Physical exercise increases autophagic signaling through ULK1 in human skeletal muscle

scientific article published on 12 February 2015

Physical exercise increases autophagic signaling through ULK1 in human skeletal muscle is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1152/JAPPLPHYSIOL.01116.2014
P698PubMed publication ID25678702

P50authorJens Otto Lunde JørgensenQ40020467
Niels JessenQ41440737
P2093author name stringNiels Møller
Andreas Buch Møller
Ann Mosegaard Bak
Berthil Forrest Clasen
Britt Christensen
Mikkel Holm Vendelbo
P2860cites workFasting-related autophagic response in slow- and fast-twitch skeletal muscleQ43149447
Autophagy and protein turnover signaling in slow-twitch muscle during exercise.Q45238955
Resistance exercise, but not endurance exercise, induces IKKβ phosphorylation in human skeletal muscle of training-accustomed individualsQ46122800
The serine/threonine kinase ULK1 is a target of multiple phosphorylation events.Q50335546
Differentiated mTOR but not AMPK signaling after strength vs endurance exercise in training-accustomed individuals.Q50877255
Autophagy is required to maintain muscle mass.Q51411361
Modulation of autophagy and ubiquitin-proteasome pathways during ultra-endurance running.Q54528865
Autophagy is defective in collagen VI muscular dystrophies and its reactivation rescues myofiber degenerationQ57639972
Guidelines for the use and interpretation of assays for monitoring autophagyQ21996341
Mammalian autophagy: core molecular machinery and signaling regulationQ24595447
Exercise-induced BCL2-regulated autophagy is required for muscle glucose homeostasisQ24601575
Physical exercise stimulates autophagy in normal skeletal muscles but is detrimental for collagen VI-deficient musclesQ24629897
mTOR: from growth signal integration to cancer, diabetes and ageingQ24633662
Dissociation of AMPK activity and ACCbeta phosphorylation in human muscle during prolonged exerciseQ28212929
AMPK and mTOR regulate autophagy through direct phosphorylation of Ulk1Q28506431
The role of autophagy during the early neonatal starvation periodQ29547489
How to interpret LC3 immunoblottingQ29614175
Phosphorylation of ULK1 (hATG1) by AMP-activated protein kinase connects energy sensing to mitophagyQ29615616
Autophagy is required for exercise training-induced skeletal muscle adaptation and improvement of physical performanceQ30410425
5'-AMP-activated protein kinase activity and protein expression are regulated by endurance training in human skeletal muscleQ34276280
Exercise increases TBC1D1 phosphorylation in human skeletal muscleQ35086804
Fasting increases human skeletal muscle net phenylalanine release and this is associated with decreased mTOR signalingQ35206436
Hyperammonemia-mediated autophagy in skeletal muscle contributes to sarcopenia of cirrhosis.Q36312414
The ULK1 complex: sensing nutrient signals for autophagy activation.Q38072216
Emerging regulation and functions of autophagyQ38118567
Skeletal muscle, autophagy, and physical activity: the ménage à trois of metabolic regulation in health and disease.Q38165741
AMP kinase in exercise adaptation of skeletal muscleQ38196625
Expression of the autophagy substrate SQSTM1/p62 is restored during prolonged starvation depending on transcriptional upregulation and autophagy-derived amino acidsQ39037029
Stain-Free technology as a normalization tool in Western blot analysisQ39255965
Direct effects of TNF-α on local fuel metabolism and cytokine levels in the placebo-controlled, bilaterally infused human leg: increased insulin sensitivity, increased net protein breakdown, and increased IL-6 release.Q42441090
Exercise induces autophagy in peripheral tissues and in the brainQ42531938
P433issue8
P407language of work or nameEnglishQ1860
P921main subjectautophagyQ288322
P304page(s)971-979
P577publication date2015-02-12
P1433published inJournal of Applied PhysiologyQ1091719
P1476titlePhysical exercise increases autophagic signaling through ULK1 in human skeletal muscle
P478volume118

Reverse relations

cites work (P2860)
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Q97529411Late Exercise Preconditioning Promotes Autophagy against Exhaustive Exercise-Induced Myocardial Injury through the Activation of the AMPK-mTOR-ULK1 Pathway
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