanding of bile acid diarrhea | Q34412381 | ||
Pharmacological activation of the bile acid nuclear farnesoid X receptor is feasible in patients with quiescent Crohn's colitis. | Q34493836 | ||
Orocecal transit time and bacterial overgrowth in patients with Crohn's disease. | Q34509907 | ||
Effect of jejunal infusion of bile acids on small bowel transit and fasting jejunal motility in man. | Q34530410 | ||
A new mechanism for bile acid diarrhea: defective feedback inhibition of bile acid biosynthesis | Q34979989 | ||
Enterohepatic cycling of bilirubin as a cause of 'black' pigment gallstones in adult life. | Q35202140 | ||
New molecular insights into inflammatory bowel disease-induced diarrhea | Q35582973 | ||
Human ileal bile acid transporter gene ASBT (SLC10A2) is transactivated by the glucocorticoid receptor | Q35596270 | ||
TGR5 signalling inhibits the production of pro-inflammatory cytokines by in vitro differentiated inflammatory and intestinal macrophages in Crohn's disease | Q36790557 | ||
Induced and natural regulatory T cells in the development of inflammatory bowel disease | Q36948341 | ||
Bile acids: chemistry, pathochemistry, biology, pathobiology, and therapeutics | Q37167278 | ||
Vitamin D regulates the gut microbiome and protects mice from dextran sodium sulfate-induced colitis | Q37167679 | ||
Serum bile acid profiling reflects enterohepatic detoxification state and intestinal barrier function in inflammatory bowel disease | Q37250362 | ||
Chenodeoxycholic acid stimulates Cl(-) secretion via cAMP signaling and increases cystic fibrosis transmembrane conductance regulator phosphorylation in T84 cells | Q37476428 | ||
Mechanisms of diarrhea in inflammatory bowel diseases | Q37524292 | ||
Bile acids as regulators of hepatic lipid and glucose metabolism | Q37749352 | ||
New insights into bile acid malabsorption. | Q37909078 | ||
Transport and biological activities of bile acids | Q38100563 | ||
Recycling rate of bile acids in the enterohepatic recirculation as a major determinant of whole body 75SeHCAT retention. | Q38112366 | ||
The bile acid membrane receptor TGR5: a novel pharmacological target in metabolic, inflammatory and neoplastic disorders. | Q38115636 | ||
The bile acid TGR5 membrane receptor: from basic research to clinical application | Q38177434 | ||
Luminal regulation of normal and neoplastic human EC cell serotonin release is mediated by bile salts, amines, tastants, and olfactants | Q39971991 | ||
Small intestinal bacterial overgrowth and orocecal transit time in patients of inflammatory bowel disease | Q40248633 | ||
Unconjugated bilirubin modulates the intestinal epithelial barrier function in a human-derived in vitro model. | Q40280858 | ||
TNF-alpha down-regulates the Na+-K+ ATPase and the Na+-K+-2Cl-cotransporter in the rat colon via PGE2. | Q40413555 | ||
Fibroblast Growth Factor 19 and 7α-Hydroxy-4-Cholesten-3-one in the Diagnosis of Patients With Possible Bile Acid Diarrhea. | Q42263341 | ||
The receptor TGR5 mediates the prokinetic actions of intestinal bile acids and is required for normal defecation in mice | Q42715520 | ||
Bile salt inhibition of motility in the isolated perfused rabbit terminal ileum | Q43213016 | ||
Bile acid malabsorption assessed by 7 alpha-hydroxy-4-cholesten-3-one in pediatric inflammatory bowel disease: correlation to clinical and laboratory findings | Q43745060 | ||
Assessment of orocaecal transit time in different localization of Crohn's disease and its possible influence on clinical response to therapy | Q44289270 | ||
Oral bile acids reduce bacterial overgrowth, bacterial translocation, and endotoxemia in cirrhotic rats | Q44324969 | ||
The peristaltic reflex induced by short-chain fatty acids is mediated by sequential release of 5-HT and neuronal CGRP but not BDNF. | Q44923280 | ||
Epistasis between Toll-like receptor-9 polymorphisms and variants in NOD2 and IL23R modulates susceptibility to Crohn's disease | Q45758087 | ||
A national survey of the prevalence and impact of Clostridium difficile infection among hospitalized inflammatory bowel disease patients | Q46044713 | ||
Improved HPLC analysis of serum 7alpha-hydroxycholest-4-en-3-one, a marker of bile acid malabsorption | Q46570526 | ||
Rifaximin is a gut-specific human pregnane X receptor activator | Q48605501 | ||
Bile acid malabsorption deactivates pregnane X receptor in patients with Crohn's disease. | Q51076088 | ||
Bile acid malabsorption in inflammatory bowel disease: assessment by serum markers. | Q51639717 | ||
Altered ENaC expression leads to impaired sodium absorption in the noninflamed intestine in Crohn's disease. | Q52919538 | ||
Pregnane X receptor (PXR/NR1I2) gene haplotypes modulate susceptibility to inflammatory bowel disease. | Q54354711 | ||
Ileal bile acid malabsorption in colonic Crohn's disease. | Q54641638 | ||
P433 | issue | 2 | |
P921 | main subject | inflammatory bowel diseases | Q917447 |
P304 | page(s) | 476-483 | |
P577 | publication date | 2015-02-01 | |
P1433 | published in | Inflammatory Bowel Diseases | Q15749161 |
P1476 | title | Bile acid malabsorption in inflammatory bowel disease | |
P478 | volume | 21 |
Q60932297 | A distinct epigenetic profile distinguishes stenotic from non-inflamed fibroblasts in the ileal mucosa of Crohn's disease patients |
Q90448558 | A secondary bile acid from microbiota metabolism attenuates ileitis and bile acid reduction in subclinical necrotic enteritis in chickens |
Q26774563 | Advances in nutritional therapy in inflammatory bowel diseases: Review |
Q88434179 | Alterations in Lipid, Amino Acid, and Energy Metabolism Distinguish Crohn's Disease from Ulcerative Colitis and Control Subjects by Serum Metabolomic Profiling |
Q93035767 | Cholestyramine decreases apparent total tract macronutrient digestibility and alters fecal characteristics and metabolites of healthy adult dogs |
Q57135941 | Clostridium difficile Infection in Patients with Inflammatory Bowel Disease |
Q57471722 | Comparison of intestinal expression of the apical sodium-dependent bile acid transporter between dogs with and without chronic inflammatory enteropathy |
Q93184524 | Gastrointestinal motility and absorptive disorders in patients with inflammatory bowel diseases: Prevalence, diagnosis and treatment |
Q38739354 | How to Diagnose and Treat IBD Mimics in the Refractory IBD Patient Who Does Not Have IBD. |
Q92383004 | Multi-omics of the gut microbial ecosystem in inflammatory bowel diseases |
Q55345479 | Obeticholic acid for severe bile acid diarrhea with intestinal failure: A case report and review of the literature. |
Q38551892 | Pathogenesis of Clostridium difficile Infection and Its Potential Role in Inflammatory Bowel Disease. |
Q36987335 | Pharmacology of bile acid receptors: Evolution of bile acids from simple detergents to complex signaling molecules |
Q92677838 | Physiology of Electrolyte Transport in the Gut: Implications for Disease |
Q42362727 | Repression of intestinal transporters and FXR-FGF15 signaling explains bile acids dysregulation in experimental colitis-associated colon cancer. |
Q54943140 | Role of bile acids in inflammatory bowel disease. |
Q38438128 | Symptom management in inflammatory bowel disease |
Q33740929 | The Yin and Yang of bile acid action on tight junctions in a model colonic epithelium |
Q26770807 | The role of bile acids in metabolic regulation |
Q40622519 | Vitamin D deficiency in patients with inflammatory bowel disease |
Search more.