scholarly article | Q13442814 |
P356 | DOI | 10.1016/S0076-6879(97)88011-1 |
P698 | PubMed publication ID | 9356991 |
P2093 | author name string | Long D | |
Doms RW | |||
Rucker J | |||
Berson JF | |||
Doranz BJ | |||
Edinger AL | |||
P2860 | cites work | Human immunodeficiency virus envelope glycoprotein/CD4-mediated fusion of nonprimate cells with human cells | Q36805743 |
Regions in beta-chemokine receptors CCR5 and CCR2b that determine HIV-1 cofactor specificity | Q39413074 | ||
Replication-deficient vaccinia virus encoding bacteriophage T7 RNA polymerase for transient gene expression in mammalian cells | Q41334221 | ||
Initial stages of HIV-1 envelope glycoprotein-mediated cell fusion monitored by a new assay based on redistribution of fluorescent dyes | Q41663436 | ||
Endoproteolytic cleavage of gp160 is required for the activation of human immunodeficiency virus | Q41876028 | ||
CD4-independent infection by HIV-2 is mediated by fusin/CXCR4. | Q42813563 | ||
HIV blocked by chemokine antagonist | Q59084831 | ||
Common mechanism of infection by lentiviruses | Q59096936 | ||
Secreted placental alkaline phosphatase: a powerful new quantitative indicator of gene expression in eukaryotic cells | Q68504983 | ||
HIV-1 Entry Cofactor: Functional cDNA Cloning of a Seven-Transmembrane, G Protein-Coupled Receptor | Q22242268 | ||
Identification of a major co-receptor for primary isolates of HIV-1 | Q22251282 | ||
A dual-tropic primary HIV-1 isolate that uses fusin and the beta-chemokine receptors CKR-5, CKR-3, and CKR-2b as fusion cofactors | Q28118386 | ||
Identification of RANTES, MIP-1 alpha, and MIP-1 beta as the major HIV-suppressive factors produced by CD8+ T cells | Q28270680 | ||
The beta-chemokine receptors CCR3 and CCR5 facilitate infection by primary HIV-1 isolates | Q28282895 | ||
CC CKR5: a RANTES, MIP-1alpha, MIP-1beta receptor as a fusion cofactor for macrophage-tropic HIV-1 | Q28646859 | ||
Resistance to HIV-1 infection in caucasian individuals bearing mutant alleles of the CCR-5 chemokine receptor gene | Q29614892 | ||
Homozygous defect in HIV-1 coreceptor accounts for resistance of some multiply-exposed individuals to HIV-1 infection | Q29614956 | ||
HIV-1 entry into CD4+ cells is mediated by the chemokine receptor CC-CKR-5 | Q29616094 | ||
The T4 gene encodes the AIDS virus receptor and is expressed in the immune system and the brain | Q29618472 | ||
Membrane fusion mediated by the influenza virus hemagglutinin requires the concerted action of at least three hemagglutinin trimers | Q30442203 | ||
A seven-transmembrane domain receptor involved in fusion and entry of T-cell-tropic human immunodeficiency virus type 1 strains. | Q35868457 | ||
Dilation of the influenza hemagglutinin fusion pore revealed by the kinetics of individual cell-cell fusion events | Q36237488 | ||
Fusogenic mechanisms of enveloped-virus glycoproteins analyzed by a novel recombinant vaccinia virus-based assay quantitating cell fusion-dependent reporter gene activation. | Q36634800 | ||
Failure of human immunodeficiency virus entry and infection in CD4-positive human brain and skin cells | Q36799015 | ||
P407 | language of work or name | English | Q1860 |
P921 | main subject | membrane fusion involved in viral entry into host cell | Q22295497 |
P304 | page(s) | 118-133 | |
P577 | publication date | 1997-01-01 | |
P1433 | published in | Methods in Enzymology | Q2076903 |
P1476 | title | Cell-cell fusion assay to study role of chemokine receptors in human immunodeficiency virus type 1 entry | |
P478 | volume | 288 |
Q39682625 | A replication-competent, neutralization-sensitive variant of simian immunodeficiency virus lacking 100 amino acids of envelope |
Q34610959 | Adaptive mutations in a human immunodeficiency virus type 1 envelope protein with a truncated V3 loop restore function by improving interactions with CD4. |
Q40579277 | Amino acid 324 in the simian immunodeficiency virus SIVmac V3 loop can confer CD4 independence and modulate the interaction with CCR5 and alternative coreceptors |
Q39581093 | An orphan seven-transmembrane domain receptor expressed widely in the brain functions as a coreceptor for human immunodeficiency virus type 1 and simian immunodeficiency virus |
Q41857872 | Baseline resistance of primary human immunodeficiency virus type 1 strains to the CXCR4 inhibitor AMD3100. |
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Q40179461 | Clinical resistance to enfuvirtide does not affect susceptibility of human immunodeficiency virus type 1 to other classes of entry inhibitors |
Q35947621 | Conserved changes in envelope function during human immunodeficiency virus type 1 coreceptor switching |
Q36878906 | Derivation and Characterization of a CD4-Independent, Non-CD4-Tropic Simian Immunodeficiency Virus |
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Q33825418 | Determinants of CD4 independence for a human immunodeficiency virus type 1 variant map outside regions required for coreceptor specificity |
Q33796067 | Determinants of syncytium formation in microglia by human immunodeficiency virus type 1: role of the V1/V2 domains |
Q55508034 | Differential functional phenotypes of two primary HIV-1 strains resulting from homologous point mutations in the LLP domains of the envelope gp41 intracytoplasmic domain. |
Q34045610 | Distinct functional sites for human immunodeficiency virus type 1 and stromal cell-derived factor 1alpha on CXCR4 transmembrane helical domains |
Q34055508 | Distinct molecular pathways to X4 tropism for a V3-truncated human immunodeficiency virus type 1 lead to differential coreceptor interactions and sensitivity to a CXCR4 antagonist |
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Q24678540 | Functional deletion of the CCR5 receptor by intracellular immunization produces cells that are refractory to CCR5-dependent HIV-1 infection and cell fusion |
Q36923795 | HIV envelope binding by macrophage-expressed gp340 promotes HIV-1 infection. |
Q33807529 | HIV type 1 from a patient with baseline resistance to CCR5 antagonists uses drug-bound receptor for entry |
Q34465007 | Identification of gp120 binding sites on CXCR4 by using CD4-independent human immunodeficiency virus type 2 Env proteins |
Q40561855 | Impact of mutations in the coreceptor binding site on human immunodeficiency virus type 1 fusion, infection, and entry inhibitor sensitivity |
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Q30368471 | Mutations in HIV-1 envelope that enhance entry with the macaque CD4 receptor alter antibody recognition by disrupting quaternary interactions within the trimer. |
Q36565090 | N-terminal substitutions in HIV-1 gp41 reduce the expression of non-trimeric envelope glycoproteins on the virus |
Q33874171 | Patterns of changes in human immunodeficiency virus type 1 V3 sequence populations late in infection |
Q37547061 | Quantification of entry phenotypes of macrophage-tropic HIV-1 across a wide range of CD4 densities |
Q34342950 | Rapid progression to simian AIDS can be accompanied by selection of CD4-independent gp120 variants with impaired ability to bind CD4. |
Q33984243 | Regulation of human immunodeficiency virus type 1 envelope glycoprotein fusion by a membrane-interactive domain in the gp41 cytoplasmic tail |
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Q36098903 | Replication-competent variants of human immunodeficiency virus type 2 lacking the V3 loop exhibit resistance to chemokine receptor antagonists |
Q34415921 | Sensitivity of HIV-1 to entry inhibitors correlates with envelope/coreceptor affinity, receptor density, and fusion kinetics |
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Q37207862 | Stable exposure of the coreceptor-binding site in a CD4-independent HIV-1 envelope protein |
Q36891087 | Structure and function of CC-chemokine receptor 5 homologues derived from representative primate species and subspecies of the taxonomic suborders Prosimii and Anthropoidea |
Q34332519 | Truncation of the cytoplasmic domain induces exposure of conserved regions in the ectodomain of human immunodeficiency virus type 1 envelope protein |
Q34996824 | Unique Pattern of Convergent Envelope Evolution in Simian Immunodeficiency Virus-Infected Rapid Progressor Macaques: Association with CD4-Independent Usage of CCR5 |
Q28469283 | V3 loop truncations in HIV-1 envelope impart resistance to coreceptor inhibitors and enhanced sensitivity to neutralizing antibodies |
Q35750908 | Vaccine-delivered HIV envelope inhibits CD4(+) T-cell activation, a mechanism for poor HIV vaccine responses |
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