Regulation of cell proliferation through a KIT-mediated mechanism in benign prostatic hyperplasia.

scientific article published on 7 February 2012

Regulation of cell proliferation through a KIT-mediated mechanism in benign prostatic hyperplasia. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1002/PROS.22500
P698PubMed publication ID22314612

P50authorTakahiro YasuiQ40212291
P2093author name stringKenjiro Kohri
Shoichi Sasaki
Takashi Hamakawa
Yutaro Hayashi
Yoshiyuki Kojima
Yasue Kubota
Makoto Imura
P2860cites workStat1 associates with c-kit and is activated in response to stem cell factorQ24530497
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Efficacy and safety of imatinib mesylate in advanced gastrointestinal stromal tumorsQ29618610
Autophagy facilitates IFN-gamma-induced Jak2-STAT1 activation and cellular inflammationQ30496571
Current status of 5alpha-reductase inhibitors in the management of lower urinary tract symptoms and BPH.Q33602452
Molecular and cellular pathogenesis of benign prostatic hyperplasiaQ35947115
Gastrointestinal stromal tumors: past, present, and futureQ37224463
Imatinib treatment in chronic myelogenous leukemia: What have we learned so far?Q37809277
Targeting non-malignant disorders with tyrosine kinase inhibitors.Q37814085
Imatinib as adjuvant therapy for gastrointestinal stromal tumors: a systematic review.Q37851064
The effects of combination therapy with dutasteride and tamsulosin on clinical outcomes in men with symptomatic benign prostatic hyperplasia: 4-year results from the CombAT studyQ38378384
Attenuation of bladder overactivity in KIT mutant ratsQ39815784
Interstitial cells in the human prostate: a new therapeutic target?Q42444586
The long-term effect of doxazosin, finasteride, and combination therapy on the clinical progression of benign prostatic hyperplasiaQ44697461
A novel role for STAT1 in regulating murine erythropoiesis: deletion of STAT1 results in overall reduction of erythroid progenitors and alters their distributionQ44948873
Identification of c-kit-positive cells in the human prostate: the interstitial cells of CajalQ46420053
Altered distribution of interstitial cells in the guinea pig bladder following bladder outlet obstructionQ46971585
Altered expression of bladder mast cell growth factor receptor (c-kit) in interstitial cystitisQ48020238
P433issue14
P304page(s)1506-1513
P577publication date2012-02-07
P1433published inThe ProstateQ7758608
P1476titleRegulation of cell proliferation through a KIT-mediated mechanism in benign prostatic hyperplasia
P478volume72

Reverse relations

cites work (P2860)
Q38249926Calcium signalling in Cajal-like interstitial cells of the lower urinary tract
Q92316366Cellular and Molecular Mechanisms Underlying Prostate Cancer Development: Therapeutic Implications
Q38447424Molecular classification of benign prostatic hyperplasia: A gene expression profiling study in a rat model
Q35662979Normal peripheral prostate stromal cells stimulate prostate cancer development: roles of c-kit signal
Q42154804Prostate stem cells in the development of benign prostate hyperplasia and prostate cancer: emerging role and concepts
Q39399618Role of prostatic interstitial cells in prostate motility
Q57096896Tamsulosin modulates, but does not abolish the spontaneous activity in the guinea pig prostate gland
Q38264052The SCF/c-KIT system in the male: Survival strategies in fertility and cancer
Q39401758The stem cell factor (SCF)/c-KIT signalling in testis and prostate cancer