review article | Q7318358 |
scholarly article | Q13442814 |
P356 | DOI | 10.1111/J.1749-6632.1993.TB23045.X |
P698 | PubMed publication ID | 8239276 |
P2093 | author name string | Mattson MP | |
Rydel RE | |||
Smith-Swintosky VL | |||
Barger SW | |||
P2860 | cites work | Processing of Alzheimer beta/A4 amyloid precursor protein: modulation by agents that regulate protein phosphorylation | Q33731523 |
Isolation and quantification of soluble Alzheimer's beta-peptide from biological fluids | Q34255552 | ||
Neurotoxicity of a fragment of the amyloid precursor associated with Alzheimer's disease | Q34415666 | ||
Calcium as sculptor and destroyer of neural circuitry | Q35680713 | ||
Alzheimer's disease: a cell biological perspective | Q36129542 | ||
Intracellular accumulation and resistance to degradation of the Alzheimer amyloid A4/beta protein | Q37149874 | ||
Excitatory amino acids and Alzheimer's disease | Q38301056 | ||
Secretory processing of the Alzheimer amyloid beta/A4 protein precursor is increased by protein phosphorylation | Q43705003 | ||
In vitro aging of beta-amyloid protein causes peptide aggregation and neurotoxicity | Q44418518 | ||
Secreted form of amyloid beta protein precursor is involved in the growth regulation of fibroblasts. | Q45966280 | ||
Basic FGF, NGF, and IGFs protect hippocampal and cortical neurons against iron-induced degeneration | Q48291438 | ||
Basic fibroblast growth factor protects against hypoxia-ischemia and NMDA neurotoxicity in neonatal rats | Q48324531 | ||
Evidence for excitoprotective and intraneuronal calcium-regulating roles for secreted forms of the beta-amyloid precursor protein | Q48342880 | ||
βA4 amyloid protein deposition in brain after head trauma | Q48611315 | ||
Selective induction of Kunitz-type protease inhibitor domain-containing amyloid precursor protein mRNA after persistent focal ischemia in rat cerebral cortex | Q48737468 | ||
Diminished glucose transport in Alzheimer's disease: dynamic PET studies | Q48783668 | ||
Solution Structures of β Peptide and Its Constituent Fragments: Relation to Amyloid Deposition | Q53182604 | ||
P407 | language of work or name | English | Q1860 |
P921 | main subject | Alzheimer's disease | Q11081 |
P304 | page(s) | 158-164 | |
P577 | publication date | 1993-09-01 | |
P1433 | published in | Annals of the New York Academy of Sciences | Q2431664 |
P1476 | title | beta-Amyloid precursor protein mismetabolism and loss of calcium homeostasis in Alzheimer's disease | |
P478 | volume | 695 |
Q34109343 | AICD Overexpression in Neuro 2A Cells Regulates Expression of PTCH1 and TRPC5 |
Q44122940 | Abeta(25-35) and Abeta(1-40) act on different calcium channels in CA1 hippocampal neurons |
Q45969414 | Activation of group III metabotropic glutamate receptor reduces intracellular calcium in beta-amyloid peptide [31-35]-treated cortical neurons. |
Q44071716 | Electrophysiologic properties of channels induced by Abeta25-35 in planar lipid bilayers |
Q35688992 | Lead decreases cell survival, proliferation, and neuronal differentiation of primary cultured adult neural precursor cells through activation of the JNK and p38 MAP kinases. |
Q37772809 | Neuroinflammatory processes in Alzheimer's disease |
Q73457845 | Oxidative stress and Ca2+ influx upregulate calpain and induce apoptosis in PC12 cells |
Q37928994 | Oxidative stress and β-amyloid protein in Alzheimer's disease |
Q38183363 | Role of Activated Glia and of Glial Cytokines in Alzheimer's Disease: A Review |
Q34039304 | Strategies to delay the onset of Alzheimer's disease. |
Q41060580 | The significance of glucose turnover in the brain in the pathogenetic mechanisms of Alzheimer's disease |
Q34018961 | Theoretical models of the ion channel structure of amyloid beta-protein |
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