scholarly article | Q13442814 |
P356 | DOI | 10.1002/MUS.25049 |
P698 | PubMed publication ID | 26799358 |
P2093 | author name string | Carmel Armon | |
P2860 | cites work | The age distribution of cancer and a multi-stage theory of carcinogenesis | Q24564904 |
ALS motor phenotype heterogeneity, focality, and spread: deconstructing motor neuron degeneration | Q37593829 | ||
Clinical diagnosis and management of amyotrophic lateral sclerosis | Q37944429 | ||
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Distinct clinical characteristics of C9orf72 expansion carriers compared with GRN, MAPT, and nonmutation carriers in a Flanders-Belgian FTLD cohort | Q48200005 | ||
Limited corticospinal tract involvement in amyotrophic lateral sclerosis subjects with the A4V mutation in the copper/zinc superoxide dismutase gene | Q48447713 | ||
Respiratory apraxia in amyotrophic lateral sclerosis | Q48467430 | ||
Familial amyotrophic lateral sclerosis with frontotemporal dementia is linked to a locus on chromosome 9p13.2-21.3. | Q48640341 | ||
Stages of the pathologic process in Alzheimer disease: age categories from 1 to 100 years | Q48835886 | ||
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The natural history of amyotrophic lateral sclerosis and the use of natural history controls in therapeutic trials. | Q51058673 | ||
Consensus criteria for the diagnosis of frontotemporal cognitive and behavioural syndromes in amyotrophic lateral sclerosis. | Q51868850 | ||
From clues to mechanisms: understanding ALS initiation and spread. | Q54397878 | ||
Tobacco smoking and cancer: A meta-analysis | Q56779419 | ||
Concordance between site of onset and limb dominance in amyotrophic lateral sclerosis | Q57357950 | ||
Familial ALS with extreme phenotypic variability due to the I113T SOD1 mutation | Q58305775 | ||
Frequency of the C9orf72 hexanucleotide repeat expansion in patients with amyotrophic lateral sclerosis and frontotemporal dementia: a cross-sectional study | Q24600803 | ||
Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS | Q24633692 | ||
A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTD | Q24634583 | ||
Is neurodegenerative disease a long-latency response to early-life genotoxin exposure? | Q26824755 | ||
State of play in amyotrophic lateral sclerosis genetics | Q26864816 | ||
Emerging mechanisms of molecular pathology in ALS | Q28081239 | ||
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Animal models of brain maldevelopment induced by cycad plant genotoxins | Q28655111 | ||
Frontotemporal lobar degeneration: a consensus on clinical diagnostic criteria | Q29614410 | ||
Neurodegenerative diseases target large-scale human brain networks | Q29614723 | ||
Damage and repair of nerve cell DNA in toxic stress | Q30759422 | ||
Prevalence and correlates of neuropsychological deficits in amyotrophic lateral sclerosis | Q33735560 | ||
Evolution in health and medicine Sackler colloquium: Somatic evolutionary genomics: mutations during development cause highly variable genetic mosaicism with risk of cancer and neurodegeneration. | Q33844436 | ||
Identification of two novel loci for dominantly inherited familial amyotrophic lateral sclerosis | Q33905231 | ||
Are amyotrophic lateral sclerosis patients cognitively normal? | Q33965351 | ||
Amyotrophic lateral sclerosis-motor neuron disease, monoclonal gammopathy, hyperparathyroidism, and B12 deficiency: case report and review of the literature. | Q34138933 | ||
Analysis of amyotrophic lateral sclerosis as a multistep process: a population-based modelling study | Q34340508 | ||
The widening spectrum of C9ORF72-related disease; genotype/phenotype correlations and potential modifiers of clinical phenotype | Q34402056 | ||
Correlates of human handedness in primary motor cortex: a review and hypothesis | Q34659328 | ||
An evidence-based medicine approach to the evaluation of the role of exogenous risk factors in sporadic amyotrophic lateral sclerosis | Q35147590 | ||
Cognitive impairment, frontotemporal dementia, and the motor neuron diseases | Q35165800 | ||
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Smoking and risk of amyotrophic lateral sclerosis: a pooled analysis of 5 prospective cohorts | Q35869201 | ||
A two-stage theory of carcinogenesis in relation to the age distribution of human cancer | Q36132593 | ||
Acquired nucleic acid changes may trigger sporadic amyotrophic lateral sclerosis | Q36158213 | ||
ALS and Frontotemporal Dysfunction: A Review | Q36178404 | ||
Amyotrophic lateral sclerosis as a complex genetic disease | Q36594346 | ||
Sports and trauma in amyotrophic lateral sclerosis revisited | Q36903268 | ||
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Stages of pTDP-43 pathology in amyotrophic lateral sclerosis | Q37202139 | ||
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DNA repair deficiency and neurological disease | Q37368873 | ||
Amyotrophic lateral sclerosis: A 40-year personal perspective | Q37397392 | ||
Smoking may be considered an established risk factor for sporadic ALS. | Q37459710 | ||
P433 | issue | 6 | |
P921 | main subject | amyotrophic lateral sclerosis | Q206901 |
P304 | page(s) | 842-849 | |
P577 | publication date | 2016-01-21 | |
P1433 | published in | Muscle and Nerve | Q15764281 |
P1476 | title | Accrued somatic mutations (nucleic acid changes) trigger ALS: 2005-2015 update | |
P478 | volume | 53 |
Q57464271 | ALSUntangled 38: L-serine |
Q58869833 | Amyotrophic lateral sclerosis and exposure to diesel exhaust in a Danish cohort |
Q50197773 | Ethics of clinical research in patients with ALS: is there a risk of exploitation? |
Q40091016 | Olfactory Function in Latino Farmworkers Over 2 Years: Longitudinal Exploration of Subclinical Neurological Effects of Pesticide Exposure |
Q40341875 | Puzzles in modern biology. IV. Neurodegeneration, localized origin and widespread decay. |
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