scholarly article | Q13442814 |
P6179 | Dimensions Publication ID | 1027753240 |
P356 | DOI | 10.1186/1476-4598-11-18 |
P932 | PMC publication ID | 3337324 |
P698 | PubMed publication ID | 22452792 |
P5875 | ResearchGate publication ID | 221977792 |
P2093 | author name string | Xiaoli Wu | |
Hao Ding | |||
Sabine Mai | |||
Mojgan Rastegar | |||
Sam Kung | |||
Sumit Sandhu | |||
Zinnatun Nabi | |||
P2860 | cites work | Cloning of an SNF2/SWI2-related protein that binds specifically to the SPH motifs of the SV40 enhancer and to the HIV-1 promoter | Q24315927 |
HLTF gene silencing in human colon cancer | Q24536137 | ||
The role of chromosomal instability in tumor initiation | Q24541508 | ||
Polyubiquitination of proliferating cell nuclear antigen by HLTF and SHPRH prevents genomic instability from stalled replication forks | Q24642282 | ||
Human HLTF functions as a ubiquitin ligase for proliferating cell nuclear antigen polyubiquitination | Q24652076 | ||
A genetic model for colorectal tumorigenesis | Q27860582 | ||
Regulation of murine telomere length by Rtel: an essential gene encoding a helicase-like protein | Q28587423 | ||
Multiple intestinal neoplasia caused by a mutation in the murine homolog of the APC gene | Q29616490 | ||
Genetic instability in colorectal cancers | Q29618711 | ||
Microsatellite instability in cancer of the proximal colon | Q29620692 | ||
SHPRH and HLTF act in a damage-specific manner to coordinate different forms of postreplication repair and prevent mutagenesis | Q29871514 | ||
Annual report to the nation on the status of cancer, 1975-2006, featuring colorectal cancer trends and impact of interventions (risk factors, screening, and treatment) to reduce future rates | Q30538531 | ||
MECP2 isoform-specific vectors with regulated expression for Rett syndrome gene therapy | Q33497321 | ||
A resistant genetic background leading to incomplete penetrance of intestinal neoplasia and reduced loss of heterozygosity in ApcMin/+ mice | Q33556956 | ||
Role of double-stranded DNA translocase activity of human HLTF in replication of damaged DNA | Q33614018 | ||
Genomic and epigenetic instability in colorectal cancer pathogenesis | Q33836026 | ||
Adenomatous polyposis coli protein regulates the cellular response to DNA replication stress | Q33929860 | ||
Coordinated protein and DNA remodeling by human HLTF on stalled replication fork | Q33973384 | ||
Intestinal adenomas can develop with a stable karyotype and stable microsatellites | Q34066329 | ||
Role of yeast Rad5 and its human orthologs, HLTF and SHPRH in DNA damage tolerance | Q34094363 | ||
Inactivation of helicase-like transcription factor by promoter hypermethylation in human gastric cancer. | Q34199910 | ||
DNA hypermethylation and histone hypoacetylation of the HLTF gene are associated with reduced expression in gastric carcinoma | Q34220062 | ||
Genetic identification of Mom-1, a major modifier locus affecting Min-induced intestinal neoplasia in the mouse | Q34344768 | ||
Characterization of a helicase-like transcription factor involved in the expression of the human plasminogen activator inhibitor-1 gene | Q34385798 | ||
Developmental regulation of Zbu1, a DNA-binding member of the SWI2/SNF2 family | Q34423818 | ||
Stable karyotypes in epithelial cancer cell lines despite high rates of ongoing structural and numerical chromosomal instability | Q34775052 | ||
Pathology of mouse models of intestinal cancer: consensus report and recommendations | Q35075841 | ||
Generation of a mouse model for studying the role of upregulated RTEL1 activity in tumorigenesis | Q36205393 | ||
Biallelic inactivation of hMLH1 by epigenetic gene silencing, a novel mechanism causing human MSI cancers. | Q36213486 | ||
The checkpoint response to replication stress | Q37501718 | ||
DNA methylation markers in colorectal cancer | Q37687919 | ||
Maintaining genome stability at the replication fork | Q37696954 | ||
Cytogenetic characterization of two colon cell lines by using conventional G-banding, comparative genomic hybridization, and whole chromosome painting | Q38495887 | ||
Quantitative detection of promoter hypermethylation in multiple genes in the serum of patients with colorectal cancer. | Q46718275 | ||
Cloning, characterization, and steroid-dependent posttranscriptional processing of RUSH-1 alpha and beta, two uteroglobin promoter-binding proteins | Q48058368 | ||
Generation of conditional knockout alleles for PDGF-C. | Q50887587 | ||
Methylation of helicase-like transcription factor in serum of patients with colorectal cancer is an independent predictor of disease recurrence. | Q51771364 | ||
DNA-dependent adenosine triphosphatase (helicaselike transcription factor) activates beta-globin transcription in K562 cells. | Q52126123 | ||
Promoter methylation of helicase-like transcription factor is associated with the early stages of gastric cancer with family history. | Q53348996 | ||
Promoter hypermethylation of tumor-related genes in the progression of colorectal neoplasia. | Q53364696 | ||
The significance of unstable chromosomes in colorectal cancer. | Q53376250 | ||
Astrocyte-specific expression of activated p21-ras results in malignant astrocytoma formation in a transgenic mouse model of human gliomas. | Q53399373 | ||
Highly-methylated colorectal cancers show poorly-differentiated phenotype. | Q54564453 | ||
The structural nature of chromosomal instability in colon cancer cells | Q58109841 | ||
Mismatch repair gene defects in sporadic colorectal cancers with microsatellite instability | Q72127255 | ||
Methylation of serum DNA is an independent prognostic marker in colorectal cancer | Q79451531 | ||
CpG island methylator phenotype in cancer | Q81080870 | ||
Mouse model of colonic adenoma-carcinoma progression based on somatic Apc inactivation | Q81454513 | ||
Molecular Genetics of Colorectal Cancer | Q82521439 | ||
CpG island methylation of genes accumulates during the adenoma progression step of the multistep pathogenesis of colorectal cancer | Q83365535 | ||
P921 | main subject | carcinogenesis | Q1637543 |
P304 | page(s) | 18 | |
P577 | publication date | 2012-03-27 | |
P1433 | published in | Molecular Cancer | Q15724585 |
P1476 | title | Loss of HLTF function promotes intestinal carcinogenesis | |
P478 | volume | 11 |
Q35268479 | Alterations of DNA repair genes in the NCI-60 cell lines and their predictive value for anticancer drug activity. |
Q34460069 | Cellular commitment in the developing cerebellum |
Q33611481 | Circulating cell-free methylated DNA and lactate dehydrogenase release in colorectal cancer |
Q47735296 | Functions of SMARCAL1, ZRANB3, and HLTF in maintaining genome stability |
Q36904912 | HIV-1 Vpr degrades the HLTF DNA translocase in T cells and macrophages |
Q35762843 | HLTF's Ancient HIRAN Domain Binds 3' DNA Ends to Drive Replication Fork Reversal. |
Q92956640 | Helicase-like transcription factor (Hltf) gene-deletion promotes oxidative phosphorylation (OXPHOS) in colorectal tumors of AOM/DSS-treated mice |
Q27301898 | Helicase-like transcription factor (Hltf) regulates G2/M transition, Wt1/Gata4/Hif-1a cardiac transcription networks, and collagen biogenesis |
Q52719002 | Helicase-like transcription factor expression is associated with a poor prognosis in Non-Small-Cell Lung Cancer (NSCLC). |
Q30434300 | Helicase-like transcription factor: a new marker of well-differentiated thyroid cancers |
Q36672248 | Heterogeneous DNA methylation contributes to tumorigenesis through inducing the loss of coexpression connectivity in colorectal cancer |
Q38866681 | Homologous recombination maintenance of genome integrity during DNA damage tolerance |
Q29871515 | Human HLTF mediates postreplication repair by its HIRAN domain-dependent replication fork remodelling. |
Q38301131 | MicroRNA-145 contributes to enhancing radiosensitivity of cervical cancer cells |
Q34796010 | Role of helicase-like transcription factor (hltf) in the G2/m transition and apoptosis in brain |
Q38893314 | The Role of Stem Cell DNA Methylation in Colorectal Carcinogenesis |
Q38608560 | The helicase-like transcription factor (HLTF) in cancer: loss of function or oncomorphic conversion of a tumor suppressor? |
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