| scholarly article | Q13442814 |
| P2093 | author name string | Sheng Y | |
| Chang KW | |||
| Gombold JL | |||
| P2860 | cites work | MHV-A59 fusion mutants are attenuated and display altered hepatotropism | Q44985679 |
| Adoptive transfer of EAE-like lesions from rats with coronavirus-induced demyelinating encephalomyelitis | Q45232320 | ||
| Mouse hepatitis virus A59-induced demyelination can occur in the absence of CD8+ T cells. | Q45788721 | ||
| Experimental demyelination produced by the A59 strain of mouse hepatitis virus | Q45796770 | ||
| The predicted primary structure of the peplomer protein E2 of the porcine coronavirus transmissible gastroenteritis virus | Q45834645 | ||
| Fatty acid acylation of viral proteins in murine hepatitis virus-infected cells. Brief report | Q45834701 | ||
| Demyelination induced by murine hepatitis virus JHM strain (MHV-4) is immunologically mediated. | Q45850723 | ||
| Limbic encephalitis after inhalation of a murine coronavirus | Q48166565 | ||
| Molecular cloning and expression of a spike protein of neurovirulent murine coronavirus JHMV variant c1-2 | Q48176245 | ||
| Dissociation of demyelination and viral clearance in congenitally immunodeficient mice infected with murine coronavirus JHM. | Q49022525 | ||
| Membrane fusion | Q67584890 | ||
| Deduced sequence of the bovine coronavirus spike protein and identification of the internal proteolytic cleavage site | Q68505958 | ||
| Pathogenesis of demyelination induced by a mouse hepatitis | Q68658605 | ||
| Interaction of immune and central nervous systems: contribution of anti-viral Thy-1+ cells to demyelination induced by coronavirus JHM | Q72228101 | ||
| An efficient site-directed mutagenesis method based on PCR | Q72443360 | ||
| Acylation of virol. spike glycoproteins: A feature of enveloped RNA viruses | Q72924789 | ||
| Proteolytic cleavage of the murine coronavirus surface glycoprotein is not required for fusion activity | Q72947935 | ||
| Site-directed mutagenesis: a two-step method using PCR and DpnI | Q73818682 | ||
| Interactions of the cytoplasmic domains of human and simian retroviral transmembrane proteins with components of the clathrin adaptor complexes modulate intracellular and cell surface expression of envelope glycoproteins | Q24517016 | ||
| Coronavirus induction of class I major histocompatibility complex expression in murine astrocytes is virus strain specific | Q27485104 | ||
| Eukaryotic transient-expression system based on recombinant vaccinia virus that synthesizes bacteriophage T7 RNA polymerase | Q27860943 | ||
| Possible role for fatty acyl-coenzyme A in intracellular protein transport | Q28305277 | ||
| Mutations within the proteolytic cleavage site of the Rous sarcoma virus glycoprotein that block processing to gp85 and gp37 | Q30403429 | ||
| Inner but not outer membrane leaflets control the transition from glycosylphosphatidylinositol-anchored influenza hemagglutinin-induced hemifusion to full fusion | Q30442161 | ||
| GPI-anchored influenza hemagglutinin induces hemifusion to both red blood cell and planar bilayer membranes | Q30442210 | ||
| Virus-cell and cell-cell fusion | Q30469833 | ||
| Three-dimensional structure of membrane and surface proteins | Q34258165 | ||
| Molecular cloning of the gene encoding the putative polymerase of mouse hepatitis coronavirus, strain A59. | Q35467555 | ||
| Differences in the role of the cytoplasmic domain of human parainfluenza virus fusion proteins | Q35850959 | ||
| Analysis of the cell fusion activities of chimeric simian immunodeficiency virus-murine leukemia virus envelope proteins: inhibitory effects of the R peptide | Q35853520 | ||
| Palmitylation of the influenza virus hemagglutinin (H3) is not essential for virus assembly or infectivity | Q35855976 | ||
| Murine coronavirus membrane fusion is blocked by modification of thiols buried within the spike protein | Q35864708 | ||
| Analysis of the murine leukemia virus R peptide: delineation of the molecular determinants which are important for its fusion inhibition activity | Q35898515 | ||
| The transmembrane domain in viral fusion: essential role for a conserved glycine residue in vesicular stomatitis virus G protein | Q36001197 | ||
| Truncation of the COOH-terminal region of the paramyxovirus SV5 fusion protein leads to hemifusion but not complete fusion | Q36237467 | ||
| The presence of cysteine in the cytoplasmic domain of the vesicular stomatitis virus glycoprotein is required for palmitate addition | Q36266218 | ||
| Postassembly cleavage of a retroviral glycoprotein cytoplasmic domain removes a necessary incorporation signal and activates fusion activity. | Q36623006 | ||
| Function of the cytoplasmic domain of a retroviral transmembrane protein: p15E-p2E cleavage activates the membrane fusion capability of the murine leukemia virus Env protein | Q36629882 | ||
| Uncoupled expression of Moloney murine leukemia virus envelope polypeptides SU and TM: a functional analysis of the role of TM domains in viral entry. | Q36633589 | ||
| pH-independent murine leukemia virus ecotropic envelope-mediated cell fusion: implications for the role of the R peptide and p12E TM in viral entry | Q36633601 | ||
| Fusogenic mechanisms of enveloped-virus glycoproteins analyzed by a novel recombinant vaccinia virus-based assay quantitating cell fusion-dependent reporter gene activation. | Q36634800 | ||
| Mutations at palmitylation sites of the influenza virus hemagglutinin affect virus formation | Q36635084 | ||
| Expression and characterization of glycophospholipid-anchored human immunodeficiency virus type 1 envelope glycoproteins | Q36651824 | ||
| Alteration of the pH dependence of coronavirus-induced cell fusion: effect of mutations in the spike glycoprotein | Q36684115 | ||
| Truncation of the human immunodeficiency virus type 1 transmembrane glycoprotein cytoplasmic domain blocks virus infectivity | Q36685270 | ||
| Effects of altering palmitylation sites on biosynthesis and function of the influenza virus hemagglutinin | Q36687652 | ||
| Quantitative basic residue requirements in the cleavage-activation site of the fusion glycoprotein as a determinant of virulence for Newcastle disease virus | Q36826673 | ||
| Use of a hybrid vaccinia virus-T7 RNA polymerase system for expression of target genes | Q36843702 | ||
| Proteolytic cleavage of the E2 glycoprotein of murine coronavirus: activation of cell-fusing activity of virions by trypsin and separation of two different 90K cleavage fragments. | Q36854786 | ||
| Proteolytic cleavage of the E2 glycoprotein of murine coronavirus: host-dependent differences in proteolytic cleavage and cell fusion. | Q36854808 | ||
| Sequence requirements for cleavage activation of influenza virus hemagglutinin expressed in mammalian cells | Q37481771 | ||
| Structural and functional analysis of the surface protein of human coronavirus OC43. | Q38317987 | ||
| Fatty acylation of proteins | Q38729752 | ||
| Fusion activity of transmembrane and cytoplasmic domain chimeras of the influenza virus glycoprotein hemagglutinin. | Q39577087 | ||
| The covalent modification of eukaryotic proteins with lipid | Q39655733 | ||
| Functional analysis of the cytoplasmic tail of Moloney murine leukemia virus envelope protein | Q39879338 | ||
| Influence of membrane anchoring and cytoplasmic domains on the fusogenic activity of vesicular stomatitis virus glycoprotein G | Q39881634 | ||
| The function of the spike protein of mouse hepatitis virus strain A59 can be studied on virus-like particles: cleavage is not required for infectivity | Q40015426 | ||
| Fusion-defective mutants of mouse hepatitis virus A59 contain a mutation in the spike protein cleavage signal | Q40046320 | ||
| Intracellular transport of recombinant coronavirus spike proteins: implications for virus assembly | Q40107532 | ||
| Isolation of coronavirus envelope glycoproteins and interaction with the viral nucleocapsid | Q40306426 | ||
| Paramyxovirus fusion: a hypothesis for changes | Q40767649 | ||
| Mouse hepatitis virus A59 increases steady-state levels of MHC mRNAs in primary glial cell cultures and in the murine central nervous system | Q41087971 | ||
| The role of the cytoplasmic tail region of influenza virus hemagglutinin in formation and growth of fusion pores | Q41093333 | ||
| Mutational analysis of the murine coronavirus spike protein: effect on cell-to-cell fusion | Q41258530 | ||
| Acylation of viral glycoproteins: structural requirements for palmitoylation of transmembrane proteins | Q41314369 | ||
| Cell fusion activity of the simian immunodeficiency virus envelope protein is modulated by the intracytoplasmic domain | Q41517820 | ||
| Aminopeptidase N is a major receptor for the entero-pathogenic coronavirus TGEV | Q41620183 | ||
| The complete sequence (22 kilobases) of murine coronavirus gene 1 encoding the putative proteases and RNA polymerase | Q41697955 | ||
| Nucleotide sequence of the gene encoding the spike glycoprotein of human coronavirus HCV 229E. | Q41734901 | ||
| Entry of mouse hepatitis virus into cells by endosomal and nonendosomal pathways | Q41928239 | ||
| Molecular characterization of the S protein gene of human coronavirus OC43. | Q42615390 | ||
| Cloning and sequencing of the gene encoding the spike protein of the coronavirus IBV. | Q42651884 | ||
| Coronavirus glycoprotein E1, a new type of viral glycoprotein | Q43597537 | ||
| Sequence analysis reveals extensive polymorphism and evidence of deletions within the E2 glycoprotein gene of several strains of murine hepatitis virus | Q44314306 | ||
| Primary structure of the glycoprotein E2 of coronavirus MHV-A59 and identification of the trypsin cleavage site | Q44628204 | ||
| Lipid-anchored influenza hemagglutinin promotes hemifusion, not complete fusion | Q44633908 | ||
| Retained in vitro infectivity and cytopathogenicity of HIV-1 despite truncation of the C-terminal tail of the env gene product | Q44898633 | ||
| P433 | issue | 1 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | Murine coronavirus | Q18907882 |
| structural biology | Q908902 | ||
| P304 | page(s) | 212-224 | |
| P577 | publication date | 2000-03-01 | |
| P1433 | published in | Virology | Q7934867 |
| P1476 | title | Coronavirus-induced membrane fusion requires the cysteine-rich domain in the spike protein | |
| P478 | volume | 269 |
| Q40527153 | A single amino acid mutation in the spike protein of coronavirus infectious bronchitis virus hampers its maturation and incorporation into virions at the nonpermissive temperature |
| Q39871099 | Aquareovirus effects syncytiogenesis by using a novel member of the FAST protein family translated from a noncanonical translation start site |
| Q38294473 | Aromatic amino acids in the juxtamembrane domain of severe acute respiratory syndrome coronavirus spike glycoprotein are important for receptor-dependent virus entry and cell-cell fusion |
| Q47941525 | Characterization of the 3a protein of SARS-associated coronavirus in infected vero E6 cells and SARS patients |
| Q39499054 | Characterization of the spike protein of human coronavirus NL63 in receptor binding and pseudotype virus entry |
| Q34193942 | Coronavirus pathogenesis and the emerging pathogen severe acute respiratory syndrome coronavirus |
| Q37492706 | Genetic analysis of determinants for spike glycoprotein assembly into murine coronavirus virions: distinct roles for charge-rich and cysteine-rich regions of the endodomain |
| Q81057074 | Genetic analysis of the SARS-coronavirus spike glycoprotein functional domains involved in cell-surface expression and cell-to-cell fusion |
| Q36621999 | Genetic and molecular biological analysis of protein-protein interactions in coronavirus assembly |
| Q45326273 | Identification of N-linked glycosylation sites in the spike protein and their functional impact on the replication and infectivity of coronavirus infectious bronchitis virus in cell culture. |
| Q40329154 | Important role for the transmembrane domain of severe acute respiratory syndrome coronavirus spike protein during entry |
| Q35544383 | Incorporation of spike and membrane glycoproteins into coronavirus virions |
| Q84315456 | Membrane binding proteins of coronaviruses |
| Q81155962 | Molecular interactions in the assembly of coronaviruses |
| Q39759201 | Mutagenesis of the transmembrane domain of the SARS coronavirus spike glycoprotein: refinement of the requirements for SARS coronavirus cell entry |
| Q39490649 | Mutation in the cytoplasmic retrieval signal of porcine epidemic diarrhea virus spike (S) protein is responsible for enhanced fusion activity |
| Q37168282 | Negatively charged residues in the endodomain are critical for specific assembly of spike protein into murine coronavirus |
| Q103826761 | New Consensus pattern in Spike CoV-2: potential implications in coagulation process and cell-cell fusion |
| Q34037293 | Palmitoylation of SARS-CoV S protein is necessary for partitioning into detergent-resistant membranes and cell-cell fusion but not interaction with M protein |
| Q38966996 | Palmitoylation of the Alphacoronavirus TGEV spike protein S is essential for incorporation into virus-like particles but dispensable for S-M interaction. |
| Q53585772 | Palmitoylation of the cysteine-rich endodomain of the SARS-coronavirus spike glycoprotein is important for spike-mediated cell fusion. |
| Q39996417 | Palmitoylation, membrane-proximal basic residues, and transmembrane glycine residues in the reovirus p10 protein are essential for syncytium formation |
| Q34302034 | Palmitoylations on murine coronavirus spike proteins are essential for virion assembly and infectivity. |
| Q39613956 | Partial deletion in the spike endodomain of mouse hepatitis virus decreases the cytopathic effect but maintains foreign protein expression in infected cells |
| Q35731257 | Phylogenomics and bioinformatics of SARS-CoV. |
| Q34267537 | Protein-Lipid Interplay in Fusion and Fission of Biological Membranes |
| Q39378520 | Replication of murine coronavirus requires multiple cysteines in the endodomain of spike protein |
| Q37511480 | Retroviruses pseudotyped with the severe acute respiratory syndrome coronavirus spike protein efficiently infect cells expressing angiotensin-converting enzyme 2. |
| Q37432095 | Role of spike protein endodomains in regulating coronavirus entry |
| Q36415424 | Severe acute respiratory syndrome coronavirus entry into host cells: Opportunities for therapeutic intervention |
| Q40444606 | Spike protein assembly into the coronavirion: exploring the limits of its sequence requirements |
| Q84315921 | Structural insights into coronavirus entry |
| Q39992400 | Synthesis and characterization of a native, oligomeric form of recombinant severe acute respiratory syndrome coronavirus spike glycoprotein |
| Q39879563 | The Autographa californica multicapsid nucleopolyhedrovirus GP64 protein: analysis of transmembrane domain length and sequence requirements |
| Q36550910 | The molecular biology of coronaviruses |
| Q39685296 | The transmembrane domain and cytoplasmic tail of herpes simplex virus type 1 glycoprotein H play a role in membrane fusion |
| Q45360494 | Two palmitylated cysteine residues of the severe acute respiratory syndrome coronavirus spike (S) protein are critical for S incorporation into virus-like particles, but not for M-S co-localization |
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