Acute and chronic nicotine exposure differentially facilitate the induction of LTP.

scientific article published on October 1999

Acute and chronic nicotine exposure differentially facilitate the induction of LTP. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1016/S0006-8993(99)01982-4
P8608Fatcat IDrelease_qdswuo23nbfmxc37s6qam4dquq
P698PubMed publication ID10536221

P2093author name stringFujii S
Ji Z
Morita N
Sumikawa K
P2860cites workLong-lasting potentiation of synaptic transmission in the dentate area of the anaesthetized rabbit following stimulation of the perforant pathQ24538964
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Hippocampal synaptic transmission enhanced by low concentrations of nicotine.Q48887721
Dissociation of the apparent relationship between nicotine tolerance and up-regulation of nicotinic receptorsQ48902723
Relationship between up-regulation of nicotine binding sites in rat brain and delayed cognitive enhancement observed after chronic or acute nicotinic receptor stimulationQ49029100
Effects of acute subcutaneous nicotine on attention, information processing and short-term memory in Alzheimer's disease.Q52046529
Nicotinic systems and cognitive functionQ52081673
Chronic treatments with cholinoceptor drugs influence spatial learning in rats.Q52228711
Nicotine and tetrahydroaminoacradine: Evidence for improved attention in patients with dementia of the Alzheimer typeQ57913221
P433issue1
P921main subjectnicotineQ12144
P304page(s)137-143
P577publication date1999-10-01
P1433published inBrain ResearchQ4955782
P1476titleAcute and chronic nicotine exposure differentially facilitate the induction of LTP.
P478volume846

Reverse relations

cites work (P2860)
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