| scholarly article | Q13442814 |
| retracted paper | Q45182324 |
| P2093 | author name string | Fang Ma | |
| Ning Wang | |||
| Li Han | |||
| Yubing Zhou | |||
| Yafeng Wang | |||
| Xiaojuan Guo | |||
| Jingmin Zhang | |||
| Jinhua Jiang | |||
| Qingduan Wang | |||
| P2860 | cites work | Multidrug resistance: can new drugs help chemotherapy score against cancer? | Q53940527 |
| COX-2 contributes to P-glycoprotein-mediated multidrug resistance via phosphorylation of c-Jun at Ser63/73 in colorectal cancer. | Q54389169 | ||
| [The effects of Cepharanthin on the recovery of hematopoietic stem cells after X-ray irradiation] | Q68183038 | ||
| [Correlation between reversing effect of cepharanthine hydrochloride on multidrug resistance and P-glycoprotein expression and function of K562/ADR cells] | Q84588118 | ||
| Reactive oxygen special (ROS), troublemakers between nuclear factor-kB (NF-kB) and c-Jun NH2-terminal kinase (JNK) | Q23924002 | ||
| Binocular diplopia and ptosis due to snakebite (Agkistrodon blomhoffi "mamushi")--a case report | Q28276656 | ||
| Activation of Bak in ultrasound-induced, JNK- and p38-independent apoptosis and its inhibition by Bcl-2 | Q30496914 | ||
| Gold nanoparticles inhibit VEGF165-induced migration and tube formation of endothelial cells via the Akt pathway | Q33759351 | ||
| In vitro and in vivo multidrug resistance reversal activity by a Betti-base derivative of tylosin | Q34005841 | ||
| Molecular mechanism and biological functions of c-Jun N-terminal kinase signalling via the c-Jun transcription factor. | Q34602976 | ||
| Interaction of cepharanthine with immobilized heat shock protein 90α (Hsp90α) and screening of Hsp90α inhibitors | Q36246298 | ||
| Redundancy of biological regulation as the basis of emergence of multidrug resistance. | Q36258245 | ||
| Predicting the future burden of cancer. | Q36350218 | ||
| Multidrug resistance: retrospect and prospects in anti-cancer drug treatment. | Q36536479 | ||
| The effects of biscoclaurine alkaloid cepharanthine on mammalian cells: implications for cancer, shock, and inflammatory diseases | Q36699612 | ||
| JNK1/2 Activation by an Extract from the Roots of Morus alba L. Reduces the Viability of Multidrug-Resistant MCF-7/Dox Cells by Inhibiting YB-1-Dependent MDR1 Expression. | Q37091032 | ||
| Systemic treatment of hepatocellular carcinoma: dawn of a new era? | Q37734303 | ||
| Nanomedicinal strategies to treat multidrug-resistant tumors: current progress | Q37763152 | ||
| Transcription factor c-Jun activation represses mdr-1 gene expression. | Q38351649 | ||
| Cepharanthine exerts antitumor activity on cholangiocarcinoma by inhibiting NF-kappaB. | Q39711864 | ||
| P-Glycoprotein inhibitory activity of lipophilic constituents of Echinacea pallida roots in a human proximal tubular cell line | Q39990056 | ||
| Reversal of P-glycoprotein-mediated multidrug resistance in cancer cells by the c-Jun NH2-terminal kinase | Q40333053 | ||
| Reversal of P-glycoprotein-mediated paclitaxel resistance by new synthetic isoprenoids in human bladder cancer cell line | Q40700402 | ||
| Exposure to the JNK inhibitor SP600125 (anthrapyrazolone) during early zebrafish development results in morphological defects | Q43930222 | ||
| Inhibitory effect of fruit extracts on P-glycoprotein-related efflux carriers: an in-vitro screening. | Q44160439 | ||
| JNK inhibitors increase osteogenesis in Nf1-deficient cells | Q46292665 | ||
| Reversal of multidrug resistance in drug-resistant cell line EAC/ADR by cepharanthine hydrochloride and its mechanism | Q46543031 | ||
| c-Jun is essential for the induction of Il-1β gene expression in in vitro activated Bergmann glial cells. | Q48878884 | ||
| P275 | copyright license | Creative Commons Attribution 3.0 Unported | Q14947546 |
| P6216 | copyright status | copyrighted | Q50423863 |
| P407 | language of work or name | English | Q1860 |
| P304 | page(s) | 164391 | |
| P577 | publication date | 2014-10-16 | |
| P1433 | published in | BioMed Research International | Q17509958 |
| P1476 | title | Downregulation of MDR1 gene by cepharanthine hydrochloride is related to the activation of c-Jun/JNK in K562/ADR cells | |
| P478 | volume | 2014 |
| Q50887482 | Astragaloside IV downregulates the expression of MDR1 in Bel‑7402/FU human hepatic cancer cells by inhibiting the JNK/c‑Jun/AP‑1 signaling pathway. |
| Q47975320 | Cepharanthine hydrochloride reverses P‑glycoprotein-mediated multidrug resistance in human ovarian carcinoma A2780/Taxol cells by inhibiting the PI3K/Akt signaling pathway |
| Q49492743 | Cepharanthine hydrochloride reverses the mdr1 (P-glycoprotein)-mediated esophageal squamous cell carcinoma cell cisplatin resistance through JNK and p53 signals |
| Q50637693 | Determination of cepharanthine in rat plasma by LC-MS/MS and its application to a pharmacokinetic study. |
| Q33750818 | Effect of traditional Chinese medicine components on multidrug resistance in tumors mediated by P-glycoprotein |
| Q36953023 | Guizhi Fuling Wan, a Traditional Chinese Herbal Formula, Sensitizes Cisplatin-Resistant Human Ovarian Cancer Cells through Inactivation of the PI3K/AKT/mTOR Pathway |
| Q49137075 | Inhibition of p38 mitogen-activated protein kinase signaling reduces multidrug transporter activity and anti-epileptic drug resistance in refractory epileptic rats |
| Q89100239 | MG‑132 reverses multidrug resistance by activating the JNK signaling pathway in FaDu/T cells |
| Q91260992 | Naturally occurring anti-cancer compounds: shining from Chinese herbal medicine |
| Q42349323 | Retracted: Downregulation of MDR1 Gene by Cepharanthine Hydrochloride Is Related to the Activation of c-Jun/JNK in K562/ADR Cells |
| Q30008841 | SH3GL1 inhibition reverses multidrug resistance in colorectal cancer cells by downregulation of MDR1/P-glycoprotein via EGFR/ERK/AP-1 pathway |
| Q55240749 | Solanine reverses multidrug resistance in human myelogenous leukemia K562/ADM cells by downregulating MRP1 expression. |
| Q41561662 | TRAM1 protect HepG2 cells from palmitate induced insulin resistance through ER stress-JNK pathway |
| Q35929260 | Wenyang Huazhuo Tongluo formula, a Chinese herbal decoction, improves skin fibrosis by promoting apoptosis and inhibiting proliferation through down-regulation of survivin and cyclin D1 in systemic sclerosis |
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