The natural scorpion peptide, BmK NT1 activates voltage-gated sodium channels and produces neurotoxicity in primary cultured cerebellar granule cells.

scientific article published on 17 November 2015

The natural scorpion peptide, BmK NT1 activates voltage-gated sodium channels and produces neurotoxicity in primary cultured cerebellar granule cells. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1016/J.TOXICON.2015.11.005
P698PubMed publication ID26598793

P50authorZhengyu CaoQ86778576
P2093author name stringChunlei Zhang
Yuwei He
Jinping Qiao
Xiaohan Zou
P921main subjecttoxic encephalopathyQ7830379
P304page(s)33-41
P577publication date2015-11-17
P1433published inToxiconQ7830412
P1476titleThe natural scorpion peptide, BmK NT1 activates voltage-gated sodium channels and produces neurotoxicity in primary cultured cerebellar granule cells
P478volume109

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cites work (P2860)
Q39672702Activation of sodium channels by α-scorpion toxin, BmK NT1, produced neurotoxicity in cerebellar granule cells: an association with intracellular Ca2+ overloading.
Q36633107Development of a Rapid Throughput Assay for Identification of hNav1.7 Antagonist Using Unique Efficacious Sodium Channel Agonist, Antillatoxin

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