scholarly article | Q13442814 |
P2093 | author name string | Conrad C Weihl | |
P2860 | cites work | p97 and close encounters of every kind: a brief review | Q35922832 |
The UNC-45 chaperone mediates sarcomere assembly through myosin degradation in Caenorhabditis elegans | Q36118132 | ||
p97: The cell's molecular purgatory? | Q36515839 | ||
Impaired protein aggregate handling and clearance underlie the pathogenesis of p97/VCP-associated disease | Q36949378 | ||
Molecular and cellular mechanisms of learning disabilities: a focus on NF1. | Q37719525 | ||
Strumpellin is a novel valosin-containing protein binding partner linking hereditary spastic paraplegia to protein aggregation diseases. | Q39656824 | ||
The ubiquitin-selective chaperone CDC-48/p97 links myosin assembly to human myopathy | Q40157250 | ||
Cdc48/p97 promotes reformation of the nucleus by extracting the kinase Aurora B from chromatin | Q59096950 | ||
Imbalances in p97 co-factor interactions in human proteinopathy | Q24307689 | ||
Exome sequencing reveals VCP mutations as a cause of familial ALS | Q24631513 | ||
Mutations in the KIAA0196 gene at the SPG8 locus cause hereditary spastic paraplegia | Q24680886 | ||
The RasGAP proteins Ira2 and neurofibromin are negatively regulated by Gpb1 in yeast and ETEA in humans | Q27934997 | ||
AAA-ATPase p97/Cdc48p, a cytosolic chaperone required for endoplasmic reticulum-associated protein degradation | Q27939982 | ||
Specificity of Ca2+-dependent protein interactions mediated by the C2A domains of synaptotagmins | Q28138487 | ||
Inclusion body myopathy-associated mutations in p97/VCP impair endoplasmic reticulum-associated degradation | Q28284708 | ||
Proteasome and p97 mediate mitophagy and degradation of mitofusins induced by Parkin | Q29615623 | ||
Inclusion body myopathy associated with Paget disease of bone and frontotemporal dementia is caused by mutant valosin-containing protein | Q29619232 | ||
Endolysosomal sorting of ubiquitylated caveolin-1 is regulated by VCP and UBXD1 and impaired by VCP disease mutations | Q30179386 | ||
Valosin-containing protein (VCP) is required for autophagy and is disrupted in VCP disease | Q33589965 | ||
Neuronal remodeling and apoptosis require VCP-dependent degradation of the apoptosis inhibitor DIAP1. | Q34592532 | ||
Mutational analysis of the VCP gene in Parkinson's disease | Q35566788 | ||
Valosin-containing protein and neurofibromin interact to regulate dendritic spine density | Q35578562 | ||
Valosin containing protein associated fronto-temporal lobar degeneration: clinical presentation, pathologic features and pathogenesis | Q35635102 | ||
P433 | issue | 12 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 4627-4630 | |
P577 | publication date | 2011-11-21 | |
P1433 | published in | Journal of Clinical Investigation | Q3186904 |
P1476 | title | Another VCP interactor: NF is enough | |
P478 | volume | 121 |
Q27303589 | Cardiac-Restricted Expression of VCP/TER94 RNAi or Disease Alleles Perturbs Drosophila Heart Structure and Impairs Function. |
Q27015865 | From neurodevelopment to neurodegeneration: the interaction of neurofibromin and valosin-containing protein/p97 in regulation of dendritic spine formation |
Q30425624 | Proteomic profiling of a mouse model for ovarian granulosa cell tumor identifies VCP as a highly sensitive serum tumor marker in several human cancers |
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