Lysophosphatidylcholine reduces the organ injury and dysfunction in rodent models of gram-negative and gram-positive shock

scientific article published on 5 June 2006

Lysophosphatidylcholine reduces the organ injury and dysfunction in rodent models of gram-negative and gram-positive shock is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1038/SJ.BJP.0706788
P932PMC publication ID1617069
P698PubMed publication ID16751791
P5875ResearchGate publication ID7030775

P50authorHelder Mota-FilipeQ43121445
Christoph ThiemermannQ82195682
Marika CollinQ82206141
P2093author name stringSimon J Foster
Oliver Murch
Bruno Sepodes
P2860cites workExpression of G2A, a receptor for lysophosphatidylcholine, by macrophages in murine, rabbit, and human atherosclerotic plaquesQ28217576
Therapeutic effects of lysophosphatidylcholine in experimental sepsisQ28238163
Lysophosphatidylcholine enhances glucose-dependent insulin secretion via an orphan G-protein-coupled receptorQ28509942
The epidemiology of sepsis in the United States from 1979 through 2000Q29547760
Carboxy-PTIO, a scavenger of nitric oxide, selectively inhibits the increase in medullary perfusion and improves renal function in endotoxemiaQ32147377
The cell wall components peptidoglycan and lipoteichoic acid from Staphylococcus aureus act in synergy to cause shock and multiple organ failureQ33838051
RETRACTED: Lysophosphatidylcholine as a ligand for the immunoregulatory receptor G2A.Q34085160
Inhibition of endogenous hydrogen sulfide formation reduces the organ injury caused by endotoxemiaQ35545028
Lysophosphatidylcholine, a component of atherogenic lipoproteins, induces mononuclear leukocyte adhesion molecules in cultured human and rabbit arterial endothelial cellsQ35822759
Analysis of the autolysins of Bacillus subtilis 168 during vegetative growth and differentiation by using renaturing polyacrylamide gel electrophoresisQ39933090
Mechanism of gram-positive shock: identification of peptidoglycan and lipoteichoic acid moieties essential in the induction of nitric oxide synthase, shock, and multiple organ failureQ41024121
Transcriptional regulation of endothelial nitric-oxide synthase by lysophosphatidylcholineQ42456169
Effects of perfusion pressure on tissue perfusion in septic shockQ44182473
Specific phospholipid oxidation products inhibit ligand activation of toll-like receptors 4 and 2.Q44459170
Reconstituted high-density lipoprotein attenuates organ injury and adhesion molecule expression in a rodent model of endotoxic shockQ44661051
The action mode of lysophosphatidylcholine in human monocytesQ44743508
Peptidoglycan of Staphylococcus aureus causes inflammation and organ injury in the rat.Q44752818
Suppression of HMGB1 release by stearoyl lysophosphatidylcholine:an additional mechanism for its therapeutic effects in experimental sepsisQ45248637
Increasing mean arterial pressure in patients with septic shock: effects on oxygen variables and renal functionQ46428102
Efficient drug delivery to atherosclerotic lesions and the antiatherosclerotic effect by dexamethasone incorporated into liposomes in atherogenic miceQ46624409
GSK-3beta inhibitors attenuate the organ injury/dysfunction caused by endotoxemia in the rat.Q46691574
Selective inhibition of the activity of inducible nitric oxide synthase prevents the circulatory failure, but not the organ injury/dysfunction, caused by endotoxinQ48001412
Plasma ceramide and lysophosphatidylcholine inversely correlate with mortality in sepsis patientsQ57375076
Transcriptional induction of endothelial nitric oxide synthase type III by lysophosphatidylcholineQ71702928
Lysophosphatidylcholine in oxidized low-density lipoprotein increases endothelial susceptibility to polymorphonuclear leukocyte-induced endothelial dysfunction in porcine coronary arteries. Role of protein kinase CQ72314416
Tissue factor expression of human monocytes is suppressed by lysophosphatidylcholineQ77795517
P433issue6
P407language of work or nameEnglishQ1860
P921main subjectGram-negative bacteriaQ632006
gram-positive bacteriaQ857288
P304page(s)769-777
P577publication date2006-06-05
P1433published inBritish Journal of PharmacologyQ919631
P1476titleLysophosphatidylcholine reduces the organ injury and dysfunction in rodent models of gram-negative and gram-positive shock
P478volume148

Reverse relations

cites work (P2860)
Q40117936Antimicrobial effects of lysophosphatidylcholine on methicillin-resistant Staphylococcus aureus
Q98471321Diagnosis and mortality prediction of sepsis via lysophosphatidylcholine 16:0 measured by MALDI-TOF MS
Q37340646Identification of lysophosphatidylcholine-chlorohydrin in human atherosclerotic lesions.
Q92264971Immune Response Resetting in Ongoing Sepsis
Q90293440Immunometabolism and Sepsis: A Role for HIF?
Q46442015Liver injury due to 3-amino-1-methyl-5h-pyrido [4,3-b] indole (Trp-P-2) and its prevention by miso
Q35061594Lysophosphatidylcholine triggers TLR2- and TLR4-mediated signaling pathways but counteracts LPS-induced NO synthesis in peritoneal macrophages by inhibiting NF-κB translocation and MAPK/ERK phosphorylation
Q90461389Lysophosphatidylcholines and Chlorophyll-Derived Molecules from the Diatom Cylindrotheca closterium with Anti-Inflammatory Activity
Q39993096Lysophospholipid metabolism facilitates Toll-like receptor 4 membrane translocation to regulate the inflammatory response.
Q84744195Monitoring mmp-9 gene expression in stromal cells using a novel transgenic mouse model
Q36924678Multivariate metabotyping of plasma predicts survival in patients with decompensated cirrhosis
Q36967194Product inhibition of secreted phospholipase A2 may explain lysophosphatidylcholines' unexpected therapeutic properties
Q34364867Recognition of lysophosphatidylcholine by type II NKT cells and protection from an inflammatory liver disease
Q28727959Response and recovery in the plasma metabolome tracks the acute LCMV-induced immune response
Q58200911Selective NOD1 Agonists Cause Shock and Organ Injury/DysfunctionIn Vivo
Q37689825Sepsis biomarkers: a review
Q49392673Stearoyl Lysophosphatidylcholine Inhibits Endotoxin-Induced Caspase-11 Activation
Q61808053The Systemic Alterations of Lipids, Alanine-Glucose Cycle and Inter-Organ Amino Acid Metabolism in Swine Model Confirms the Role of Liver in Early Phase of Septic Shock