Evasion of early cellular response mechanisms following low level radiation-induced DNA damage.

scientific article published on 17 September 2004

Evasion of early cellular response mechanisms following low level radiation-induced DNA damage. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1074/JBC.M409600200
P698PubMed publication ID15377658
P5875ResearchGate publication ID8336575

P50authorSpencer J. CollisQ55303040
P2093author name stringWilliam G Nelson
Julie M Schwaninger
Larry E Dillehay
Theodore L Deweese
Alfred J Ntambi
Thomas W Keller
P2860cites workEndogenous DNA double-strand breaks: Production, fidelity of repair, and induction of cancerQ24647338
Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray dosesQ24677973
Megabase chromatin domains involved in DNA double-strand breaks in vivoQ24680284
DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139Q28131715
Genome maintenance mechanisms for preventing cancerQ28131737
A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damageQ28144576
ATM phosphorylates histone H2AX in response to DNA double-strand breaksQ28188651
Chromosomal stability and the DNA double-stranded break connectionQ28205315
DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociationQ28206029
DNA damage-induced G2-M checkpoint activation by histone H2AX and 53BP1Q28215916
Genomic instability in mice lacking histone H2AXQ28589826
ATM and related protein kinases: safeguarding genome integrityQ29547735
Inverse radiation dose-rate effects on somatic and germ-line mutations and DNA damage ratesQ33900279
Increased ionizing radiation sensitivity and genomic instability in the absence of histone H2AXQ34031720
Low-dose hypersensitivity: current status and possible mechanisms.Q34137832
Two molecularly distinct G(2)/M checkpoints are induced by ionizing irradiationQ34300338
Direct activation of the ATM protein kinase by the Mre11/Rad50/Nbs1 complexQ34311086
Sensing and repairing DNA double-strand breaksQ34642563
Low-dose hyper-radiosensitivity: a consequence of ineffective cell cycle arrest of radiation-damaged G2-phase cells.Q35674966
Requirement of the MRN complex for ATM activation by DNA damageQ36267304
DNA-PK is activated by nucleosomes and phosphorylates H2AX within the nucleosomes in an acetylation-dependent mannerQ40315391
ATM and DNA-PK function redundantly to phosphorylate H2AX after exposure to ionizing radiationQ40570996
Phosphorylation of histone H2AX as a measure of radiosensitivityQ40594249
Effects of exposure to low-dose-rate (60)co gamma rays on human tumor cells in vitro.Q40711171
Distinct functional domains of Nbs1 modulate the timing and magnitude of ATM activation after low doses of ionizing radiationQ44817175
An association between the radiation-induced arrest of G2-phase cells and low-dose hyper-radiosensitivity: a plausible underlying mechanism?Q47817819
Expression of phosphorylated histone H2AX in cultured cell lines following exposure to X-rays.Q51834143
Protective packaging for DNAQ59068412
P433issue48
P407language of work or nameEnglishQ1860
P921main subjectDNA damageQ5205747
P304page(s)49624-49632
P577publication date2004-09-17
P1433published inJournal of Biological ChemistryQ867727
P1476titleEvasion of early cellular response mechanisms following low level radiation-induced DNA damage
P478volume279

Reverse relations

cites work (P2860)
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