scholarly article | Q13442814 |
P50 | author | Spencer J. Collis | Q55303040 |
P2093 | author name string | William G Nelson | |
Julie M Schwaninger | |||
Larry E Dillehay | |||
Theodore L Deweese | |||
Alfred J Ntambi | |||
Thomas W Keller | |||
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Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses | Q24677973 | ||
Megabase chromatin domains involved in DNA double-strand breaks in vivo | Q24680284 | ||
DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139 | Q28131715 | ||
Genome maintenance mechanisms for preventing cancer | Q28131737 | ||
A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage | Q28144576 | ||
ATM phosphorylates histone H2AX in response to DNA double-strand breaks | Q28188651 | ||
Chromosomal stability and the DNA double-stranded break connection | Q28205315 | ||
DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation | Q28206029 | ||
DNA damage-induced G2-M checkpoint activation by histone H2AX and 53BP1 | Q28215916 | ||
Genomic instability in mice lacking histone H2AX | Q28589826 | ||
ATM and related protein kinases: safeguarding genome integrity | Q29547735 | ||
Inverse radiation dose-rate effects on somatic and germ-line mutations and DNA damage rates | Q33900279 | ||
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Low-dose hypersensitivity: current status and possible mechanisms. | Q34137832 | ||
Two molecularly distinct G(2)/M checkpoints are induced by ionizing irradiation | Q34300338 | ||
Direct activation of the ATM protein kinase by the Mre11/Rad50/Nbs1 complex | Q34311086 | ||
Sensing and repairing DNA double-strand breaks | Q34642563 | ||
Low-dose hyper-radiosensitivity: a consequence of ineffective cell cycle arrest of radiation-damaged G2-phase cells. | Q35674966 | ||
Requirement of the MRN complex for ATM activation by DNA damage | Q36267304 | ||
DNA-PK is activated by nucleosomes and phosphorylates H2AX within the nucleosomes in an acetylation-dependent manner | Q40315391 | ||
ATM and DNA-PK function redundantly to phosphorylate H2AX after exposure to ionizing radiation | Q40570996 | ||
Phosphorylation of histone H2AX as a measure of radiosensitivity | Q40594249 | ||
Effects of exposure to low-dose-rate (60)co gamma rays on human tumor cells in vitro. | Q40711171 | ||
Distinct functional domains of Nbs1 modulate the timing and magnitude of ATM activation after low doses of ionizing radiation | Q44817175 | ||
An association between the radiation-induced arrest of G2-phase cells and low-dose hyper-radiosensitivity: a plausible underlying mechanism? | Q47817819 | ||
Expression of phosphorylated histone H2AX in cultured cell lines following exposure to X-rays. | Q51834143 | ||
Protective packaging for DNA | Q59068412 | ||
P433 | issue | 48 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | DNA damage | Q5205747 |
P304 | page(s) | 49624-49632 | |
P577 | publication date | 2004-09-17 | |
P1433 | published in | Journal of Biological Chemistry | Q867727 |
P1476 | title | Evasion of early cellular response mechanisms following low level radiation-induced DNA damage | |
P478 | volume | 279 |
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